Atoh8 expression inhibition promoted osteogenic differentiation of ADSCs and inhibited cell proliferation in vitro and rat bone defect models

Stem cell-based bone tissue engineering offers a promising approach for treating oral and cranio-maxillofacial bone defects. This study investigated the role of Atoh8, a key regulator in various cells, in the osteogenic potential of adipose-derived stem cells (ADSCs). ADSCs transfected with small in...

Full description

Saved in:
Bibliographic Details
Main Authors: Zian Yi, Shuang Song, Yuxin Bai, Guanhua Zhang, Yuxi Wang, Zijun Chen, Xuefeng Chen, Banglian Deng, Xiangdong Liu, Zuolin Jin
Format: Article
Language:English
Published: Taylor & Francis Group 2025-12-01
Series:Adipocyte
Subjects:
Online Access:https://www.tandfonline.com/doi/10.1080/21623945.2025.2494089
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850190253398163456
author Zian Yi
Shuang Song
Yuxin Bai
Guanhua Zhang
Yuxi Wang
Zijun Chen
Xuefeng Chen
Banglian Deng
Xiangdong Liu
Zuolin Jin
author_facet Zian Yi
Shuang Song
Yuxin Bai
Guanhua Zhang
Yuxi Wang
Zijun Chen
Xuefeng Chen
Banglian Deng
Xiangdong Liu
Zuolin Jin
author_sort Zian Yi
collection DOAJ
description Stem cell-based bone tissue engineering offers a promising approach for treating oral and cranio-maxillofacial bone defects. This study investigated the role of Atoh8, a key regulator in various cells, in the osteogenic potential of adipose-derived stem cells (ADSCs). ADSCs transfected with small interfering RNA (siRNA) targeting Atoh8 were evaluated for proliferation, migration, adhesion, and osteogenic capacity. In vivo, 20 SD rats were used to assess bone regeneration using Atoh8-knockdown ADSC sheets, with new bone formation quantified via micro-CT and histological analysis. Atoh8 knockdown in vitro reduced ADSC proliferation and migration but enhanced osteogenic differentiation and upregulation of osteogenic-related factors. This approach improved bone healing in rat defect models, accelerating repair both in vitro and in vivo. The findings underscore the clinical potential of ADSCs in bone tissue engineering and elucidate Atoh8’s regulatory role in ADSC osteogenesis, providing a novel therapeutic strategy for enhancing bone regeneration through targeted modulation of stem cell differentiation pathways.
format Article
id doaj-art-a4a1b86f73e14d74bf1b3545b7496d19
institution OA Journals
issn 2162-3945
2162-397X
language English
publishDate 2025-12-01
publisher Taylor & Francis Group
record_format Article
series Adipocyte
spelling doaj-art-a4a1b86f73e14d74bf1b3545b7496d192025-08-20T02:15:20ZengTaylor & Francis GroupAdipocyte2162-39452162-397X2025-12-0114110.1080/21623945.2025.2494089Atoh8 expression inhibition promoted osteogenic differentiation of ADSCs and inhibited cell proliferation in vitro and rat bone defect modelsZian Yi0Shuang Song1Yuxin Bai2Guanhua Zhang3Yuxi Wang4Zijun Chen5Xuefeng Chen6Banglian Deng7Xiangdong Liu8Zuolin Jin9State Key Laboratory of Oral & Maxillofacial Reconstruction and Regeneration & National Clinical Research Center for Oral Diseases & Shaanxi Clinical Research Center for Oral Diseases, Department of Orthodontics, School of Stomatology, Air Force Medical University, Xi’an, ChinaKey Laboratory of Shaanxi Province for Craniofacial Precision Medicine Research, College of Stomatology, Xi’an Jiaotong University, Xi’an, ChinaState Key Laboratory of Oral & Maxillofacial Reconstruction and Regeneration & National Clinical Research Center for Oral Diseases & Shaanxi Clinical Research Center for Oral Diseases, Department of Oral Implantology, School of Stomatology, Air Force Medical University, Xi’an, ChinaDepartment of Stomatology, Electric Power Teaching Hospital, Capital Medical University, Beijing, ChinaState Key Laboratory of Oral & Maxillofacial Reconstruction and Regeneration & National Clinical Research Center for Oral Diseases & Shaanxi Clinical Research Center for Oral Diseases, Department of Oral Implantology, School of Stomatology, Air Force Medical University, Xi’an, ChinaState Key Laboratory of Oral & Maxillofacial Reconstruction and Regeneration & National Clinical Research Center for Oral Diseases & Shaanxi Clinical Research Center for Oral Diseases, Department of Oral Implantology, School of Stomatology, Air Force Medical University, Xi’an, ChinaLianbang Research Institute of Oral Technology, Lianbang Hospital of Stomatology, Xi’an, ChinaLianbang Research Institute of Oral Technology, Lianbang Hospital of Stomatology, Xi’an, ChinaState Key Laboratory of Oral & Maxillofacial Reconstruction and Regeneration & National Clinical Research