Postbiotic Intervention in Sarcopenia: The Role of <i>Lactiplantibacillus plantarum</i> HY7715 and Its Extracellular Vesicles

Sarcopenia, the age-related loss of skeletal muscle mass and function, is associated with inflammation, mitochondrial dysfunction, and gut barrier impairment. This study investigates the postbiotic effects of heat-killed <i>Lactiplantibacillus plantarum</i> HY7715 (HY7715) and its extrac...

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Main Authors: Kippeum Lee, Soo Dong Park, Joo Yun Kim, Jae Jung Shim, Jae Hwan Lee
Format: Article
Language:English
Published: MDPI AG 2025-07-01
Series:Life
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Online Access:https://www.mdpi.com/2075-1729/15/7/1101
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author Kippeum Lee
Soo Dong Park
Joo Yun Kim
Jae Jung Shim
Jae Hwan Lee
author_facet Kippeum Lee
Soo Dong Park
Joo Yun Kim
Jae Jung Shim
Jae Hwan Lee
author_sort Kippeum Lee
collection DOAJ
description Sarcopenia, the age-related loss of skeletal muscle mass and function, is associated with inflammation, mitochondrial dysfunction, and gut barrier impairment. This study investigates the postbiotic effects of heat-killed <i>Lactiplantibacillus plantarum</i> HY7715 (HY7715) and its extracellular vesicles (EVs) on muscle health and intestinal integrity. In C2C12 myotubes, both treatments enhanced myogenic differentiation by upregulating Myf5 and MYOG, and improved mitochondrial activity and biogenesis via increased PGC1α and mTOR expression. Under TNFα-induced muscle atrophy, they suppressed expression of atrophy-related markers (Fbox32, MuRF1, and myostatin). EVs showed stronger anti-inflammatory effects by reducing IL6 expression in muscle cells. In intestinal Caco-2 cells, HY7715-derived EVs improved barrier function by upregulating tight junction proteins (ZO-1, occludin, and claudins), and effectively reduced LPS-induced inflammation. These findings suggest that heat-killed HY7715 and its EVs may alleviate sarcopenia by enhancing muscle regeneration and maintaining intestinal homeostasis, highlighting their potential as safe, gut–muscle axis-targeting postbiotic interventions for healthy aging.
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spelling doaj-art-a684ae6e47874db595f0aa32bc36f0652025-08-20T02:45:46ZengMDPI AGLife2075-17292025-07-01157110110.3390/life15071101Postbiotic Intervention in Sarcopenia: The Role of <i>Lactiplantibacillus plantarum</i> HY7715 and Its Extracellular VesiclesKippeum Lee0Soo Dong Park1Joo Yun Kim2Jae Jung Shim3Jae Hwan Lee4R&BD Center, Hy Co., Ltd., 22 Giheungdanji-ro 24 Beon-gil, Giheung-gu, Yongin-si 17086, Republic of KoreaR&BD Center, Hy Co., Ltd., 22 Giheungdanji-ro 24 Beon-gil, Giheung-gu, Yongin-si 17086, Republic of KoreaR&BD Center, Hy Co., Ltd., 22 Giheungdanji-ro 24 Beon-gil, Giheung-gu, Yongin-si 17086, Republic of KoreaR&BD Center, Hy Co., Ltd., 22 Giheungdanji-ro 24 Beon-gil, Giheung-gu, Yongin-si 17086, Republic of KoreaR&BD Center, Hy Co., Ltd., 22 Giheungdanji-ro 24 Beon-gil, Giheung-gu, Yongin-si 17086, Republic of KoreaSarcopenia, the age-related loss of skeletal muscle mass and function, is associated with inflammation, mitochondrial dysfunction, and gut barrier impairment. This study investigates the postbiotic effects of heat-killed <i>Lactiplantibacillus plantarum</i> HY7715 (HY7715) and its extracellular vesicles (EVs) on muscle health and intestinal integrity. In C2C12 myotubes, both treatments enhanced myogenic differentiation by upregulating Myf5 and MYOG, and improved mitochondrial activity and biogenesis via increased PGC1α and mTOR expression. Under TNFα-induced muscle atrophy, they suppressed expression of atrophy-related markers (Fbox32, MuRF1, and myostatin). EVs showed stronger anti-inflammatory effects by reducing IL6 expression in muscle cells. In intestinal Caco-2 cells, HY7715-derived EVs improved barrier function by upregulating tight junction proteins (ZO-1, occludin, and claudins), and effectively reduced LPS-induced inflammation. These findings suggest that heat-killed HY7715 and its EVs may alleviate sarcopenia by enhancing muscle regeneration and maintaining intestinal homeostasis, highlighting their potential as safe, gut–muscle axis-targeting postbiotic interventions for healthy aging.https://www.mdpi.com/2075-1729/15/7/1101postbiotics<i>Lactiplantibacillus plantarum</i> HY7715extracellular vesiclesarcopeniagut barrier impairment
spellingShingle Kippeum Lee
Soo Dong Park
Joo Yun Kim
Jae Jung Shim
Jae Hwan Lee
Postbiotic Intervention in Sarcopenia: The Role of <i>Lactiplantibacillus plantarum</i> HY7715 and Its Extracellular Vesicles
Life
postbiotics
<i>Lactiplantibacillus plantarum</i> HY7715
extracellular vesicle
sarcopenia
gut barrier impairment
title Postbiotic Intervention in Sarcopenia: The Role of <i>Lactiplantibacillus plantarum</i> HY7715 and Its Extracellular Vesicles
title_full Postbiotic Intervention in Sarcopenia: The Role of <i>Lactiplantibacillus plantarum</i> HY7715 and Its Extracellular Vesicles
title_fullStr Postbiotic Intervention in Sarcopenia: The Role of <i>Lactiplantibacillus plantarum</i> HY7715 and Its Extracellular Vesicles
title_full_unstemmed Postbiotic Intervention in Sarcopenia: The Role of <i>Lactiplantibacillus plantarum</i> HY7715 and Its Extracellular Vesicles
title_short Postbiotic Intervention in Sarcopenia: The Role of <i>Lactiplantibacillus plantarum</i> HY7715 and Its Extracellular Vesicles
title_sort postbiotic intervention in sarcopenia the role of i lactiplantibacillus plantarum i hy7715 and its extracellular vesicles
topic postbiotics
<i>Lactiplantibacillus plantarum</i> HY7715
extracellular vesicle
sarcopenia
gut barrier impairment
url https://www.mdpi.com/2075-1729/15/7/1101
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