CD47/SIRPα blocking peptide identification and synergistic effect with irradiation for cancer immunotherapy

Background Immunotherapy has achieved remarkable advances via a variety of strategies against tumor cells that evade immune surveillance. As important innate immune cells, macrophages play important roles in maintaining homeostasis, preventing pathogen invasion, resisting tumor cells and promoting a...

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Main Authors: Hongfei Wang, Yixuan Sun, Xiuman Zhou, Chunxia Chen, Ling Jiao, Wanqiong Li, Shanshan Gou, Yanying Li, Jiangfeng Du, Guanyu Chen, Wenjie Zhai, Yahong Wu, Yuanming Qi, Yanfeng Gao
Format: Article
Language:English
Published: BMJ Publishing Group 2020-10-01
Series:Journal for ImmunoTherapy of Cancer
Online Access:https://jitc.bmj.com/content/8/2/e000905.full
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author Hongfei Wang
Yixuan Sun
Xiuman Zhou
Chunxia Chen
Ling Jiao
Wanqiong Li
Shanshan Gou
Yanying Li
Jiangfeng Du
Guanyu Chen
Wenjie Zhai
Yahong Wu
Yuanming Qi
Yanfeng Gao
author_facet Hongfei Wang
Yixuan Sun
Xiuman Zhou
Chunxia Chen
Ling Jiao
Wanqiong Li
Shanshan Gou
Yanying Li
Jiangfeng Du
Guanyu Chen
Wenjie Zhai
Yahong Wu
Yuanming Qi
Yanfeng Gao
author_sort Hongfei Wang
collection DOAJ
description Background Immunotherapy has achieved remarkable advances via a variety of strategies against tumor cells that evade immune surveillance. As important innate immune cells, macrophages play important roles in maintaining homeostasis, preventing pathogen invasion, resisting tumor cells and promoting adaptive immune response. CD47 is found to be overexpressed on tumor cells and act as a don’t eat me’ signal, which contributes to immune evasion. Macrophages mediated phagocytosis via blockade CD47/SIRPα (signal regulatory protein alpha) interaction was proved to induce effective antitumor immune response.Methods A novel peptide pep-20, specifically targeting CD47 and blocking CD47/SIRPα interaction, was identified via high-throughput phage display library bio-panning. The capability to enhance the macrophage-mediated phagocytosis activities and antitumor effects of pep-20 were investigated. The mechanism of pep-20 to induce T-cell response was explored by ex vivo analysis and confirmed via macrophage depleting strategy. The structure-activity relationship and D-amino acid substitution of pep-20 were also studied. The antitumor effects and mechanism of a proteolysis resistant D-amino acid derivate pep-20-D12 combined with irradiation (IR) were also investigated.Results Pep-20 showed remarkable enhancement of macrophage-mediated phagocytosis to both solid and hematologic tumor cells in vitro, and inhibited tumor growth in immune-competent tumor-bearing mice. Furthermore, pep-20 promoted macrophages to mobilize the antitumor T-cell response with minimal toxicity. Furthermore, systemic administration of the derivate pep-20-D12 showed robust synergistic antitumor efficacy in combination with IR.Conclusion In summary, these results demonstrated that CD47/SIRPα blocking peptides, pep-20 and its derivate, could serve as promising candidates to promote macrophages-mediated phagocytosis and immune response in cancer immunotherapy.
