Human Brain Organoids: A New Model to Study <i>Cryptococcus neoformans</i> Neurotropism

With the rise in immunocompromised individuals and patients with immune-related comorbidities such as COVID-19, the rate of fungal infections is growing. This increase, along with the current plateau in antifungal drug development, has made understanding the pathogenesis and dissemination of these o...

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Main Authors: Alfred T. Harding, Lee Gehrke, Jatin M. Vyas, Hannah Brown Harding
Format: Article
Language:English
Published: MDPI AG 2025-07-01
Series:Journal of Fungi
Subjects:
Online Access:https://www.mdpi.com/2309-608X/11/7/539
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author Alfred T. Harding
Lee Gehrke
Jatin M. Vyas
Hannah Brown Harding
author_facet Alfred T. Harding
Lee Gehrke
Jatin M. Vyas
Hannah Brown Harding
author_sort Alfred T. Harding
collection DOAJ
description With the rise in immunocompromised individuals and patients with immune-related comorbidities such as COVID-19, the rate of fungal infections is growing. This increase, along with the current plateau in antifungal drug development, has made understanding the pathogenesis and dissemination of these organisms more pertinent than ever. The mouse model of fungal infection, while informative on a basic scientific level, has severe limitations in terms of translation to the human disease. Here we present data supporting the implementation of the human cerebral organoid model, which is generated from human embryonic stem cells and accurately recapitulates relevant brain cell types and structures, to study fungal infection and dissemination to the central nervous system (CNS). This approach provides direct insight into the relevant pathogenesis of specific fungal organisms in human tissues where in vivo models are impossible. With this model system we assessed the specific brain tropisms and cellular effects of fungal pathogens known to cross the blood–brain barrier (BBB), such as <i>Cryptococcus neoformans</i>. We determined the effects of this fungal pathogen on the overall gross morphology, cellular architecture, and cytokine release from these model organoids. Furthermore, we demonstrated that <i>C. neoformans</i> penetrates and invades the organoid tissue and remains present throughout the course of infection. These results demonstrate the utility of this new model to the field and highlight the potential for this system to elucidate fungal pathogenesis to develop new therapeutic strategies to prevent and treat the disseminated stages of fungal diseases such as cryptococcal meningitis.
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spelling doaj-art-9f909fd9aa564b0d9a646a99f1d32bd22025-08-20T02:45:56ZengMDPI AGJournal of Fungi2309-608X2025-07-0111753910.3390/jof11070539Human Brain Organoids: A New Model to Study <i>Cryptococcus neoformans</i> NeurotropismAlfred T. Harding0Lee Gehrke1Jatin M. Vyas2Hannah Brown Harding3Institute for Medical Engineering and Science, Massachusetts Institute of Technology, Cambridge, MA 02139, USAInstitute for Medical Engineering and Science, Massachusetts Institute of Technology, Cambridge, MA 02139, USADivision of Infectious Diseases, Department of Medicine, Massachusetts General Hospital, Boston, MA 02114, USADivision of Infectious Diseases, Department of Medicine, Massachusetts General Hospital, Boston, MA 02114, USAWith the rise in immunocompromised individuals and patients with immune-related comorbidities such as COVID-19, the rate of fungal infections is growing. This increase, along with the current plateau in antifungal drug development, has made understanding the pathogenesis and dissemination of these organisms more pertinent than ever. The mouse model of fungal infection, while informative on a basic scientific level, has severe limitations in terms of translation to the human disease. Here we present data supporting the implementation of the human cerebral organoid model, which is generated from human embryonic stem cells and accurately recapitulates relevant brain cell types and structures, to study fungal infection and dissemination to the central nervous system (CNS). This approach provides direct insight into the relevant pathogenesis of specific fungal organisms in human tissues where in vivo models are impossible. With this model system we assessed the specific brain tropisms and cellular effects of fungal pathogens known to cross the blood–brain barrier (BBB), such as <i>Cryptococcus neoformans</i>. We determined the effects of this fungal pathogen on the overall gross morphology, cellular architecture, and cytokine release from these model organoids. Furthermore, we demonstrated that <i>C. neoformans</i> penetrates and invades the organoid tissue and remains present throughout the course of infection. These results demonstrate the utility of this new model to the field and highlight the potential for this system to elucidate fungal pathogenesis to develop new therapeutic strategies to prevent and treat the disseminated stages of fungal diseases such as cryptococcal meningitis.https://www.mdpi.com/2309-608X/11/7/539cerebral organoidfungal pathogenmodel system<i>Cryptococcus neoformans</i>
spellingShingle Alfred T. Harding
Lee Gehrke
Jatin M. Vyas
Hannah Brown Harding
Human Brain Organoids: A New Model to Study <i>Cryptococcus neoformans</i> Neurotropism
Journal of Fungi
cerebral organoid
fungal pathogen
model system
<i>Cryptococcus neoformans</i>
title Human Brain Organoids: A New Model to Study <i>Cryptococcus neoformans</i> Neurotropism
title_full Human Brain Organoids: A New Model to Study <i>Cryptococcus neoformans</i> Neurotropism
title_fullStr Human Brain Organoids: A New Model to Study <i>Cryptococcus neoformans</i> Neurotropism
title_full_unstemmed Human Brain Organoids: A New Model to Study <i>Cryptococcus neoformans</i> Neurotropism
title_short Human Brain Organoids: A New Model to Study <i>Cryptococcus neoformans</i> Neurotropism
title_sort human brain organoids a new model to study i cryptococcus neoformans i neurotropism
topic cerebral organoid
fungal pathogen
model system
<i>Cryptococcus neoformans</i>
url https://www.mdpi.com/2309-608X/11/7/539
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AT leegehrke humanbrainorganoidsanewmodeltostudyicryptococcusneoformansineurotropism
AT jatinmvyas humanbrainorganoidsanewmodeltostudyicryptococcusneoformansineurotropism
AT hannahbrownharding humanbrainorganoidsanewmodeltostudyicryptococcusneoformansineurotropism