A review on the relationship between the distal 1q21.1 microdeletion and schizophrenia

Schizophrenia is a complex neuropsychiatric disorder closely associated with genetic factors. Copy number variations (CNVs) play a key role in the genetic etiology of schizophrenia, with the distal 1q21.1 microdeletion identified as a rare CNV that serves as a significant genetic risk factor for the...

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Main Authors: Xinpeng Guo, Junrong Guo, Xijing Liu, Ting Hu
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-07-01
Series:Frontiers in Genetics
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Online Access:https://www.frontiersin.org/articles/10.3389/fgene.2025.1612654/full
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author Xinpeng Guo
Xinpeng Guo
Xinpeng Guo
Junrong Guo
Junrong Guo
Xijing Liu
Xijing Liu
Ting Hu
Ting Hu
author_facet Xinpeng Guo
Xinpeng Guo
Xinpeng Guo
Junrong Guo
Junrong Guo
Xijing Liu
Xijing Liu
Ting Hu
Ting Hu
author_sort Xinpeng Guo
collection DOAJ
description Schizophrenia is a complex neuropsychiatric disorder closely associated with genetic factors. Copy number variations (CNVs) play a key role in the genetic etiology of schizophrenia, with the distal 1q21.1 microdeletion identified as a rare CNV that serves as a significant genetic risk factor for the disorder. This microdeletion is found in 0.2%–0.6% of individuals with schizophrenia and is associated with an eightfold increased risk of developing the condition. The distal 1q21.1 region contains several schizophrenia risk genes, including PRKAB2, BCL9, CHD1L, GJA5, and GJA8. This review focuses on the roles of these five genes in brain function and explores their potential pathophysiological mechanisms in schizophrenia. By synthesizing current evidence, this review aims to deepen the understanding of schizophrenia by outlining its genetic architecture and molecular mechanisms, thereby providing a comprehensive framework for exploring disease pathogenesis.
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spelling doaj-art-a2255105b9e74a0cb3d1bf759fbac09e2025-08-20T02:48:54ZengFrontiers Media S.A.Frontiers in Genetics1664-80212025-07-011610.3389/fgene.2025.16126541612654A review on the relationship between the distal 1q21.1 microdeletion and schizophreniaXinpeng Guo0Xinpeng Guo1Xinpeng Guo2Junrong Guo3Junrong Guo4Xijing Liu5Xijing Liu6Ting Hu7Ting Hu8Department of Medical Genetics, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, ChinaWest China School of Medicine, Sichuan University, Chengdu, Sichuan, ChinaKey Laboratory of Birth Defects and Related Diseases of Women and Children (Sichuan University), Ministry of Education, Chengdu, Sichuan, ChinaDepartment of Medical Genetics, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, ChinaKey Laboratory of Birth Defects and Related Diseases of Women and Children (Sichuan University), Ministry of Education, Chengdu, Sichuan, ChinaDepartment of Medical Genetics, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, ChinaKey Laboratory of Birth Defects and Related Diseases of Women and Children (Sichuan University), Ministry of Education, Chengdu, Sichuan, ChinaDepartment of Medical Genetics, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, ChinaKey Laboratory of Birth Defects and Related Diseases of Women and Children (Sichuan University), Ministry of Education, Chengdu, Sichuan, ChinaSchizophrenia is a complex neuropsychiatric disorder closely associated with genetic factors. Copy number variations (CNVs) play a key role in the genetic etiology of schizophrenia, with the distal 1q21.1 microdeletion identified as a rare CNV that serves as a significant genetic risk factor for the disorder. This microdeletion is found in 0.2%–0.6% of individuals with schizophrenia and is associated with an eightfold increased risk of developing the condition. The distal 1q21.1 region contains several schizophrenia risk genes, including PRKAB2, BCL9, CHD1L, GJA5, and GJA8. This review focuses on the roles of these five genes in brain function and explores their potential pathophysiological mechanisms in schizophrenia. By synthesizing current evidence, this review aims to deepen the understanding of schizophrenia by outlining its genetic architecture and molecular mechanisms, thereby providing a comprehensive framework for exploring disease pathogenesis.https://www.frontiersin.org/articles/10.3389/fgene.2025.1612654/fullschizophreniadistal 1q21.1 microdeletionPRKAB2BCL9CHD1L
spellingShingle Xinpeng Guo
Xinpeng Guo
Xinpeng Guo
Junrong Guo
Junrong Guo
Xijing Liu
Xijing Liu
Ting Hu
Ting Hu
A review on the relationship between the distal 1q21.1 microdeletion and schizophrenia
Frontiers in Genetics
schizophrenia
distal 1q21.1 microdeletion
PRKAB2
BCL9
CHD1L
title A review on the relationship between the distal 1q21.1 microdeletion and schizophrenia
title_full A review on the relationship between the distal 1q21.1 microdeletion and schizophrenia
title_fullStr A review on the relationship between the distal 1q21.1 microdeletion and schizophrenia
title_full_unstemmed A review on the relationship between the distal 1q21.1 microdeletion and schizophrenia
title_short A review on the relationship between the distal 1q21.1 microdeletion and schizophrenia
title_sort review on the relationship between the distal 1q21 1 microdeletion and schizophrenia
topic schizophrenia
distal 1q21.1 microdeletion
PRKAB2
BCL9
CHD1L
url https://www.frontiersin.org/articles/10.3389/fgene.2025.1612654/full
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