Common genetic variation near the phospholamban gene is associated with cardiac repolarisation: meta-analysis of three genome-wide association studies.

To identify loci affecting the electrocardiographic QT interval, a measure of cardiac repolarisation associated with risk of ventricular arrhythmias and sudden cardiac death, we conducted a meta-analysis of three genome-wide association studies (GWAS) including 3,558 subjects from the TwinsUK and BR...

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Main Authors: Ilja M Nolte, Chris Wallace, Stephen J Newhouse, Daryl Waggott, Jingyuan Fu, Nicole Soranzo, Rhian Gwilliam, Panos Deloukas, Irina Savelieva, Dongling Zheng, Chrysoula Dalageorgou, Martin Farrall, Nilesh J Samani, John Connell, Morris Brown, Anna Dominiczak, Mark Lathrop, Eleftheria Zeggini, Louise V Wain, Wellcome Trust Case Control Consortium, DCCT/EDIC Research Group, Christopher Newton-Cheh, Mark Eijgelsheim, Kenneth Rice, Paul I W de Bakker, QTGEN consortium, Arne Pfeufer, Serena Sanna, Dan E Arking, QTSCD consortium, Folkert W Asselbergs, Tim D Spector, Nicholas D Carter, Steve Jeffery, Martin Tobin, Mark Caulfield, Harold Snieder, Andrew D Paterson, Patricia B Munroe, Yalda Jamshidi
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Language:English
Published: Public Library of Science (PLoS) 2009-07-01
Series:PLoS ONE
Online Access:https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0006138&type=printable
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author Ilja M Nolte
Chris Wallace
Stephen J Newhouse
Daryl Waggott
Jingyuan Fu
Nicole Soranzo
Rhian Gwilliam
Panos Deloukas
Irina Savelieva
Dongling Zheng
Chrysoula Dalageorgou
Martin Farrall
Nilesh J Samani
John Connell
Morris Brown
Anna Dominiczak
Mark Lathrop
Eleftheria Zeggini
Louise V Wain
Wellcome Trust Case Control Consortium
DCCT/EDIC Research Group
Christopher Newton-Cheh
Mark Eijgelsheim
Kenneth Rice
Paul I W de Bakker
QTGEN consortium
Arne Pfeufer
Serena Sanna
Dan E Arking
QTSCD consortium
Folkert W Asselbergs
Tim D Spector
Nicholas D Carter
Steve Jeffery
Martin Tobin
Mark Caulfield
Harold Snieder
Andrew D Paterson
Patricia B Munroe
Yalda Jamshidi
author_facet Ilja M Nolte
Chris Wallace
Stephen J Newhouse
Daryl Waggott
Jingyuan Fu
Nicole Soranzo
Rhian Gwilliam
Panos Deloukas
Irina Savelieva
Dongling Zheng
Chrysoula Dalageorgou
Martin Farrall
Nilesh J Samani
John Connell
Morris Brown
Anna Dominiczak
Mark Lathrop
Eleftheria Zeggini
Louise V Wain
Wellcome Trust Case Control Consortium
DCCT/EDIC Research Group
Christopher Newton-Cheh
Mark Eijgelsheim
Kenneth Rice
Paul I W de Bakker
QTGEN consortium
Arne Pfeufer
Serena Sanna
Dan E Arking
QTSCD consortium
Folkert W Asselbergs
Tim D Spector
Nicholas D Carter
Steve Jeffery
Martin Tobin
Mark Caulfield
Harold Snieder
Andrew D Paterson
Patricia B Munroe
Yalda Jamshidi
author_sort Ilja M Nolte
collection DOAJ
description To identify loci affecting the electrocardiographic QT interval, a measure of cardiac repolarisation associated with risk of ventricular arrhythmias and sudden cardiac death, we conducted a meta-analysis of three genome-wide association studies (GWAS) including 3,558 subjects from the TwinsUK and BRIGHT cohorts in the UK and the DCCT/EDIC cohort from North America. Five loci were significantly associated with QT interval at P<1x10(-6). To validate these findings we performed an in silico comparison with data from two QT consortia: QTSCD (n = 15,842) and QTGEN (n = 13,685). Analysis confirmed the association between common variants near NOS1AP (P = 1.4x10(-83)) and the phospholamban (PLN) gene (P = 1.9x10(-29)). The most associated SNP near NOS1AP (rs12143842) explains 0.82% variance; the SNP near PLN (rs11153730) explains 0.74% variance of QT interval duration. We found no evidence for interaction between these two SNPs (P = 0.99). PLN is a key regulator of cardiac diastolic function and is involved in regulating intracellular calcium cycling, it has only recently been identified as a susceptibility locus for QT interval. These data offer further mechanistic insights into genetic influence on the QT interval which may predispose to life threatening arrhythmias and sudden cardiac death.
