Inhibitory effect of luteolin on the metabolism of vandetanib in vivo and in vitro

This study aimed to examine the potential drug-drug interaction (DDI) between vandetanib and luteolin in vivo and in vitro, with the objective of establishing a scientific foundation for their appropriate utilization in clinical settings. Sprague-Dawley (SD) rats were randomly divided into two group...

Full description

Saved in:
Bibliographic Details
Main Authors: Yuxin Shen, Fengsheng Hong, Hualu Wu, Xiaohai Chen, Hailun Xia, Ren-ai Xu, Guanyang Lin, Lu Shi
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-03-01
Series:Frontiers in Pharmacology
Subjects:
Online Access:https://www.frontiersin.org/articles/10.3389/fphar.2025.1526159/full
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850239699139952640
author Yuxin Shen
Yuxin Shen
Fengsheng Hong
Hualu Wu
Xiaohai Chen
Hailun Xia
Ren-ai Xu
Ren-ai Xu
Guanyang Lin
Lu Shi
Lu Shi
author_facet Yuxin Shen
Yuxin Shen
Fengsheng Hong
Hualu Wu
Xiaohai Chen
Hailun Xia
Ren-ai Xu
Ren-ai Xu
Guanyang Lin
Lu Shi
Lu Shi
author_sort Yuxin Shen
collection DOAJ
description This study aimed to examine the potential drug-drug interaction (DDI) between vandetanib and luteolin in vivo and in vitro, with the objective of establishing a scientific foundation for their appropriate utilization in clinical settings. Sprague-Dawley (SD) rats were randomly divided into two groups: a control group (vandetanib administered by gavage alone) and an experimental group (vandetanib and luteolin administered together). A series of blood samples were collected at different time intervals. The plasma concentrations of vandetanib and its metabolite N-demethyl vandetanib in rats were determined using an ultra performance liquid chromatography tandem mass spectrometry (UPLC-MS/MS). Incubation systems were set up with rat liver microsomes (RLM) and human liver microsomes (HLM) to measure the Michaelis-Menten constant (Km) and half-maximum inhibitory concentration (IC50) values. Additionally, the inhibitory mechanism of luteolin on vandetanib was also investigated. Ultimately, the molecular mechanism of inhibition was examined through the utilization of molecular docking techniques. In vivo animal experiment results showed that compared with the control group, the AUC(0-t) and Cmax of vandetanib in the experimental group were significantly increased. The findings from the in vitro experiments revealed that luteolin exhibited a moderate inhibitory effect on the metabolism of vandetanib. The IC50 values for RLM and HLM were determined to be 8.56 μM and 15.84 μM, respectively. The identified inhibition mechanism was classified as mixed. This study utilized molecular docking analysis to provide additional evidence supporting the competitive inhibition of luteolin on vandetanib in CYP3A4. The data presented in our study indicated a potential interaction between vandetanib and luteolin, which may necessitate the need for dose adjustment during their co-administration in clinical settings.
format Article
id doaj-art-a04e19f28be247cd834ad02a04ddc8af
institution OA Journals
issn 1663-9812
language English
publishDate 2025-03-01
publisher Frontiers Media S.A.
record_format Article
series Frontiers in Pharmacology
spelling doaj-art-a04e19f28be247cd834ad02a04ddc8af2025-08-20T02:01:05ZengFrontiers Media S.A.Frontiers in Pharmacology1663-98122025-03-011610.3389/fphar.2025.15261591526159Inhibitory effect of luteolin on the metabolism of vandetanib in vivo and in vitroYuxin Shen0Yuxin Shen1Fengsheng Hong2Hualu Wu3Xiaohai Chen4Hailun Xia5Ren-ai Xu6Ren-ai Xu7Guanyang Lin8Lu Shi9Lu Shi10The First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, ChinaZhejiang Key Laboratory of Intelligent Cancer Biomarker Discovery and Translation, First Affiliated Hospital, Wenzhou Medical University, Wenzhou, Zhejiang, ChinaThe First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, ChinaThe First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, ChinaThe First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, ChinaThe First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, ChinaThe First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, ChinaZhejiang Key Laboratory of Intelligent Cancer Biomarker Discovery and Translation, First Affiliated Hospital, Wenzhou Medical