Nitrogen‐Doped Multiwalled Carbon Nanotubes Trigger Immune Responses and Inhibit Fat Deposition

Abstract Multiwalled carbon nanotubes (MWCNTs) offer immense opportunities to deliver drugs and biomolecules to targeted tissues. However, it's unclear to their effects on fat metabolism. Here, it is demonstrated that nitrogen‐doped carboxylate‐functionalized MWCNTs (N‐MWCNTs) inhibit fat depos...

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Main Authors: Dalin He, Xue Xiao, Geng Hu, Wenqian Zhang, Guanliu Yu, Yan Liu, Yun Lin, Hai Lin, Xianyao Li, Youxiang Diao, Yi Tang, Haifang Li
Format: Article
Language:English
Published: Wiley-VCH 2024-12-01
Series:Advanced Materials Interfaces
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Online Access:https://doi.org/10.1002/admi.202400007
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author Dalin He
Xue Xiao
Geng Hu
Wenqian Zhang
Guanliu Yu
Yan Liu
Yun Lin
Hai Lin
Xianyao Li
Youxiang Diao
Yi Tang
Haifang Li
author_facet Dalin He
Xue Xiao
Geng Hu
Wenqian Zhang
Guanliu Yu
Yan Liu
Yun Lin
Hai Lin
Xianyao Li
Youxiang Diao
Yi Tang
Haifang Li
author_sort Dalin He
collection DOAJ
description Abstract Multiwalled carbon nanotubes (MWCNTs) offer immense opportunities to deliver drugs and biomolecules to targeted tissues. However, it's unclear to their effects on fat metabolism. Here, it is demonstrated that nitrogen‐doped carboxylate‐functionalized MWCNTs (N‐MWCNTs) inhibit fat deposition both in vivo and in vitro. N‐MWCNTs <0.5 µg mL−1 do not affect the viability of HEK293 cells and adipose‐derived stem cells (ASCs). Intramuscular administration of N‐MWCNTs does not affect the body weight gain and feed intake of mice, but reduces the fat mass. In in vitro‐cultured adipocytes, N‐MWCNTs suppress fat accumulation, accompanied by decreased and increased expression of adipogenic and lipolysis genes, respectively. Transcriptome analysis further certifies the N‐MWCNT alteration of fat metabolism‐related genes. Interestingly, the internalization of N‐MWCNTs by macrophage‐like cells via Transmission electron microscopy (TEM) imaging is observed. The mRNA sequencing data also shows remarkable variation of the genes involved in the Toll‐like receptors (TLRs) pathway, exhibiting down‐ or up‐regulation of inflammatory factors, of which TNF‐α, IL‐1, IL‐7, IL‐10, and IL‐12 are decreased, whereas IL‐6 and IL‐11 are increased. In conclusion, N‐MWCNTs trigger immune responses and reduction of fat deposition.
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spelling doaj-art-a480808832ae49b49d36930d6987f82a2025-08-20T02:52:27ZengWiley-VCHAdvanced Materials Interfaces2196-73502024-12-011136n/an/a10.1002/admi.202400007Nitrogen‐Doped Multiwalled Carbon Nanotubes Trigger Immune Responses and Inhibit Fat DepositionDalin He0Xue Xiao1Geng Hu2Wenqian Zhang3Guanliu Yu4Yan Liu5Yun Lin6Hai Lin7Xianyao Li8Youxiang Diao9Yi Tang10Haifang Li11College of Veterinary Medicine Shandong Agricultural University Tai'an 271018 ChinaCollege of Life Sciences Shandong Agricultural University Tai'an 271018 ChinaDepartment of Biotechnology Engineering Taishan Polytechnic College Tai'an 271018 ChinaCollege of Veterinary Medicine Shandong Agricultural University Tai'an 271018 ChinaCollege of Life Sciences Shandong Agricultural University Tai'an 271018 ChinaCollege of Life Sciences Shandong Agricultural University Tai'an 271018 ChinaCollege of Veterinary Medicine Shandong Agricultural University Tai'an 271018 ChinaCollege of Veterinary Medicine Shandong Agricultural University Tai'an 271018 ChinaCollege of Veterinary Medicine Shandong Agricultural University Tai'an 271018 ChinaCollege of Veterinary Medicine Shandong Agricultural University Tai'an 271018 ChinaCollege of Veterinary Medicine Shandong Agricultural University Tai'an 271018 ChinaCollege of Life Sciences Shandong Agricultural University Tai'an 271018 ChinaAbstract Multiwalled carbon nanotubes (MWCNTs) offer immense opportunities to deliver drugs and biomolecules to targeted tissues. However, it's unclear to their effects on fat metabolism. Here, it is demonstrated that nitrogen‐doped carboxylate‐functionalized MWCNTs (N‐MWCNTs) inhibit fat deposition both in vivo and in vitro. N‐MWCNTs <0.5 µg mL−1 do not affect the viability of HEK293 cells and adipose‐derived stem cells (ASCs). Intramuscular administration of N‐MWCNTs does not affect the body weight gain and feed intake of mice, but reduces the fat mass. In in vitro‐cultured adipocytes, N‐MWCNTs suppress fat accumulation, accompanied by decreased and increased expression of adipogenic and lipolysis genes, respectively. Transcriptome analysis further certifies the N‐MWCNT alteration of fat metabolism‐related genes. Interestingly, the internalization of N‐MWCNTs by macrophage‐like cells via Transmission electron microscopy (TEM) imaging is observed. The mRNA sequencing data also shows remarkable variation of the genes involved in the Toll‐like receptors (TLRs) pathway, exhibiting down‐ or up‐regulation of inflammatory factors, of which TNF‐α, IL‐1, IL‐7, IL‐10, and IL‐12 are decreased, whereas IL‐6 and IL‐11 are increased. In conclusion, N‐MWCNTs trigger immune responses and reduction of fat deposition.https://doi.org/10.1002/admi.202400007N‐MWCNTsfat depositionimmune responseTLRs pathway
spellingShingle Dalin He
Xue Xiao
Geng Hu
Wenqian Zhang
Guanliu Yu
Yan Liu
Yun Lin
Hai Lin
Xianyao Li
Youxiang Diao
Yi Tang
Haifang Li
Nitrogen‐Doped Multiwalled Carbon Nanotubes Trigger Immune Responses and Inhibit Fat Deposition
Advanced Materials Interfaces
N‐MWCNTs
fat deposition
immune response
TLRs pathway
title Nitrogen‐Doped Multiwalled Carbon Nanotubes Trigger Immune Responses and Inhibit Fat Deposition
title_full Nitrogen‐Doped Multiwalled Carbon Nanotubes Trigger Immune Responses and Inhibit Fat Deposition
title_fullStr Nitrogen‐Doped Multiwalled Carbon Nanotubes Trigger Immune Responses and Inhibit Fat Deposition
title_full_unstemmed Nitrogen‐Doped Multiwalled Carbon Nanotubes Trigger Immune Responses and Inhibit Fat Deposition
title_short Nitrogen‐Doped Multiwalled Carbon Nanotubes Trigger Immune Responses and Inhibit Fat Deposition
title_sort nitrogen doped multiwalled carbon nanotubes trigger immune responses and inhibit fat deposition
topic N‐MWCNTs
fat deposition
immune response
TLRs pathway
url https://doi.org/10.1002/admi.202400007
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