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spelling doaj-art-a73fbb488d6d4721a1bbd7f6e80730522025-08-20T02:13:19ZengBMJ Publishing GroupJournal for ImmunoTherapy of Cancer2051-14262020-10-018210.1136/jitc-2020-000905CD47/SIRPα blocking peptide identification and synergistic effect with irradiation for cancer immunotherapyHongfei Wang0Yixuan Sun1Xiuman Zhou2Chunxia Chen3Ling Jiao4Wanqiong Li5Shanshan Gou6Yanying Li7Jiangfeng Du8Guanyu Chen9Wenjie Zhai10Yahong Wu11Yuanming Qi12Yanfeng Gao131 School of Life Sciences, Zhengzhou University, Zhengzhou, Henan, China1 School of Life Sciences, Zhengzhou University, Zhengzhou, Henan, ChinaSchool of Pharmaceutical Sciences (Shenzhen), Shenzhen Campus of Sun Yat-sen University, Shenzhen, ChinaSchool of Life Sciences, Zhengzhou University, Zhengzhou, China1 School of Life Sciences, Zhengzhou University, Zhengzhou, Henan, China2 School of Pharmaceutical Sciences (Shenzhen), Sun Yat-sen University, Shenzhen, Guangdong, China1 School of Life Sciences, Zhengzhou University, Zhengzhou, Henan, ChinaDepartment of Endocrinology, Affiliated Hospital of Jining Medical University, Jining, Shandong, China1 School of Life Sciences, Zhengzhou University, Zhengzhou, Henan, ChinaSchool of Pharmaceutical Sciences (Shenzhen), Shenzhen Campus of Sun Yat-sen University, Shenzhen, ChinaSchool of Life Sciences, Zhengzhou University, Zhengzhou, ChinaSchool of Life Sciences, Zhengzhou University, Zhengzhou, China1 School of Life Sciences, Zhengzhou University, Zhengzhou, Henan, ChinaSchool of Pharmaceutical Sciences (Shenzhen), Shenzhen Campus of Sun Yat-sen University, Shenzhen, ChinaBackground Immunotherapy has achieved remarkable advances via a variety of strategies against tumor cells that evade immune surveillance. As important innate immune cells, macrophages play important roles in maintaining homeostasis, preventing pathogen invasion, resisting tumor cells and promoting adaptive immune response. CD47 is found to be overexpressed on tumor cells and act as a don’t eat me’ signal, which contributes to immune evasion. Macrophages mediated phagocytosis via blockade CD47/SIRPα (signal regulatory protein alpha) interaction was proved to induce effective antitumor immune response.Methods A novel peptide pep-20, specifically targeting CD47 and blocking CD47/SIRPα interaction, was identified via high-throughput phage display library bio-panning. The capability to enhance the macrophage-mediated phagocytosis activities and antitumor effects of pep-20 were investigated. The mechanism of pep-20 to induce T-cell response was explored by ex vivo analysis and confirmed via macrophage depleting strategy. The structure-activity relationship and D-amino acid substitution of pep-20 were also studied. The antitumor effects and mechanism of a proteolysis resistant D-amino acid derivate pep-20-D12 combined with irradiation (IR) were also investigated.Results Pep-20 showed remarkable enhancement of macrophage-mediated phagocytosis to both solid and hematologic tumor cells in vitro, and inhibited tumor growth in immune-competent tumor-bearing mice. Furthermore, pep-20 promoted macrophages to mobilize the antitumor T-cell response with minimal toxicity. Furthermore, systemic administration of the derivate pep-20-D12 showed robust synergistic antitumor efficacy in combination with IR.Conclusion In summary, these results demonstrated that CD47/SIRPα blocking peptides, pep-20 and its derivate, could serve as promising candidates to promote macrophages-mediated phagocytosis and immune response in cancer immunotherapy.https://jitc.bmj.com/content/8/2/e000905.full
spellingShingle Hongfei Wang
Yixuan Sun
Xiuman Zhou
Chunxia Chen
Ling Jiao
Wanqiong Li
Shanshan Gou
Yanying Li
Jiangfeng Du
Guanyu Chen
Wenjie Zhai
Yahong Wu
Yuanming Qi
Yanfeng Gao
CD47/SIRPα blocking peptide identification and synergistic effect with irradiation for cancer immunotherapy
Journal for ImmunoTherapy of Cancer
title CD47/SIRPα blocking peptide identification and synergistic effect with irradiation for cancer immunotherapy
title_full CD47/SIRPα blocking peptide identification and synergistic effect with irradiation for cancer immunotherapy
title_fullStr CD47/SIRPα blocking peptide identification and synergistic effect with irradiation for cancer immunotherapy
title_full_unstemmed CD47/SIRPα blocking peptide identification and synergistic effect with irradiation for cancer immunotherapy
title_short CD47/SIRPα blocking peptide identification and synergistic effect with irradiation for cancer immunotherapy
title_sort cd47 sirpα blocking peptide identification and synergistic effect with irradiation for cancer immunotherapy
url https://jitc.bmj.com/content/8/2/e000905.full
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