University, Wenzhou, Zhejiang, ChinaThe First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, ChinaThe First Affiliated Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, ChinaZhejiang Key Laboratory of Intelligent Cancer Biomarker Discovery and Translation, First Affiliated Hospital, Wenzhou Medical University, Wenzhou, Zhejiang, ChinaThis study aimed to examine the potential drug-drug interaction (DDI) between vandetanib and luteolin in vivo and in vitro, with the objective of establishing a scientific foundation for their appropriate utilization in clinical settings. Sprague-Dawley (SD) rats were randomly divided into two groups: a control group (vandetanib administered by gavage alone) and an experimental group (vandetanib and luteolin administered together). A series of blood samples were collected at different time intervals. The plasma concentrations of vandetanib and its metabolite N-demethyl vandetanib in rats were determined using an ultra performance liquid chromatography tandem mass spectrometry (UPLC-MS/MS). Incubation systems were set up with rat liver microsomes (RLM) and human liver microsomes (HLM) to measure the Michaelis-Menten constant (Km) and half-maximum inhibitory concentration (IC50) values. Additionally, the inhibitory mechanism of luteolin on vandetanib was also investigated. Ultimately, the molecular mechanism of inhibition was examined through the utilization of molecular docking techniques. In vivo animal experiment results showed that compared with the control group, the AUC(0-t) and Cmax of vandetanib in the experimental group were significantly increased. The findings from the in vitro experiments revealed that luteolin exhibited a moderate inhibitory effect on the metabolism of vandetanib. The IC50 values for RLM and HLM were determined to be 8.56 μM and 15.84 μM, respectively. The identified inhibition mechanism was classified as mixed. This study utilized molecular docking analysis to provide additional evidence supporting the competitive inhibition of luteolin on vandetanib in CYP3A4. The data presented in our study indicated a potential interaction between vandetanib and luteolin, which may necessitate the need for dose adjustment during their co-administration in clinical settings.https://www.frontiersin.org/articles/10.3389/fphar.2025.1526159/fullvandetanibluteolindrug-drug interactionUPLC-MS/MSmolecular docking
spellingShingle Yuxin Shen
Yuxin Shen
Fengsheng Hong
Hualu Wu
Xiaohai Chen
Hailun Xia
Ren-ai Xu
Ren-ai Xu
Guanyang Lin
Lu Shi
Lu Shi
Inhibitory effect of luteolin on the metabolism of vandetanib in vivo and in vitro
Frontiers in Pharmacology
vandetanib
luteolin
drug-drug interaction
UPLC-MS/MS
molecular docking
title Inhibitory effect of luteolin on the metabolism of vandetanib in vivo and in vitro
title_full Inhibitory effect of luteolin on the metabolism of vandetanib in vivo and in vitro
title_fullStr Inhibitory effect of luteolin on the metabolism of vandetanib in vivo and in vitro
title_full_unstemmed Inhibitory effect of luteolin on the metabolism of vandetanib in vivo and in vitro
title_short Inhibitory effect of luteolin on the metabolism of vandetanib in vivo and in vitro
title_sort inhibitory effect of luteolin on the metabolism of vandetanib in vivo and in vitro
topic vandetanib
luteolin
drug-drug interaction
UPLC-MS/MS
molecular docking
url https://www.frontiersin.org/articles/10.3389/fphar.2025.1526159/full
work_keys_str_mv AT yuxinshen inhibitoryeffectofluteolinonthemetabolismofvandetanibinvivoandinvitro
AT yuxinshen inhibitoryeffectofluteolinonthemetabolismofvandetanibinvivoandinvitro
AT fengshenghong inhibitoryeffectofluteolinonthemetabolismofvandetanibinvivoandinvitro
AT hualuwu inhibitoryeffectofluteolinonthemetabolismofvandetanibinvivoandinvitro
AT xiaohaichen inhibitoryeffectofluteolinonthemetabolismofvandetanibinvivoandinvitro
AT hailunxia inhibitoryeffectofluteolinonthemetabolismofvandetanibinvivoandinvitro
AT renaixu inhibitoryeffectofluteolinonthemetabolismofvandetanibinvivoandinvitro
AT renaixu inhibitoryeffectofluteolinonthemetabolismofvandetanibinvivoandinvitro
AT guanyanglin inhibitoryeffectofluteolinonthemetabolismofvandetanibinvivoandinvitro
AT lushi inhibitoryeffectofluteolinonthemetabolismofvandetanibinvivoandinvitro
AT lushi inhibitoryeffectofluteolinonthemetabolismofvandetanibinvivoandinvitro