The regulatory role of ACP5 in the diesel exhaust particle-induced AHR inflammatory signaling pathway in a human bronchial epithelial cell line

The regulatory role of ACP5 in the diesel exhaust particle-induced AHR inflammatory signaling pathway in a human bronchial epithelial cell line

Abstract Exposure to diesel exhaust particles (DEPs), which are major constituents of urban air pollution, is associated with adverse health outcomes. Previous studies have shown that DEPs enhance the expression of pro-inflammatory cytokines and immune responses. However, few studies have focused on...

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Main Authors: Aaron Yu, Dankyu Yoon, Hye Bin An, Uimook Choi, Hye-Ryeon Heo, Dong-Hoon Chae, Hyun Sung Park, Jae Han Park, Myoung Hee Han, Jiyoung Heo, Keonwoo Cho, Ki-Sun Park, Hee Min Yoo, Hyung-Sik Kim, Kyung-Sun Kang, Mi-Kyung Oh, Hyun Joung Lim, Kyung-Rok Yu
Format: Article
Language:English
Published: Nature Portfolio 2025-03-01
Series:Scientific Reports
Subjects:
ACP5
Diesel exhaust particles
BEAS-2B
Aryl hydrocarbon receptor
Inflammatory response
Online Access:https://doi.org/10.1038/s41598-024-84280-9
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