Left ventricular longitudinal function is reduced but partially compensated by increased radial function after heart transplantation

Background: In healthy hearts, left ventricular atrioventricular plane displacement (LVAVPD) measured by cardiac magnetic resonance (CMR) contributes to ∼60% of stroke volume. LVAVPD has been shown to correlate with maximal cardiac output and exercise capacity and is an independent predictor of outc...

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Main Authors: Grunde Gjesdal, MD PhD, Anna Székely, MD, Henrik Engblom, MD PhD, Håkan Arheden, MD PhD, Oscar Ö Braun, MD PhD, Katarina Steding-Ehrenborg, RPT PhD
Format: Article
Language:English
Published: Elsevier 2025-08-01
Series:JHLT Open
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Online Access:http://www.sciencedirect.com/science/article/pii/S295013342500103X
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author Grunde Gjesdal, MD PhD
Anna Székely, MD
Henrik Engblom, MD PhD
Håkan Arheden, MD PhD
Oscar Ö Braun, MD PhD
Katarina Steding-Ehrenborg, RPT PhD
author_facet Grunde Gjesdal, MD PhD
Anna Székely, MD
Henrik Engblom, MD PhD
Håkan Arheden, MD PhD
Oscar Ö Braun, MD PhD
Katarina Steding-Ehrenborg, RPT PhD
author_sort Grunde Gjesdal, MD PhD
collection DOAJ
description Background: In healthy hearts, left ventricular atrioventricular plane displacement (LVAVPD) measured by cardiac magnetic resonance (CMR) contributes to ∼60% of stroke volume. LVAVPD has been shown to correlate with maximal cardiac output and exercise capacity and is an independent predictor of outcomes in patients with heart failure. We aimed to assess if longitudinal pumping is altered, if LVAVPD is associated with exercise capacity, and if any difference in longitudinal pumping could be explained by the presence of a right bundle branch block (RBBB) in heart-transplanted patients. Method: This single-center study included 34 heart-transplanted patients who had undergone CMR and a cardiopulmonary exercise test as part of a clinical post-transplant surveillance program. Data was compared to 34 healthy sex- and age-matched controls. Results: Heart-transplanted patients had decreased LVAVPD (10.3 vs 13.7 mm, p < 0.01), lower longitudinal contribution (46% vs 53%, p < 0.01), and lower septal contribution (−3% vs 8%, p < 0.01) to stroke volume compared to controls. Furthermore, the lateral contribution was increased (44% vs 28%, p < 0.01) in the heart-transplanted patients. Longitudinal contribution to stroke volume was neither associated with exercise capacity (p = 0.20) nor cardiac output at rest (p = 0.62). There was no difference in LVAVPD in patients with and without RBBB (p = 0.81). Conclusion: Heart-transplanted patients have decreased left ventricular longitudinal function compared to healthy controls, in part compensated by an augmented lateral function. Longitudinal function is not associated with cardiac output at rest or exercise capacity in this patient group. Whether the altered pumping mechanics seen are associated with outcome remains to be investigated.
