Exploring the Ophthalmic Features of Cornelia de Lange Syndrome: A Case Report

Cornelia de Lange Syndrome (CdLS) is a rare genetic disorder that affects growth, development and various body systems, with symptoms ranging from mild to severe. The condition is marked by distinctive facial features such as arched eyebrows, long eyelashes, a small nose and a thin upper lip. Growth...

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Main Authors: Radhika Paranjpe, Shikha Rai, Tushar Agrawal, Gufran Ali Kamdar
Format: Article
Language:English
Published: JCDR Research and Publications Private Limited 2025-04-01
Series:Journal of Clinical and Diagnostic Research
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Online Access:https://jcdr.net/articles/PDF/20870/76379_CE[Ra1]_F(IS)_QC(PS_IS)_PF1(AG_OM)_PFA_NC(IS)_PB(AG_IS)_PN(IS).pdf
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author Radhika Paranjpe
Shikha Rai
Tushar Agrawal
Gufran Ali Kamdar
author_facet Radhika Paranjpe
Shikha Rai
Tushar Agrawal
Gufran Ali Kamdar
author_sort Radhika Paranjpe
collection DOAJ
description Cornelia de Lange Syndrome (CdLS) is a rare genetic disorder that affects growth, development and various body systems, with symptoms ranging from mild to severe. The condition is marked by distinctive facial features such as arched eyebrows, long eyelashes, a small nose and a thin upper lip. Growth delays are noticeable both before and after birth, leading to short stature. Affected individuals often have limb abnormalities, including missing or underdeveloped fingers and toes, as well as cognitive impairments that can vary from mild to severe. CdLS is caused by mutations in genes crucial for early development, particularly those involved in forming facial, limb and brain structures. The most common mutations occur in the NIPBL, SMC1A and SMC3 genes, although other genetic factors may also contribute. These mutations disrupt normal cellular functions, resulting in the physical and developmental challenges characteristic of the disorder. Effective management of CdLS requires a collaborative approach involving a team of medical professionals, therapists and educators who work together to address the diverse needs of affected individuals. This may include Physical Therapy (PT) for limb abnormalities, speech therapy and Occupational Therapy (OT) to support cognitive and developmental progress, as well as regular monitoring of growth and health. A seven-year-old child presented with involuntary eye movements (horizontal nystagmus) in both eyes since early childhood, along with global developmental delay, reduced psychomotor skills and intellectual disability. Clinical findings included a unibrow, hirsutism, long eyelashes and visual acuity of 6/60 in both eyes, improving to 6/36 with correction.
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spelling doaj-art-9964d991fc38476a93d0186094aedfb12025-08-20T02:18:21ZengJCDR Research and Publications Private LimitedJournal of Clinical and Diagnostic Research2249-782X0973-709X2025-04-01194ND01ND0210.7860/JCDR/2025/76379.20870Exploring the Ophthalmic Features of Cornelia de Lange Syndrome: A Case ReportRadhika Paranjpe0Shikha Rai1Tushar Agrawal2Gufran Ali Kamdar3Associate Professor, Department of Ophthalmology, Dr. D. Y. Patil Medical College, Pune, Maharashtra, India.Resident, Department of Ophthalmology, Dr. D. Y. Patil Medical College, Pune, Maharashtra, India.Resident, Department of Ophthalmology, Dr. D. Y. Patil Medical College, Pune, Maharashtra, India.Resident, Department of Ophthalmology, Dr. D. Y. Patil Medical College, Pune, Maharashtra, India.Cornelia de Lange Syndrome (CdLS) is a rare genetic disorder that affects growth, development and various body systems, with symptoms ranging from mild to severe. The condition is marked by distinctive facial features such as arched eyebrows, long eyelashes, a small nose and a thin upper lip. Growth delays are noticeable both before and after birth, leading to short stature. Affected individuals often have limb abnormalities, including missing or underdeveloped fingers and toes, as well as cognitive impairments that can vary from mild to severe. CdLS is caused by mutations in genes crucial for early development, particularly those involved in forming facial, limb and brain structures. The most common mutations occur in the NIPBL, SMC1A and SMC3 genes, although other genetic factors may also contribute. These mutations disrupt normal cellular functions, resulting in the physical and developmental challenges characteristic of the disorder. Effective management of CdLS requires a collaborative approach involving a team of medical professionals, therapists and educators who work together to address the diverse needs of affected individuals. This may include Physical Therapy (PT) for limb abnormalities, speech therapy and Occupational Therapy (OT) to support cognitive and developmental progress, as well as regular monitoring of growth and health. A seven-year-old child presented with involuntary eye movements (horizontal nystagmus) in both eyes since early childhood, along with global developmental delay, reduced psychomotor skills and intellectual disability. Clinical findings included a unibrow, hirsutism, long eyelashes and visual acuity of 6/60 in both eyes, improving to 6/36 with correction.https://jcdr.net/articles/PDF/20870/76379_CE[Ra1]_F(IS)_QC(PS_IS)_PF1(AG_OM)_PFA_NC(IS)_PB(AG_IS)_PN(IS).pdfcorneal diameterhirsutismlong lashes
spellingShingle Radhika Paranjpe
Shikha Rai
Tushar Agrawal
Gufran Ali Kamdar
Exploring the Ophthalmic Features of Cornelia de Lange Syndrome: A Case Report
Journal of Clinical and Diagnostic Research
corneal diameter
hirsutism
long lashes
title Exploring the Ophthalmic Features of Cornelia de Lange Syndrome: A Case Report
title_full Exploring the Ophthalmic Features of Cornelia de Lange Syndrome: A Case Report
title_fullStr Exploring the Ophthalmic Features of Cornelia de Lange Syndrome: A Case Report
title_full_unstemmed Exploring the Ophthalmic Features of Cornelia de Lange Syndrome: A Case Report
title_short Exploring the Ophthalmic Features of Cornelia de Lange Syndrome: A Case Report
title_sort exploring the ophthalmic features of cornelia de lange syndrome a case report
topic corneal diameter
hirsutism
long lashes
url https://jcdr.net/articles/PDF/20870/76379_CE[Ra1]_F(IS)_QC(PS_IS)_PF1(AG_OM)_PFA_NC(IS)_PB(AG_IS)_PN(IS).pdf
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