Center for Oral Diseases & Shaanxi Clinical Research Center for Oral Diseases, Department of Trauma and Orthognathic Surgery, School of Stomatology, Air Force Medical University, Xi’an, ChinaState Key Laboratory of Oral & Maxillofacial Reconstruction and Regeneration & National Clinical Research Center for Oral Diseases & Shaanxi Clinical Research Center for Oral Diseases, Department of Orthodontics, School of Stomatology, Air Force Medical University, Xi’an, ChinaStem cell-based bone tissue engineering offers a promising approach for treating oral and cranio-maxillofacial bone defects. This study investigated the role of Atoh8, a key regulator in various cells, in the osteogenic potential of adipose-derived stem cells (ADSCs). ADSCs transfected with small interfering RNA (siRNA) targeting Atoh8 were evaluated for proliferation, migration, adhesion, and osteogenic capacity. In vivo, 20 SD rats were used to assess bone regeneration using Atoh8-knockdown ADSC sheets, with new bone formation quantified via micro-CT and histological analysis. Atoh8 knockdown in vitro reduced ADSC proliferation and migration but enhanced osteogenic differentiation and upregulation of osteogenic-related factors. This approach improved bone healing in rat defect models, accelerating repair both in vitro and in vivo. The findings underscore the clinical potential of ADSCs in bone tissue engineering and elucidate Atoh8’s regulatory role in ADSC osteogenesis, providing a novel therapeutic strategy for enhancing bone regeneration through targeted modulation of stem cell differentiation pathways.https://www.tandfonline.com/doi/10.1080/21623945.2025.2494089Atoh8adipose-derived stem cellsosteogenic differentiationbone repairstem cell transplantation
spellingShingle Zian Yi
Shuang Song
Yuxin Bai
Guanhua Zhang
Yuxi Wang
Zijun Chen
Xuefeng Chen
Banglian Deng
Xiangdong Liu
Zuolin Jin
Atoh8 expression inhibition promoted osteogenic differentiation of ADSCs and inhibited cell proliferation in vitro and rat bone defect models
Adipocyte
Atoh8
adipose-derived stem cells
osteogenic differentiation
bone repair
stem cell transplantation
title Atoh8 expression inhibition promoted osteogenic differentiation of ADSCs and inhibited cell proliferation in vitro and rat bone defect models
title_full Atoh8 expression inhibition promoted osteogenic differentiation of ADSCs and inhibited cell proliferation in vitro and rat bone defect models
title_fullStr Atoh8 expression inhibition promoted osteogenic differentiation of ADSCs and inhibited cell proliferation in vitro and rat bone defect models
title_full_unstemmed Atoh8 expression inhibition promoted osteogenic differentiation of ADSCs and inhibited cell proliferation in vitro and rat bone defect models
title_short Atoh8 expression inhibition promoted osteogenic differentiation of ADSCs and inhibited cell proliferation in vitro and rat bone defect models
title_sort atoh8 expression inhibition promoted osteogenic differentiation of adscs and inhibited cell proliferation in vitro and rat bone defect models
topic Atoh8
adipose-derived stem cells
osteogenic differentiation
bone repair
stem cell transplantation
url https://www.tandfonline.com/doi/10.1080/21623945.2025.2494089
work_keys_str_mv AT zianyi atoh8expressioninhibitionpromotedosteogenicdifferentiationofadscsandinhibitedcellproliferationinvitroandratbonedefectmodels
AT shuangsong atoh8expressioninhibitionpromotedosteogenicdifferentiationofadscsandinhibitedcellproliferationinvitroandratbonedefectmodels
AT yuxinbai atoh8expressioninhibitionpromotedosteogenicdifferentiationofadscsandinhibitedcellproliferationinvitroandratbonedefectmodels
AT guanhuazhang atoh8expressioninhibitionpromotedosteogenicdifferentiationofadscsandinhibitedcellproliferationinvitroandratbonedefectmodels
AT yuxiwang atoh8expressioninhibitionpromotedosteogenicdifferentiationofadscsandinhibitedcellproliferationinvitroandratbonedefectmodels
AT zijunchen atoh8expressioninhibitionpromotedosteogenicdifferentiationofadscsandinhibitedcellproliferationinvitroandratbonedefectmodels
AT xuefengchen atoh8expressioninhibitionpromotedosteogenicdifferentiationofadscsandinhibitedcellproliferationinvitroandratbonedefectmodels
AT bangliandeng atoh8expressioninhibitionpromotedosteogenicdifferentiationofadscsandinhibitedcellproliferationinvitroandratbonedefectmodels
AT xiangdongliu atoh8expressioninhibitionpromotedosteogenicdifferentiationofadscsandinhibitedcellproliferationinvitroandratbonedefectmodels
AT zuolinjin atoh8expressioninhibitionpromotedosteogenicdifferentiationofadscsandinhibitedcellproliferationinvitroandratbonedefectmodels