A very rare cause of protein losing enteropathy: Gaucher disease

Background. Mesenteric lymphadenopathy is a rare manifestation of Gaucher disease (GD) in children and can be accompanied by protein losing enteropathy (PLE). PLE is a difficult-to-treat complication of GD. To date, only a few pediatric GD cases with PLE and massive mesenteric lymphadenopathi...

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Main Authors: Mehmet Akif Göktaş, Ersin Gümüş, Hülya Demir, Hayriye Hızarcıoğlu Gülşen, İnci Nur Saltık-Temizel, Hasan Özen, Şafak Güçer, Aysel Yüce
Format: Article
Language:English
Published: Hacettepe University Institute of Child Health 2021-08-01
Series:The Turkish Journal of Pediatrics
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Online Access:https://turkjpediatr.org/article/view/351
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author Mehmet Akif Göktaş
Ersin Gümüş
Hülya Demir
Hayriye Hızarcıoğlu Gülşen
İnci Nur Saltık-Temizel
Hasan Özen
Şafak Güçer
Aysel Yüce
author_facet Mehmet Akif Göktaş
Ersin Gümüş
Hülya Demir
Hayriye Hızarcıoğlu Gülşen
İnci Nur Saltık-Temizel
Hasan Özen
Şafak Güçer
Aysel Yüce
author_sort Mehmet Akif Göktaş
collection DOAJ
description Background. Mesenteric lymphadenopathy is a rare manifestation of Gaucher disease (GD) in children and can be accompanied by protein losing enteropathy (PLE). PLE is a difficult-to-treat complication of GD. To date, only a few pediatric GD cases with PLE and massive mesenteric lymphadenopathies have been reported. Case. Here, we report a girl with chronic neuronopathic GD, whose disease course was complicated by massive mesenteric lymphadenopathies with resultant protein losing enteropathy despite a regular and appropriate enzyme replacement therapy of 60 IU/kg/biweekly until the development of mesenteric lymphadenopathies and 120 IU/kg/biweekly thereafter. Conclusions. PLE is a devastating and life threatening complication of GD developing despite long term use of high dose ERT. Clinicians should be alert for this complication particularly in GD patients presenting with progressive abdominal distension, edema, ascites and diarrhea or in patients who have already developed mesenteric lymphadenopathies. Timely diagnosis may allow early intervention with previously suggested surgical or medical treatment options. Although there is no specific and effective treatment, surgical and aggressive medical interventions in addition to ERT were reported to relieve diarrhea and halt progression of mesenteric lymphadenopathies.
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spelling doaj-art-9a4f3a0d89e64adcb434daa1507ef5ea2025-08-20T02:01:46ZengHacettepe University Institute of Child HealthThe Turkish Journal of Pediatrics0041-43012791-64212021-08-0163410.24953/turkjped.2021.04.020A very rare cause of protein losing enteropathy: Gaucher diseaseMehmet Akif Göktaş0Ersin Gümüş1Hülya Demir2Hayriye Hızarcıoğlu Gülşen3İnci Nur Saltık-Temizel4Hasan Özen5Şafak Güçer6Aysel Yüce7Division of Pediatric Gastroenterology, Hepatology and Nutrition, Hacettepe University Faculty of Medicine, Ankara, Turkey.Division of Pediatric Gastroenterology, Hepatology and Nutrition, Hacettepe University Faculty of Medicine, Ankara, Turkey.Division of Pediatric Gastroenterology, Hepatology and Nutrition, Hacettepe University Faculty of Medicine, Ankara, Turkey.Division of Pediatric Gastroenterology, Hepatology and Nutrition, Hacettepe University Faculty of Medicine, Ankara, Turkey.Division of Pediatric Gastroenterology, Hepatology and Nutrition, Hacettepe University Faculty of Medicine, Ankara, Turkey.Division of Pediatric Gastroenterology, Hepatology and Nutrition, Hacettepe University Faculty of Medicine, Ankara, Turkey.Division of Pediatric Pathology Unit, Department of Pediatrics, Hacettepe University Faculty of Medicine, Ankara, Turkey.Division of Pediatric Gastroenterology, Hepatology and Nutrition, Hacettepe University Faculty of Medicine, Ankara, Turkey. Background. Mesenteric lymphadenopathy is a rare manifestation of Gaucher disease (GD) in children and can be accompanied by protein losing enteropathy (PLE). PLE is a difficult-to-treat complication of GD. To date, only a few pediatric GD cases with PLE and massive mesenteric lymphadenopathies have been reported. Case. Here, we report a girl with chronic neuronopathic GD, whose disease course was complicated by massive mesenteric lymphadenopathies with resultant protein losing enteropathy despite a regular and appropriate enzyme replacement therapy of 60 IU/kg/biweekly until the development of mesenteric lymphadenopathies and 120 IU/kg/biweekly thereafter. Conclusions. PLE is a devastating and life threatening complication of GD developing despite long term use of high dose ERT. Clinicians should be alert for this complication particularly in GD patients presenting with progressive abdominal distension, edema, ascites and diarrhea or in patients who have already developed mesenteric lymphadenopathies. Timely diagnosis may allow early intervention with previously suggested surgical or medical treatment options. Although there is no specific and effective treatment, surgical and aggressive medical interventions in addition to ERT were reported to relieve diarrhea and halt progression of mesenteric lymphadenopathies. https://turkjpediatr.org/article/view/351enzyme replacement therapy; lymphadenopathy; lysosomal storage disorder; Gaucher disease
spellingShingle Mehmet Akif Göktaş
Ersin Gümüş
Hülya Demir
Hayriye Hızarcıoğlu Gülşen
İnci Nur Saltık-Temizel
Hasan Özen
Şafak Güçer
Aysel Yüce
A very rare cause of protein losing enteropathy: Gaucher disease
The Turkish Journal of Pediatrics
enzyme replacement therapy; lymphadenopathy; lysosomal storage disorder; Gaucher disease
title A very rare cause of protein losing enteropathy: Gaucher disease
title_full A very rare cause of protein losing enteropathy: Gaucher disease
title_fullStr A very rare cause of protein losing enteropathy: Gaucher disease
title_full_unstemmed A very rare cause of protein losing enteropathy: Gaucher disease
title_short A very rare cause of protein losing enteropathy: Gaucher disease
title_sort very rare cause of protein losing enteropathy gaucher disease
topic enzyme replacement therapy; lymphadenopathy; lysosomal storage disorder; Gaucher disease
url https://turkjpediatr.org/article/view/351
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