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spelling doaj-art-9cc45d6a40114158a4306b6be0a1bcf12025-08-20T03:41:57ZengElsevierJHLT Open2950-13342025-08-01910030810.1016/j.jhlto.2025.100308Left ventricular longitudinal function is reduced but partially compensated by increased radial function after heart transplantationGrunde Gjesdal, MD PhD0Anna Székely, MD1Henrik Engblom, MD PhD2Håkan Arheden, MD PhD3Oscar Ö Braun, MD PhD4Katarina Steding-Ehrenborg, RPT PhD5Department of Cardiology, Clinical Sciences, Lund University and Skåne University Hospital, Lund, Sweden; Corresponding author: Grunde Gjesdal, MD, Section for Heart Failure and Valvular Disease, Department of Heart and Lung Medicine, Skåne University Hospital, 221 85, Lund, Sweden.Clinical Physiology, Department of Clinical Sciences Lund, Lund University, Lund, Sweden; Department of Clinical Physiology, Skåne University Hospital, Lund, SwedenClinical Physiology, Department of Clinical Sciences Lund, Lund University, Lund, Sweden; Department of Clinical Physiology, Skåne University Hospital, Lund, SwedenClinical Physiology, Department of Clinical Sciences Lund, Lund University, Lund, Sweden; Department of Clinical Physiology, Skåne University Hospital, Lund, SwedenDepartment of Cardiology, Clinical Sciences, Lund University and Skåne University Hospital, Lund, SwedenClinical Physiology, Department of Clinical Sciences Lund, Lund University, Lund, Sweden; Department of Clinical Physiology, Skåne University Hospital, Lund, SwedenBackground: In healthy hearts, left ventricular atrioventricular plane displacement (LVAVPD) measured by cardiac magnetic resonance (CMR) contributes to ∼60% of stroke volume. LVAVPD has been shown to correlate with maximal cardiac output and exercise capacity and is an independent predictor of outcomes in patients with heart failure. We aimed to assess if longitudinal pumping is altered, if LVAVPD is associated with exercise capacity, and if any difference in longitudinal pumping could be explained by the presence of a right bundle branch block (RBBB) in heart-transplanted patients. Method: This single-center study included 34 heart-transplanted patients who had undergone CMR and a cardiopulmonary exercise test as part of a clinical post-transplant surveillance program. Data was compared to 34 healthy sex- and age-matched controls. Results: Heart-transplanted patients had decreased LVAVPD (10.3 vs 13.7 mm, p < 0.01), lower longitudinal contribution (46% vs 53%, p < 0.01), and lower septal contribution (−3% vs 8%, p < 0.01) to stroke volume compared to controls. Furthermore, the lateral contribution was increased (44% vs 28%, p < 0.01) in the heart-transplanted patients. Longitudinal contribution to stroke volume was neither associated with exercise capacity (p = 0.20) nor cardiac output at rest (p = 0.62). There was no difference in LVAVPD in patients with and without RBBB (p = 0.81). Conclusion: Heart-transplanted patients have decreased left ventricular longitudinal function compared to healthy controls, in part compensated by an augmented lateral function. Longitudinal function is not associated with cardiac output at rest or exercise capacity in this patient group. Whether the altered pumping mechanics seen are associated with outcome remains to be investigated.http://www.sciencedirect.com/science/article/pii/S295013342500103XHeart transplantCardiac magnetic resonance imagingLeft ventricular atrioventricular plane displacementLongitudinal heart functionLongitudinal contribution to stroke volume
spellingShingle Grunde Gjesdal, MD PhD
Anna Székely, MD
Henrik Engblom, MD PhD
Håkan Arheden, MD PhD
Oscar Ö Braun, MD PhD
Katarina Steding-Ehrenborg, RPT PhD
Left ventricular longitudinal function is reduced but partially compensated by increased radial function after heart transplantation
JHLT Open
Heart transplant
Cardiac magnetic resonance imaging
Left ventricular atrioventricular plane displacement
Longitudinal heart function
Longitudinal contribution to stroke volume
title Left ventricular longitudinal function is reduced but partially compensated by increased radial function after heart transplantation
title_full Left ventricular longitudinal function is reduced but partially compensated by increased radial function after heart transplantation
title_fullStr Left ventricular longitudinal function is reduced but partially compensated by increased radial function after heart transplantation
title_full_unstemmed Left ventricular longitudinal function is reduced but partially compensated by increased radial function after heart transplantation
title_short Left ventricular longitudinal function is reduced but partially compensated by increased radial function after heart transplantation
title_sort left ventricular longitudinal function is reduced but partially compensated by increased radial function after heart transplantation
topic Heart transplant
Cardiac magnetic resonance imaging
Left ventricular atrioventricular plane displacement
Longitudinal heart function
Longitudinal contribution to stroke volume
url http://www.sciencedirect.com/science/article/pii/S295013342500103X
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