Microglial dynamics and neuroinflammation in prodromal and early Parkinson’s disease

Abstract Parkinson’s disease (PD) is characterized by a drastic loss of dopaminergic neurons already at diagnosis. As this loss of neurons starts decades before diagnosis, understanding the prodromal stages of the disease might offer novel strategies to curb its progression. While the precise pathog...

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Main Authors: Frida Lind-Holm Mogensen, Philip Seibler, Anne Grünewald, Alessandro Michelucci
Format: Article
Language:English
Published: BMC 2025-05-01
Series:Journal of Neuroinflammation
Subjects:
Online Access:https://doi.org/10.1186/s12974-025-03462-y
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author Frida Lind-Holm Mogensen
Philip Seibler
Anne Grünewald
Alessandro Michelucci
author_facet Frida Lind-Holm Mogensen
Philip Seibler
Anne Grünewald
Alessandro Michelucci
author_sort Frida Lind-Holm Mogensen
collection DOAJ
description Abstract Parkinson’s disease (PD) is characterized by a drastic loss of dopaminergic neurons already at diagnosis. As this loss of neurons starts decades before diagnosis, understanding the prodromal stages of the disease might offer novel strategies to curb its progression. While the precise pathogenic mechanisms underlying PD remain incompletely understood, growing evidence suggests that neuroinflammation and immune dysregulation play a central role in the development and progression of the disease. Here, we delve into the emerging roles of microglia, the resident immune cells of the central nervous system, in the pathogenesis of prodromal and early-stage PD. We emphasize that microglia contribute to neuroinflammation, protein aggregation and neurodegeneration, although the underlying mechanisms are not yet known. Neuroimaging studies have provided valuable insights into the patterns of microglial activation detected in individuals with prodromal PD and at the time of clinical diagnosis. Furthermore, we highlight the complex interplay between immune dysregulation and neurodegeneration along PD development, including alterations in the peripheral immune system, brain-gut interactions and brain-immune interfaces. Lastly, we outline existing models for investigating microglial involvement in prodromal PD, along with the impact of anti-inflammatory therapies and strategies to modify risk factors. In conclusion, targeting microglial activation and immune dysfunctions in individuals at risk of PD could represent a promising preventive measure and may offer novel therapeutic strategies for early intervention and disease modification.
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issn 1742-2094
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spelling doaj-art-9cbf6cb695f043debaea82d6ce58dbc22025-08-20T03:08:43ZengBMCJournal of Neuroinflammation1742-20942025-05-0122111710.1186/s12974-025-03462-yMicroglial dynamics and neuroinflammation in prodromal and early Parkinson’s diseaseFrida Lind-Holm Mogensen0Philip Seibler1Anne Grünewald2Alessandro Michelucci3Neuro-Immunology Group, Department of Cancer Research, Luxembourg Institute of HealthInstitute of Neurogenetics, University of Lübeck and University Hospital Schleswig-HolsteinLuxembourg Centre for Systems Biomedicine, University of LuxembourgNeuro-Immunology Group, Department of Cancer Research, Luxembourg Institute of HealthAbstract Parkinson’s disease (PD) is characterized by a drastic loss of dopaminergic neurons already at diagnosis. As this loss of neurons starts decades before diagnosis, understanding the prodromal stages of the disease might offer novel strategies to curb its progression. While the precise pathogenic mechanisms underlying PD remain incompletely understood, growing evidence suggests that neuroinflammation and immune dysregulation play a central role in the development and progression of the disease. Here, we delve into the emerging roles of microglia, the resident immune cells of the central nervous system, in the pathogenesis of prodromal and early-stage PD. We emphasize that microglia contribute to neuroinflammation, protein aggregation and neurodegeneration, although the underlying mechanisms are not yet known. Neuroimaging studies have provided valuable insights into the patterns of microglial activation detected in individuals with prodromal PD and at the time of clinical diagnosis. Furthermore, we highlight the complex interplay between immune dysregulation and neurodegeneration along PD development, including alterations in the peripheral immune system, brain-gut interactions and brain-immune interfaces. Lastly, we outline existing models for investigating microglial involvement in prodromal PD, along with the impact of anti-inflammatory therapies and strategies to modify risk factors. In conclusion, targeting microglial activation and immune dysfunctions in individuals at risk of PD could represent a promising preventive measure and may offer novel therapeutic strategies for early intervention and disease modification.https://doi.org/10.1186/s12974-025-03462-yMicrogliaNeuroinflammationParkinson’s diseaseProdromal stageREM-sleep behaviour disorder
spellingShingle Frida Lind-Holm Mogensen
Philip Seibler
Anne Grünewald
Alessandro Michelucci
Microglial dynamics and neuroinflammation in prodromal and early Parkinson’s disease
Journal of Neuroinflammation
Microglia
Neuroinflammation
Parkinson’s disease
Prodromal stage
REM-sleep behaviour disorder
title Microglial dynamics and neuroinflammation in prodromal and early Parkinson’s disease
title_full Microglial dynamics and neuroinflammation in prodromal and early Parkinson’s disease
title_fullStr Microglial dynamics and neuroinflammation in prodromal and early Parkinson’s disease
title_full_unstemmed Microglial dynamics and neuroinflammation in prodromal and early Parkinson’s disease
title_short Microglial dynamics and neuroinflammation in prodromal and early Parkinson’s disease
title_sort microglial dynamics and neuroinflammation in prodromal and early parkinson s disease
topic Microglia
Neuroinflammation
Parkinson’s disease
Prodromal stage
REM-sleep behaviour disorder
url https://doi.org/10.1186/s12974-025-03462-y
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AT annegrunewald microglialdynamicsandneuroinflammationinprodromalandearlyparkinsonsdisease
AT alessandromichelucci microglialdynamicsandneuroinflammationinprodromalandearlyparkinsonsdisease