Central adiposity and α-klotho: inflammatory mechanisms underlying aging biomarkers related to body roundness index

Abstract Background Obesity is a global health issue which has been widely accepted as an aging related pathogenesis. α-Klotho is a protein involved in aging process, mineral metabolism, insulin sensitivity, and the pathogenesis of various age-related diseases. Adiposity correlates with lower solubl...

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Main Authors: Rui Du, Xiaoyan Tang, Lei Guan, Yuchen Lai, Huijuan Xiang, Wei Huang
Format: Article
Language:English
Published: BMC 2025-04-01
Series:Lipids in Health and Disease
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Online Access:https://doi.org/10.1186/s12944-025-02541-6
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author Rui Du
Xiaoyan Tang
Lei Guan
Yuchen Lai
Huijuan Xiang
Wei Huang
author_facet Rui Du
Xiaoyan Tang
Lei Guan
Yuchen Lai
Huijuan Xiang
Wei Huang
author_sort Rui Du
collection DOAJ
description Abstract Background Obesity is a global health issue which has been widely accepted as an aging related pathogenesis. α-Klotho is a protein involved in aging process, mineral metabolism, insulin sensitivity, and the pathogenesis of various age-related diseases. Adiposity correlates with lower soluble α-Klotho, but the role of fat distribution and inflammation remains unclear. The body roundness index (BRI) refines central adiposity assessment beyond BMI. Herein, We aimed to investigate the relationship of BRI, inflammation and serum level of soluble α-Klotho. Methods We conducted a cross-sectional analysis of 9,958 U.S. adults (40–79 years) from the 2007–2016 NHANES. We examined association between BRI and serum α-Klotho (SαKl) levels, controlling for demographic, socioeconomic, lifestyle, and clinical factors. We also assessed whether inflammatory markers mediated the BRI–SαKl relationship. Results BRI was inversely associated with SαKl levels (P < 0.05). A significant sex interaction was found (P < 0.001), while BRI was positively correlated with multiple proinflammatory markers, which were all inversely related to SαKl levels. Mediation analyses showed inflammatory markers accounted for 20.5% (WBC), 18.0% (neutrophils), and 12.3% (platelets) of the BRI–SαKl association. Conclusion More severe central adiposity measured by BRI was related to lower SαKl, which may partly be attributed to inflammation. These findings underscore the importance of fat distribution and inflammation in obesity-related aging and may guide interventions to preserve SαKl levels. Longitudinal studies are needed to confirm causality and inform future strategies.
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spelling doaj-art-a70dbb80576447599f5b5b456e44754d2025-08-20T03:06:52ZengBMCLipids in Health and Disease1476-511X2025-04-0124111210.1186/s12944-025-02541-6Central adiposity and α-klotho: inflammatory mechanisms underlying aging biomarkers related to body roundness indexRui Du0Xiaoyan Tang1Lei Guan2Yuchen Lai3Huijuan Xiang4Wei Huang5Department of Ultrasound, General Hospital of Central Theater CommandDepartment of Cardiology, General Hospital of Central Theater CommandDepartment of Cardiology, General Hospital of Central Theater CommandSchool of Medicine, Wuhan University of Science and TechnologyDepartment of Ultrasound, General Hospital of Central Theater CommandDepartment of Cardiology, General Hospital of Central Theater CommandAbstract Background Obesity is a global health issue which has been widely accepted as an aging related pathogenesis. α-Klotho is a protein involved in aging process, mineral metabolism, insulin sensitivity, and the pathogenesis of various age-related diseases. Adiposity correlates with lower soluble α-Klotho, but the role of fat distribution and inflammation remains unclear. The body roundness index (BRI) refines central adiposity assessment beyond BMI. Herein, We aimed to investigate the relationship of BRI, inflammation and serum level of soluble α-Klotho. Methods We conducted a cross-sectional analysis of 9,958 U.S. adults (40–79 years) from the 2007–2016 NHANES. We examined association between BRI and serum α-Klotho (SαKl) levels, controlling for demographic, socioeconomic, lifestyle, and clinical factors. We also assessed whether inflammatory markers mediated the BRI–SαKl relationship. Results BRI was inversely associated with SαKl levels (P < 0.05). A significant sex interaction was found (P < 0.001), while BRI was positively correlated with multiple proinflammatory markers, which were all inversely related to SαKl levels. Mediation analyses showed inflammatory markers accounted for 20.5% (WBC), 18.0% (neutrophils), and 12.3% (platelets) of the BRI–SαKl association. Conclusion More severe central adiposity measured by BRI was related to lower SαKl, which may partly be attributed to inflammation. These findings underscore the importance of fat distribution and inflammation in obesity-related aging and may guide interventions to preserve SαKl levels. Longitudinal studies are needed to confirm causality and inform future strategies.https://doi.org/10.1186/s12944-025-02541-6α−KlothoAgingObesityBody roundness indexNHANES
spellingShingle Rui Du
Xiaoyan Tang
Lei Guan
Yuchen Lai
Huijuan Xiang
Wei Huang
Central adiposity and α-klotho: inflammatory mechanisms underlying aging biomarkers related to body roundness index
Lipids in Health and Disease
α−Klotho
Aging
Obesity
Body roundness index
NHANES
title Central adiposity and α-klotho: inflammatory mechanisms underlying aging biomarkers related to body roundness index
title_full Central adiposity and α-klotho: inflammatory mechanisms underlying aging biomarkers related to body roundness index
title_fullStr Central adiposity and α-klotho: inflammatory mechanisms underlying aging biomarkers related to body roundness index
title_full_unstemmed Central adiposity and α-klotho: inflammatory mechanisms underlying aging biomarkers related to body roundness index
title_short Central adiposity and α-klotho: inflammatory mechanisms underlying aging biomarkers related to body roundness index
title_sort central adiposity and α klotho inflammatory mechanisms underlying aging biomarkers related to body roundness index
topic α−Klotho
Aging
Obesity
Body roundness index
NHANES
url https://doi.org/10.1186/s12944-025-02541-6
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