Kinetic changes in microglia-related retinal transcripts in experimental autoimmune uveoretinitis (EAU) of B10.RIII mice

Abstract In this study the retinal transcriptome was investigated during the development of experimental autoimmune uveoretinitis (EAU) in mice. EAU was induced by immunizing B10.RIII mice with human interphotoreceptor retinoid binding protein (hIRBP) 161–180 peptide. Genome-wide transcriptional pro...

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Main Authors: Maren Kasper, Marcus Karlstetter, Lena Wildschütz, Rebecca Scholz, Martin Busch, Dirk Bauer, Gerd Meyer zu Hörste, Solon Thanos, Thomas Langmann, Arnd Heiligenhaus
Format: Article
Language:English
Published: BMC 2025-02-01
Series:Journal of Neuroinflammation
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Online Access:https://doi.org/10.1186/s12974-025-03358-x
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author Maren Kasper
Marcus Karlstetter
Lena Wildschütz
Rebecca Scholz
Martin Busch
Dirk Bauer
Gerd Meyer zu Hörste
Solon Thanos
Thomas Langmann
Arnd Heiligenhaus
author_facet Maren Kasper
Marcus Karlstetter
Lena Wildschütz
Rebecca Scholz
Martin Busch
Dirk Bauer
Gerd Meyer zu Hörste
Solon Thanos
Thomas Langmann
Arnd Heiligenhaus
author_sort Maren Kasper
collection DOAJ
description Abstract In this study the retinal transcriptome was investigated during the development of experimental autoimmune uveoretinitis (EAU) in mice. EAU was induced by immunizing B10.RIII mice with human interphotoreceptor retinoid binding protein (hIRBP) 161–180 peptide. Genome-wide transcriptional profiles of EAU (day 7, 14 or 21 after immunization) and of control retinas were generated using DNA-microarrays and bioinformatic data mining. Microglia-associated transcripts were identified. Quantitative real-time polymerase chain reaction was performed to validate the expression of differentially expressed genes. Retinal transcript validation revealed that complement and interferon-related pathways, as well as gene clusters specific for antigen-processing and -presentation, and immunosuppression are involved during the course of the disease. Immunofluorescence analysis confirm that upregulated transcripts in EAU are also expressed by retinal microglia. Furthermore, the heterogenous expression patterns observed in retinal microglia, suggests the presence of different subpopulations of retinal microglia in EAU. This study expands our knowledge of the local immune processes involved in EAU pathology.
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issn 1742-2094
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publisher BMC
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series Journal of Neuroinflammation
spelling doaj-art-a38a0cb02039415eb6dfdfaa4550fc362025-08-20T02:48:16ZengBMCJournal of Neuroinflammation1742-20942025-02-0122111110.1186/s12974-025-03358-xKinetic changes in microglia-related retinal transcripts in experimental autoimmune uveoretinitis (EAU) of B10.RIII miceMaren Kasper0Marcus Karlstetter1Lena Wildschütz2Rebecca Scholz3Martin Busch4Dirk Bauer5Gerd Meyer zu Hörste6Solon Thanos7Thomas Langmann8Arnd Heiligenhaus9Department of Ophthalmology at St. Franziskus Hospital, Ophtha-LabChair of Experimental Immunology of the Eye, Department of Ophthalmology, University of CologneDepartment of Ophthalmology at St. Franziskus Hospital, Ophtha-LabChair of Experimental Immunology of the Eye, Department of Ophthalmology, University of CologneDepartment of Ophthalmology at St. Franziskus Hospital, Ophtha-LabDepartment of Ophthalmology at St. Franziskus Hospital, Ophtha-LabDepartment of Neurology, University Hospital MünsterInstitute for Experimental Ophthalmology, Westfalian Wilhelms-University of MünsterChair of Experimental Immunology of the Eye, Department of Ophthalmology, University of CologneDepartment of Ophthalmology at St. Franziskus Hospital, Ophtha-LabAbstract In this study the retinal transcriptome was investigated during the development of experimental autoimmune uveoretinitis (EAU) in mice. EAU was induced by immunizing B10.RIII mice with human interphotoreceptor retinoid binding protein (hIRBP) 161–180 peptide. Genome-wide transcriptional profiles of EAU (day 7, 14 or 21 after immunization) and of control retinas were generated using DNA-microarrays and bioinformatic data mining. Microglia-associated transcripts were identified. Quantitative real-time polymerase chain reaction was performed to validate the expression of differentially expressed genes. Retinal transcript validation revealed that complement and interferon-related pathways, as well as gene clusters specific for antigen-processing and -presentation, and immunosuppression are involved during the course of the disease. Immunofluorescence analysis confirm that upregulated transcripts in EAU are also expressed by retinal microglia. Furthermore, the heterogenous expression patterns observed in retinal microglia, suggests the presence of different subpopulations of retinal microglia in EAU. This study expands our knowledge of the local immune processes involved in EAU pathology.https://doi.org/10.1186/s12974-025-03358-xRetinal transcriptomeEAUAutoimmune uveitisMicroglia
spellingShingle Maren Kasper
Marcus Karlstetter
Lena Wildschütz
Rebecca Scholz
Martin Busch
Dirk Bauer
Gerd Meyer zu Hörste
Solon Thanos
Thomas Langmann
Arnd Heiligenhaus
Kinetic changes in microglia-related retinal transcripts in experimental autoimmune uveoretinitis (EAU) of B10.RIII mice
Journal of Neuroinflammation
Retinal transcriptome
EAU
Autoimmune uveitis
Microglia
title Kinetic changes in microglia-related retinal transcripts in experimental autoimmune uveoretinitis (EAU) of B10.RIII mice
title_full Kinetic changes in microglia-related retinal transcripts in experimental autoimmune uveoretinitis (EAU) of B10.RIII mice
title_fullStr Kinetic changes in microglia-related retinal transcripts in experimental autoimmune uveoretinitis (EAU) of B10.RIII mice
title_full_unstemmed Kinetic changes in microglia-related retinal transcripts in experimental autoimmune uveoretinitis (EAU) of B10.RIII mice
title_short Kinetic changes in microglia-related retinal transcripts in experimental autoimmune uveoretinitis (EAU) of B10.RIII mice
title_sort kinetic changes in microglia related retinal transcripts in experimental autoimmune uveoretinitis eau of b10 riii mice
topic Retinal transcriptome
EAU
Autoimmune uveitis
Microglia
url https://doi.org/10.1186/s12974-025-03358-x
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