p38 MAPK Endogenous Inhibition Improves Neurological Deficits in Global Cerebral Ischemia/Reperfusion Mice
Cerebral ischemia/reperfusion (I/R) injury is a complex pathophysiological process that can lead to neurological function damage and the formation of cerebral infarction. The p38 MAPK pathway has attracted considerable attention in cerebral I/R injury (IRI), but little research has been carried out...
Saved in:
Main Authors: | Kun Hou, Zhi-cheng Xiao, Hai-Long Dai |
---|---|
Format: | Article |
Language: | English |
Published: |
Wiley
2022-01-01
|
Series: | Neural Plasticity |
Online Access: | http://dx.doi.org/10.1155/2022/3300327 |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Electroacupuncture Pretreatment Elicits Tolerance to Cerebral Ischemia/Reperfusion through Inhibition of the GluN2B/m-Calpain/p38 MAPK Proapoptotic Pathway
by: Bao-yu Zhang, et al.
Published: (2020-01-01) -
Inhibition of P38 MAPK Downregulates the Expression of IL-1β to Protect Lung from Acute Injury in Intestinal Ischemia Reperfusion Rats
by: De-Yi Zheng, et al.
Published: (2016-01-01) -
Cordycepin Protects against Hepatic Ischemia/Reperfusion Injury via Inhibiting MAPK/NF-κB Pathway
by: Jiameng Ding, et al.
Published: (2022-01-01) -
Gen inhibiting the Wnt/Ca2+ signaling pathway alleviates cerebral ischemia/reperfusion injury
by: Li Li, et al.
Published: (2025-02-01) -
HACE1 protects against myocardial ischemia–reperfusion injury via inhibition of mitochondrial fission in mice
by: Bang-Xia Liu, et al.
Published: (2025-02-01)