Angiotensin receptor-neprilysin inhibition by sacubitril/valsartan attenuates doxorubicin-induced cardiotoxicity in a pretreatment mice model by interfering with oxidative stress, inflammation, and Caspase 3 apoptotic pathway

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Main Authors: Ferhat Dindaş, Hüseyin Güngör, Mehmet Ekici, Pınar Akokay, Füsun Erhan, Mustafa Doğduş, Mehmet Birhan Yılmaz
Format: Article
Language:English
Published: KARE Publishing 2021-11-01
Series:Anatolian Journal of Cardiology
Online Access:https://anatoljcardiol.com/jvi.aspx?un=AJC-28709&volume=25&issue=11
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author Ferhat Dindaş
Hüseyin Güngör
Mehmet Ekici
Pınar Akokay
Füsun Erhan
Mustafa Doğduş
Mehmet Birhan Yılmaz
author_facet Ferhat Dindaş
Hüseyin Güngör
Mehmet Ekici
Pınar Akokay
Füsun Erhan
Mustafa Doğduş
Mehmet Birhan Yılmaz
author_sort Ferhat Dindaş
collection DOAJ
format Article
id doaj-art-a011776f59644852a2964dfe06d69ce9
institution OA Journals
issn 2149-2263
2149-2271
language English
publishDate 2021-11-01
publisher KARE Publishing
record_format Article
series Anatolian Journal of Cardiology
spelling doaj-art-a011776f59644852a2964dfe06d69ce92025-08-20T02:00:39ZengKARE PublishingAnatolian Journal of Cardiology2149-22632149-22712021-11-01251182182810.5152/AnatolJCardiol.2021.356Angiotensin receptor-neprilysin inhibition by sacubitril/valsartan attenuates doxorubicin-induced cardiotoxicity in a pretreatment mice model by interfering with oxidative stress, inflammation, and Caspase 3 apoptotic pathwayFerhat Dindaş0Hüseyin Güngör1Mehmet Ekici2Pınar Akokay3Füsun Erhan4Mustafa Doğduş5Mehmet Birhan Yılmaz6Department of Cardiology, Uşak Training and Research Hospital; Uşak-TurkeyDepartment of Pharmacology, Faculty of Veterinary, Cumhuriyet University; Sivas-TurkeyDepartment of Physiology, Faculty of Veterinary, Cumhuriyet University; Sivas-TurkeyDepartment of Histology, İzmir Kavram Vocational School; İzmir-TurkeyDepartment of Histology, Faculty of Veterinary, Cumhuriyet University; Sivas-TurkeyDepartment of Cardiology, Uşak Training and Research Hospital; Uşak-TurkeyDepartment of Cardiology, Faculty of Medicine, Dokuz Eylül University; İzmir-Turkeyhttps://anatoljcardiol.com/jvi.aspx?un=AJC-28709&volume=25&issue=11
spellingShingle Ferhat Dindaş
Hüseyin Güngör
Mehmet Ekici
Pınar Akokay
Füsun Erhan
Mustafa Doğduş
Mehmet Birhan Yılmaz
Angiotensin receptor-neprilysin inhibition by sacubitril/valsartan attenuates doxorubicin-induced cardiotoxicity in a pretreatment mice model by interfering with oxidative stress, inflammation, and Caspase 3 apoptotic pathway
Anatolian Journal of Cardiology
title Angiotensin receptor-neprilysin inhibition by sacubitril/valsartan attenuates doxorubicin-induced cardiotoxicity in a pretreatment mice model by interfering with oxidative stress, inflammation, and Caspase 3 apoptotic pathway
title_full Angiotensin receptor-neprilysin inhibition by sacubitril/valsartan attenuates doxorubicin-induced cardiotoxicity in a pretreatment mice model by interfering with oxidative stress, inflammation, and Caspase 3 apoptotic pathway
title_fullStr Angiotensin receptor-neprilysin inhibition by sacubitril/valsartan attenuates doxorubicin-induced cardiotoxicity in a pretreatment mice model by interfering with oxidative stress, inflammation, and Caspase 3 apoptotic pathway
title_full_unstemmed Angiotensin receptor-neprilysin inhibition by sacubitril/valsartan attenuates doxorubicin-induced cardiotoxicity in a pretreatment mice model by interfering with oxidative stress, inflammation, and Caspase 3 apoptotic pathway
title_short Angiotensin receptor-neprilysin inhibition by sacubitril/valsartan attenuates doxorubicin-induced cardiotoxicity in a pretreatment mice model by interfering with oxidative stress, inflammation, and Caspase 3 apoptotic pathway
title_sort angiotensin receptor neprilysin inhibition by sacubitril valsartan attenuates doxorubicin induced cardiotoxicity in a pretreatment mice model by interfering with oxidative stress inflammation and caspase 3 apoptotic pathway
url https://anatoljcardiol.com/jvi.aspx?un=AJC-28709&volume=25&issue=11
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AT huseyingungor angiotensinreceptorneprilysininhibitionbysacubitrilvalsartanattenuatesdoxorubicininducedcardiotoxicityinapretreatmentmicemodelbyinterferingwithoxidativestressinflammationandcaspase3apoptoticpathway
AT mehmetekici angiotensinreceptorneprilysininhibitionbysacubitrilvalsartanattenuatesdoxorubicininducedcardiotoxicityinapretreatmentmicemodelbyinterferingwithoxidativestressinflammationandcaspase3apoptoticpathway
AT pınarakokay angiotensinreceptorneprilysininhibitionbysacubitrilvalsartanattenuatesdoxorubicininducedcardiotoxicityinapretreatmentmicemodelbyinterferingwithoxidativestressinflammationandcaspase3apoptoticpathway
AT fusunerhan angiotensinreceptorneprilysininhibitionbysacubitrilvalsartanattenuatesdoxorubicininducedcardiotoxicityinapretreatmentmicemodelbyinterferingwithoxidativestressinflammationandcaspase3apoptoticpathway
AT mustafadogdus angiotensinreceptorneprilysininhibitionbysacubitrilvalsartanattenuatesdoxorubicininducedcardiotoxicityinapretreatmentmicemodelbyinterferingwithoxidativestressinflammationandcaspase3apoptoticpathway
AT mehmetbirhanyılmaz angiotensinreceptorneprilysininhibitionbysacubitrilvalsartanattenuatesdoxorubicininducedcardiotoxicityinapretreatmentmicemodelbyinterferingwithoxidativestressinflammationandcaspase3apoptoticpathway