Involvement of SIRT3/FOXO1 and TLR4/NF-κB/NLRP3 pathways in protective effects of Δ(9)-tetrahydrocannabinol on diabetic cardiomyopathy in rats

Diabetic cardiomyopathy (DCM) is a chronic complication of type 2 diabetes mellitus, leading to heart failure. Addressing DCM requires a comprehensive therapeutic approach. This study examines the protective role of Δ(9)-tetrahydrocannabinol (THC) in DCM by assessing its impact on cardiac function,...

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Main Authors: Zhang Haiping, Hui Xiaoli, Xu Hua
Format: Article
Language:English
Published: De Gruyter 2025-03-01
Series:Türk Biyokimya Dergisi
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Online Access:https://doi.org/10.1515/tjb-2024-0258
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author Zhang Haiping
Hui Xiaoli
Xu Hua
author_facet Zhang Haiping
Hui Xiaoli
Xu Hua
author_sort Zhang Haiping
collection DOAJ
description Diabetic cardiomyopathy (DCM) is a chronic complication of type 2 diabetes mellitus, leading to heart failure. Addressing DCM requires a comprehensive therapeutic approach. This study examines the protective role of Δ(9)-tetrahydrocannabinol (THC) in DCM by assessing its impact on cardiac function, inflammation, oxidative stress, and mitochondrial health and exploring the involvement of the SIRT3/FOXO1 and TLR4/NF-κB/NLRP3 signaling pathways.
format Article
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issn 1303-829X
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publishDate 2025-03-01
publisher De Gruyter
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series Türk Biyokimya Dergisi
spelling doaj-art-a66d2bcdca684e37b36d470f29f464442025-08-20T03:11:07ZengDe GruyterTürk Biyokimya Dergisi1303-829X2025-03-0150230431210.1515/tjb-2024-0258Involvement of SIRT3/FOXO1 and TLR4/NF-κB/NLRP3 pathways in protective effects of Δ(9)-tetrahydrocannabinol on diabetic cardiomyopathy in ratsZhang Haiping0Hui Xiaoli1Xu Hua2Department of Disinfection Supply, The First Affiliated Hospital of Xi’an Jiaotong University, Xi’an, ChinaSecond Department of Geriatric Internal Medicine, The First Affiliated Hospital of Xi’an Jiaotong University, Xi’an, ChinaDepartment of Tumor Radiotherapy, Xi’an International Medical Center, Xi’an, ChinaDiabetic cardiomyopathy (DCM) is a chronic complication of type 2 diabetes mellitus, leading to heart failure. Addressing DCM requires a comprehensive therapeutic approach. This study examines the protective role of Δ(9)-tetrahydrocannabinol (THC) in DCM by assessing its impact on cardiac function, inflammation, oxidative stress, and mitochondrial health and exploring the involvement of the SIRT3/FOXO1 and TLR4/NF-κB/NLRP3 signaling pathways.https://doi.org/10.1515/tjb-2024-0258diabetescannabinoidmitochondriacardiomyopathyinflammationmechanism
spellingShingle Zhang Haiping
Hui Xiaoli
Xu Hua
Involvement of SIRT3/FOXO1 and TLR4/NF-κB/NLRP3 pathways in protective effects of Δ(9)-tetrahydrocannabinol on diabetic cardiomyopathy in rats
Türk Biyokimya Dergisi
diabetes
cannabinoid
mitochondria
cardiomyopathy
inflammation
mechanism
title Involvement of SIRT3/FOXO1 and TLR4/NF-κB/NLRP3 pathways in protective effects of Δ(9)-tetrahydrocannabinol on diabetic cardiomyopathy in rats
title_full Involvement of SIRT3/FOXO1 and TLR4/NF-κB/NLRP3 pathways in protective effects of Δ(9)-tetrahydrocannabinol on diabetic cardiomyopathy in rats
title_fullStr Involvement of SIRT3/FOXO1 and TLR4/NF-κB/NLRP3 pathways in protective effects of Δ(9)-tetrahydrocannabinol on diabetic cardiomyopathy in rats
title_full_unstemmed Involvement of SIRT3/FOXO1 and TLR4/NF-κB/NLRP3 pathways in protective effects of Δ(9)-tetrahydrocannabinol on diabetic cardiomyopathy in rats
title_short Involvement of SIRT3/FOXO1 and TLR4/NF-κB/NLRP3 pathways in protective effects of Δ(9)-tetrahydrocannabinol on diabetic cardiomyopathy in rats
title_sort involvement of sirt3 foxo1 and tlr4 nf κb nlrp3 pathways in protective effects of δ 9 tetrahydrocannabinol on diabetic cardiomyopathy in rats
topic diabetes
cannabinoid
mitochondria
cardiomyopathy
inflammation
mechanism
url https://doi.org/10.1515/tjb-2024-0258
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AT huixiaoli involvementofsirt3foxo1andtlr4nfkbnlrp3pathwaysinprotectiveeffectsofd9tetrahydrocannabinolondiabeticcardiomyopathyinrats
AT xuhua involvementofsirt3foxo1andtlr4nfkbnlrp3pathwaysinprotectiveeffectsofd9tetrahydrocannabinolondiabeticcardiomyopathyinrats