Targeting NEDD8 in pediatric acute myeloid leukemia: an integrated bioinformatics and experimental approach

This study systematically explored the role of NEDD8 in pediatric acute myeloid leukemia (AML) through patient sample analysis, database mining, and in vitro experiments. Our results demonstrated that NEDD8 was significantly overexpressed in newly diagnosed pediatric AML patients and was associated...

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Main Authors: Jian Sun, Cui Liu, Guangli Yang, Qian Li, Yang An, Yin Zhu, Pingping Zhang, Yaning Guan, Chang Peng, Zuochen Du, Pei Huang, Yan Chen
Format: Article
Language:English
Published: Taylor & Francis Group 2025-12-01
Series:Hematology
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Online Access:https://www.tandfonline.com/doi/10.1080/16078454.2025.2478650
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author Jian Sun
Cui Liu
Guangli Yang
Qian Li
Yang An
Yin Zhu
Pingping Zhang
Yaning Guan
Chang Peng
Zuochen Du
Pei Huang
Yan Chen
author_facet Jian Sun
Cui Liu
Guangli Yang
Qian Li
Yang An
Yin Zhu
Pingping Zhang
Yaning Guan
Chang Peng
Zuochen Du
Pei Huang
Yan Chen
author_sort Jian Sun
collection DOAJ
description This study systematically explored the role of NEDD8 in pediatric acute myeloid leukemia (AML) through patient sample analysis, database mining, and in vitro experiments. Our results demonstrated that NEDD8 was significantly overexpressed in newly diagnosed pediatric AML patients and was associated with poor survival outcomes. Functional enrichment analysis of the TARGET database further revealed a strong correlation between NEDD8 and cancer-related pathways. In vitro experiments showed that NEDD8 knockdown significantly inhibited the proliferation of AML cells (THP-1 and MV4-11) and induced cell cycle arrest. Collectively, these findings highlight the critical role of NEDD8 in pediatric AML pathogenesis and suggest its potential as both a prognostic biomarker and a therapeutic target.
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publisher Taylor & Francis Group
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series Hematology
spelling doaj-art-9fd8dd6fb766470ca91592afb1efa73c2025-08-20T02:07:16ZengTaylor & Francis GroupHematology1607-84542025-12-0130110.1080/16078454.2025.2478650Targeting NEDD8 in pediatric acute myeloid leukemia: an integrated bioinformatics and experimental approachJian Sun0Cui Liu1Guangli Yang2Qian Li3Yang An4Yin Zhu5Pingping Zhang6Yaning Guan7Chang Peng8Zuochen Du9Pei Huang10Yan Chen11Department of Pediatrics, Affiliated Hospital of Zunyi Medical University, Zunyi, People’s Republic of ChinaDepartment of Pediatrics, Affiliated Hospital of Zunyi Medical University, Zunyi, People’s Republic of ChinaDepartment of Pediatrics, Affiliated Hospital of Zunyi Medical University, Zunyi, People’s Republic of ChinaDepartment of Pediatrics, Affiliated Hospital of Zunyi Medical University, Zunyi, People’s Republic of ChinaDepartment of Pediatrics, Affiliated Hospital of Zunyi Medical University, Zunyi, People’s Republic of ChinaDepartment of Pediatrics, Affiliated Hospital of Zunyi Medical University, Zunyi, People’s Republic of ChinaDepartment of Pediatrics, Affiliated Hospital of Zunyi Medical University, Zunyi, People’s Republic of ChinaDepartment of Pediatrics, Affiliated Hospital of Zunyi Medical University, Zunyi, People’s Republic of ChinaDepartment of Pediatrics, Affiliated Hospital of Zunyi Medical University, Zunyi, People’s Republic of ChinaDepartment of Pediatrics, Affiliated Hospital of Zunyi Medical University, Zunyi, People’s Republic of ChinaDepartment of Pediatrics, Affiliated Hospital of Zunyi Medical University, Zunyi, People’s Republic of ChinaDepartment of Pediatrics, Affiliated Hospital of Zunyi Medical University, Zunyi, People’s Republic of ChinaThis study systematically explored the role of NEDD8 in pediatric acute myeloid leukemia (AML) through patient sample analysis, database mining, and in vitro experiments. Our results demonstrated that NEDD8 was significantly overexpressed in newly diagnosed pediatric AML patients and was associated with poor survival outcomes. Functional enrichment analysis of the TARGET database further revealed a strong correlation between NEDD8 and cancer-related pathways. In vitro experiments showed that NEDD8 knockdown significantly inhibited the proliferation of AML cells (THP-1 and MV4-11) and induced cell cycle arrest. Collectively, these findings highlight the critical role of NEDD8 in pediatric AML pathogenesis and suggest its potential as both a prognostic biomarker and a therapeutic target.https://www.tandfonline.com/doi/10.1080/16078454.2025.2478650Acute myeloid leukemiapediatricNEDD8prognosiscell cycle
spellingShingle Jian Sun
Cui Liu
Guangli Yang
Qian Li
Yang An
Yin Zhu
Pingping Zhang
Yaning Guan
Chang Peng
Zuochen Du
Pei Huang
Yan Chen
Targeting NEDD8 in pediatric acute myeloid leukemia: an integrated bioinformatics and experimental approach
Hematology
Acute myeloid leukemia
pediatric
NEDD8
prognosis
cell cycle
title Targeting NEDD8 in pediatric acute myeloid leukemia: an integrated bioinformatics and experimental approach
title_full Targeting NEDD8 in pediatric acute myeloid leukemia: an integrated bioinformatics and experimental approach
title_fullStr Targeting NEDD8 in pediatric acute myeloid leukemia: an integrated bioinformatics and experimental approach
title_full_unstemmed Targeting NEDD8 in pediatric acute myeloid leukemia: an integrated bioinformatics and experimental approach
title_short Targeting NEDD8 in pediatric acute myeloid leukemia: an integrated bioinformatics and experimental approach
title_sort targeting nedd8 in pediatric acute myeloid leukemia an integrated bioinformatics and experimental approach
topic Acute myeloid leukemia
pediatric
NEDD8
prognosis
cell cycle
url https://www.tandfonline.com/doi/10.1080/16078454.2025.2478650
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