Electrophysiological evaluation of the neuromuscular junction: a brief review

The nerve terminal and muscle membrane compose the neuromuscular junction. After opening the voltage-gated calcium channels, action potentials from the motor axons provoke a cascade for the acetylcholine release from synaptic vesicles to the synaptic cleft, where it binds to its receptor at the musc...

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Main Authors: João Aris Kouyoumdjian, Eduardo de Paula Estephan
Format: Article
Language:English
Published: Thieme Revinter Publicações 2023-12-01
Series:Arquivos de Neuro-Psiquiatria
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Online Access:http://www.thieme-connect.de/DOI/DOI?10.1055/s-0043-1777749
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author João Aris Kouyoumdjian
Eduardo de Paula Estephan
author_facet João Aris Kouyoumdjian
Eduardo de Paula Estephan
author_sort João Aris Kouyoumdjian
collection DOAJ
description The nerve terminal and muscle membrane compose the neuromuscular junction. After opening the voltage-gated calcium channels, action potentials from the motor axons provoke a cascade for the acetylcholine release from synaptic vesicles to the synaptic cleft, where it binds to its receptor at the muscle membrane for depolarization. Low amplitude compound muscle action potential typically presents in presynaptic disorders, increasing by more than 100% after a 10-second effort in the Lambert-Eaton myasthenic syndrome and less in botulism. Needle electromyography may show myopathic motor unit action potentials and morphological instability (“jiggle”) due to impulse blocking. Low-frequency repetitive nerve stimulation (RNS) is helpful in postsynaptic disorders, such as myasthenia gravis and most congenital myasthenic syndromes, where the number of functioning acetylcholine receptors is reduced. Low-frequency RNS with a decrement >10% is abnormal when comparing the 4th to the first compound muscle action potential amplitude. High-frequency RNS is helpful in presynaptic disorders like Lambert-Eaton myasthenic syndrome, botulism, and some rare congenital myasthenic syndromes. The high-frequency RNS releases more calcium, increasing the acetylcholine with a compound muscle action potential increment. Concentric needle records apparent single-fiber action potentials (spikes). A voluntary activation measures the jitter between spikes from two endplates. An electrical activation measures the jitter of one spike (one endplate). The jitter is the most sensitive test for detecting a neuromuscular junction dysfunction. Most neuromuscular junction disorders are responsive to treatment.
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spelling doaj-art-9bd63dc4fee44b34a922eb000c300e562025-08-20T02:00:42ZengThieme Revinter PublicaçõesArquivos de Neuro-Psiquiatria0004-282X1678-42272023-12-0181121040105210.1055/s-0043-1777749Electrophysiological evaluation of the neuromuscular junction: a brief reviewJoão Aris Kouyoumdjian0https://orcid.org/0000-0002-9384-9964Eduardo de Paula Estephan1https://orcid.org/0000-0002-6807-1951Faculdade de Medicina de São José do Rio Preto, Departamento de Ciências Neurológicas, Psiquiatria e Psicologia Médica, São José do Rio Preto SP, Brazil.Faculdade de Medicina de São José do Rio Preto, Departamento de Ciências Neurológicas, Psiquiatria e Psicologia Médica, São José do Rio Preto SP, Brazil.The nerve terminal and muscle membrane compose the neuromuscular junction. After opening the voltage-gated calcium channels, action potentials from the motor axons provoke a cascade for the acetylcholine release from synaptic vesicles to the synaptic cleft, where it binds to its receptor at the muscle membrane for depolarization. Low amplitude compound muscle action potential typically presents in presynaptic disorders, increasing by more than 100% after a 10-second effort in the Lambert-Eaton myasthenic syndrome and less in botulism. Needle electromyography may show myopathic motor unit action potentials and morphological instability (“jiggle”) due to impulse blocking. Low-frequency repetitive nerve stimulation (RNS) is helpful in postsynaptic disorders, such as myasthenia gravis and most congenital myasthenic syndromes, where the number of functioning acetylcholine receptors is reduced. Low-frequency RNS with a decrement >10% is abnormal when comparing the 4th to the first compound muscle action potential amplitude. High-frequency RNS is helpful in presynaptic disorders like Lambert-Eaton myasthenic syndrome, botulism, and some rare congenital myasthenic syndromes. The high-frequency RNS releases more calcium, increasing the acetylcholine with a compound muscle action potential increment. Concentric needle records apparent single-fiber action potentials (spikes). A voluntary activation measures the jitter between spikes from two endplates. An electrical activation measures the jitter of one spike (one endplate). The jitter is the most sensitive test for detecting a neuromuscular junction dysfunction. Most neuromuscular junction disorders are responsive to treatment.http://www.thieme-connect.de/DOI/DOI?10.1055/s-0043-1777749Neuromuscular JunctionMyasthenia GravisRepetitive Nerve StimulationSingle-Fiber ElectromyographyJitterJunção NeuromuscularMiastenia GravisEstimulação Nervosa RepetitivaEletromiografia de Fibra ÚnicaJitter
spellingShingle João Aris Kouyoumdjian
Eduardo de Paula Estephan
Electrophysiological evaluation of the neuromuscular junction: a brief review
Arquivos de Neuro-Psiquiatria
Neuromuscular Junction
Myasthenia Gravis
Repetitive Nerve Stimulation
Single-Fiber Electromyography
Jitter
Junção Neuromuscular
Miastenia Gravis
Estimulação Nervosa Repetitiva
Eletromiografia de Fibra Única
Jitter
title Electrophysiological evaluation of the neuromuscular junction: a brief review
title_full Electrophysiological evaluation of the neuromuscular junction: a brief review
title_fullStr Electrophysiological evaluation of the neuromuscular junction: a brief review
title_full_unstemmed Electrophysiological evaluation of the neuromuscular junction: a brief review
title_short Electrophysiological evaluation of the neuromuscular junction: a brief review
title_sort electrophysiological evaluation of the neuromuscular junction a brief review
topic Neuromuscular Junction
Myasthenia Gravis
Repetitive Nerve Stimulation
Single-Fiber Electromyography
Jitter
Junção Neuromuscular
Miastenia Gravis
Estimulação Nervosa Repetitiva
Eletromiografia de Fibra Única
Jitter
url http://www.thieme-connect.de/DOI/DOI?10.1055/s-0043-1777749
work_keys_str_mv AT joaoariskouyoumdjian electrophysiologicalevaluationoftheneuromuscularjunctionabriefreview
AT eduardodepaulaestephan electrophysiologicalevaluationoftheneuromuscularjunctionabriefreview