Green Tea Catechin, EGCG, Suppresses PCB 102-Induced Proliferation in Estrogen-Sensitive Breast Cancer Cells

The persistence of polychlorinated biphenyls (PCBs) in the environment is of considerable concern since they accumulate in human breast tissue and may stimulate the growth of estrogen-sensitive tumors. Studies have shown that EGCG from green tea can modify estrogenic activity and thus may act as a c...

Full description

Saved in:
Bibliographic Details
Main Authors: Kimberly Mantzke Baker, Angela C. Bauer
Format: Article
Language:English
Published: Wiley 2015-01-01
Series:International Journal of Breast Cancer
Online Access:http://dx.doi.org/10.1155/2015/163591
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850179453231038464
author Kimberly Mantzke Baker
Angela C. Bauer
author_facet Kimberly Mantzke Baker
Angela C. Bauer
author_sort Kimberly Mantzke Baker
collection DOAJ
description The persistence of polychlorinated biphenyls (PCBs) in the environment is of considerable concern since they accumulate in human breast tissue and may stimulate the growth of estrogen-sensitive tumors. Studies have shown that EGCG from green tea can modify estrogenic activity and thus may act as a cancer chemopreventive agent. In the present study, we evaluated the individual and combined effects of PCB 102 and EGCG on cell proliferation using an estrogen-sensitive breast cancer cell line MCF-7/BOS. PCB 102 (1–10 μM) increased cell proliferation in a dose-dependent manner. Furthermore, the proliferative effects of PCB 102 were mediated by ERα and could be abrogated by the selective ERα antagonist MPP. EGCG (10–50 μM) caused a dose-dependent inhibition of PCB 102-induced cell proliferation, with nearly complete inhibition at 25 μM EGCG. The antiproliferative action of EGCG was mediated by ERβ and could be blocked by the ERβ-specific inhibitor PHTPP. In conclusion, EGCG suppressed the proliferation-stimulating activity of the environmental estrogen PCB 102 which may be helpful in the chemoprevention of breast cancer.
format Article
id doaj-art-9c6c0f188aa141ebb88768f0932d64ec
institution OA Journals
issn 2090-3170
2090-3189
language English
publishDate 2015-01-01
publisher Wiley
record_format Article
series International Journal of Breast Cancer
spelling doaj-art-9c6c0f188aa141ebb88768f0932d64ec2025-08-20T02:18:29ZengWileyInternational Journal of Breast Cancer2090-31702090-31892015-01-01201510.1155/2015/163591163591Green Tea Catechin, EGCG, Suppresses PCB 102-Induced Proliferation in Estrogen-Sensitive Breast Cancer CellsKimberly Mantzke Baker0Angela C. Bauer1Department of Biology, University of Indianapolis, Lilly Science Hall 30, 1400 East Hanna Avenue, Indianapolis, IN 46227, USADepartment of Biology, High Point University, Congdon 217, 833 Montlieu Avenue, High Point, NC 27268, USAThe persistence of polychlorinated biphenyls (PCBs) in the environment is of considerable concern since they accumulate in human breast tissue and may stimulate the growth of estrogen-sensitive tumors. Studies have shown that EGCG from green tea can modify estrogenic activity and thus may act as a cancer chemopreventive agent. In the present study, we evaluated the individual and combined effects of PCB 102 and EGCG on cell proliferation using an estrogen-sensitive breast cancer cell line MCF-7/BOS. PCB 102 (1–10 μM) increased cell proliferation in a dose-dependent manner. Furthermore, the proliferative effects of PCB 102 were mediated by ERα and could be abrogated by the selective ERα antagonist MPP. EGCG (10–50 μM) caused a dose-dependent inhibition of PCB 102-induced cell proliferation, with nearly complete inhibition at 25 μM EGCG. The antiproliferative action of EGCG was mediated by ERβ and could be blocked by the ERβ-specific inhibitor PHTPP. In conclusion, EGCG suppressed the proliferation-stimulating activity of the environmental estrogen PCB 102 which may be helpful in the chemoprevention of breast cancer.http://dx.doi.org/10.1155/2015/163591
spellingShingle Kimberly Mantzke Baker
Angela C. Bauer
Green Tea Catechin, EGCG, Suppresses PCB 102-Induced Proliferation in Estrogen-Sensitive Breast Cancer Cells
International Journal of Breast Cancer
title Green Tea Catechin, EGCG, Suppresses PCB 102-Induced Proliferation in Estrogen-Sensitive Breast Cancer Cells
title_full Green Tea Catechin, EGCG, Suppresses PCB 102-Induced Proliferation in Estrogen-Sensitive Breast Cancer Cells
title_fullStr Green Tea Catechin, EGCG, Suppresses PCB 102-Induced Proliferation in Estrogen-Sensitive Breast Cancer Cells
title_full_unstemmed Green Tea Catechin, EGCG, Suppresses PCB 102-Induced Proliferation in Estrogen-Sensitive Breast Cancer Cells
title_short Green Tea Catechin, EGCG, Suppresses PCB 102-Induced Proliferation in Estrogen-Sensitive Breast Cancer Cells
title_sort green tea catechin egcg suppresses pcb 102 induced proliferation in estrogen sensitive breast cancer cells
url http://dx.doi.org/10.1155/2015/163591
work_keys_str_mv AT kimberlymantzkebaker greenteacatechinegcgsuppressespcb102inducedproliferationinestrogensensitivebreastcancercells
AT angelacbauer greenteacatechinegcgsuppressespcb102inducedproliferationinestrogensensitivebreastcancercells