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spelling doaj-art-a075bafac4a04e268a97868576cece992025-08-20T03:22:38ZengPublic Library of Science (PLoS)PLoS ONE1932-62032009-07-0147e613810.1371/journal.pone.0006138Common genetic variation near the phospholamban gene is associated with cardiac repolarisation: meta-analysis of three genome-wide association studies.Ilja M NolteChris WallaceStephen J NewhouseDaryl WaggottJingyuan FuNicole SoranzoRhian GwilliamPanos DeloukasIrina SavelievaDongling ZhengChrysoula DalageorgouMartin FarrallNilesh J SamaniJohn ConnellMorris BrownAnna DominiczakMark LathropEleftheria ZegginiLouise V WainWellcome Trust Case Control ConsortiumDCCT/EDIC Research GroupChristopher Newton-ChehMark EijgelsheimKenneth RicePaul I W de BakkerQTGEN consortiumArne PfeuferSerena SannaDan E ArkingQTSCD consortiumFolkert W AsselbergsTim D SpectorNicholas D CarterSteve JefferyMartin TobinMark CaulfieldHarold SniederAndrew D PatersonPatricia B MunroeYalda JamshidiTo identify loci affecting the electrocardiographic QT interval, a measure of cardiac repolarisation associated with risk of ventricular arrhythmias and sudden cardiac death, we conducted a meta-analysis of three genome-wide association studies (GWAS) including 3,558 subjects from the TwinsUK and BRIGHT cohorts in the UK and the DCCT/EDIC cohort from North America. Five loci were significantly associated with QT interval at P<1x10(-6). To validate these findings we performed an in silico comparison with data from two QT consortia: QTSCD (n = 15,842) and QTGEN (n = 13,685). Analysis confirmed the association between common variants near NOS1AP (P = 1.4x10(-83)) and the phospholamban (PLN) gene (P = 1.9x10(-29)). The most associated SNP near NOS1AP (rs12143842) explains 0.82% variance; the SNP near PLN (rs11153730) explains 0.74% variance of QT interval duration. We found no evidence for interaction between these two SNPs (P = 0.99). PLN is a key regulator of cardiac diastolic function and is involved in regulating intracellular calcium cycling, it has only recently been identified as a susceptibility locus for QT interval. These data offer further mechanistic insights into genetic influence on the QT interval which may predispose to life threatening arrhythmias and sudden cardiac death.https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0006138&type=printable
spellingShingle Ilja M Nolte
Chris Wallace
Stephen J Newhouse
Daryl Waggott
Jingyuan Fu
Nicole Soranzo
Rhian Gwilliam
Panos Deloukas
Irina Savelieva
Dongling Zheng
Chrysoula Dalageorgou
Martin Farrall
Nilesh J Samani
John Connell
Morris Brown
Anna Dominiczak
Mark Lathrop
Eleftheria Zeggini
Louise V Wain
Wellcome Trust Case Control Consortium
DCCT/EDIC Research Group
Christopher Newton-Cheh
Mark Eijgelsheim
Kenneth Rice
Paul I W de Bakker
QTGEN consortium
Arne Pfeufer
Serena Sanna
Dan E Arking
QTSCD consortium
Folkert W Asselbergs
Tim D Spector
Nicholas D Carter
Steve Jeffery
Martin Tobin
Mark Caulfield
Harold Snieder
Andrew D Paterson
Patricia B Munroe
Yalda Jamshidi
Common genetic variation near the phospholamban gene is associated with cardiac repolarisation: meta-analysis of three genome-wide association studies.
PLoS ONE
title Common genetic variation near the phospholamban gene is associated with cardiac repolarisation: meta-analysis of three genome-wide association studies.
title_full Common genetic variation near the phospholamban gene is associated with cardiac repolarisation: meta-analysis of three genome-wide association studies.
title_fullStr Common genetic variation near the phospholamban gene is associated with cardiac repolarisation: meta-analysis of three genome-wide association studies.
title_full_unstemmed Common genetic variation near the phospholamban gene is associated with cardiac repolarisation: meta-analysis of three genome-wide association studies.
title_short Common genetic variation near the phospholamban gene is associated with cardiac repolarisation: meta-analysis of three genome-wide association studies.
title_sort common genetic variation near the phospholamban gene is associated with cardiac repolarisation meta analysis of three genome wide association studies
url https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0006138&type=printable
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