Molecular and Hematological Characterization of α-Thalassemia in Denizli Province

Aim: Alpha thalassemia, a common monogenic disorder, occurs with defective synthesis of the α-globin chain and has a very wide clinical spectrum depending on the disorders in the globin genes. This study aims to determine the frequency of α-globin gene mutations in patients suspected of having alpha...

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Main Authors: Derya Karaer, Tuba Uğurlu Er, Nevin Alayvaz Aslan, Taner Durak
Format: Article
Language:English
Published: Galenos Publishing House 2025-04-01
Series:Acta Haematologica Oncologica Turcica
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Online Access:https://www.actaoncologicaturcica.com/articles/molecular-and-hematological-characterization-of-a-thalassemia-in-denizli-province/doi/ahot.galenos.2025.2025-3-3
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author Derya Karaer
Tuba Uğurlu Er
Nevin Alayvaz Aslan
Taner Durak
author_facet Derya Karaer
Tuba Uğurlu Er
Nevin Alayvaz Aslan
Taner Durak
author_sort Derya Karaer
collection DOAJ
description Aim: Alpha thalassemia, a common monogenic disorder, occurs with defective synthesis of the α-globin chain and has a very wide clinical spectrum depending on the disorders in the globin genes. This study aims to determine the frequency of α-globin gene mutations in patients suspected of having alpha thalassemia in Denizli province and to evaluate the phenotypic effects of detected mutations. Methods: A total of 93 patients (55 female, 38 male) with suspected alpha thalassemia based on anemia, family history, premarital screening and high-performance liquid chromatography results were analyzed for α-thalassemia gene deletions. DNA was isolated from peripheral blood samples, and the results were evaluated using the “Seqline Alpha Thalassemia Diagnostic Kit” to detect common α-globin gene deletions: large deletions of 3.7 kb (-α3.7), 4.2 kb (-α4.2), and 20.5 kb (-(α)20.5) in HBA1-2 genes, as well as MED1 (--MED) and SEA (--SEA). Results: Among the 93 patients, mutations were detected in 38 patients, yielding a deletion detection rate of 40.9%. The most common mutation was -α3.7 (40.7%), followed by -(α)20.5 (17.1%), --MED (5.2%), and -α4.2 (2.6%). No - SEA deletions were identified. Conclusion: This study represents the first molecular characterization of α-thalassemia in Denizli province, identifying four different α-thalassemia deletions and eight distinct genotypes. The findings provide valuable insights into the regional distribution and clinical implications of these mutations.
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institution Kabale University
issn 3061-9947
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spelling doaj-art-97dee51784994d32b13c7aeb178463592025-08-20T03:40:33ZengGalenos Publishing HouseActa Haematologica Oncologica Turcica3061-99472025-04-01581465010.4274/ahot.galenos.2025.2025-3-3Molecular and Hematological Characterization of α-Thalassemia in Denizli ProvinceDerya Karaer0https://orcid.org/0000-0002-1874-0109Tuba Uğurlu Er1https://orcid.org/0009-0001-9600-166XNevin Alayvaz Aslan2https://orcid.org/0000-0003-3292-8519Taner Durak3https://orcid.org/0000-0001-6143-1670Pamukkale University Faculty of Medicine, Department of Medical Genetics, Denizli, TürkiyePamukkale University Faculty of Medicine, Department of Medical Genetics, Denizli, TürkiyePamukkale University Faculty of Medicine, Department of Hematology, Denizli, TürkiyePamukkale University Faculty of Medicine, Department of Medical Genetics, Denizli, TürkiyeAim: Alpha thalassemia, a common monogenic disorder, occurs with defective synthesis of the α-globin chain and has a very wide clinical spectrum depending on the disorders in the globin genes. This study aims to determine the frequency of α-globin gene mutations in patients suspected of having alpha thalassemia in Denizli province and to evaluate the phenotypic effects of detected mutations. Methods: A total of 93 patients (55 female, 38 male) with suspected alpha thalassemia based on anemia, family history, premarital screening and high-performance liquid chromatography results were analyzed for α-thalassemia gene deletions. DNA was isolated from peripheral blood samples, and the results were evaluated using the “Seqline Alpha Thalassemia Diagnostic Kit” to detect common α-globin gene deletions: large deletions of 3.7 kb (-α3.7), 4.2 kb (-α4.2), and 20.5 kb (-(α)20.5) in HBA1-2 genes, as well as MED1 (--MED) and SEA (--SEA). Results: Among the 93 patients, mutations were detected in 38 patients, yielding a deletion detection rate of 40.9%. The most common mutation was -α3.7 (40.7%), followed by -(α)20.5 (17.1%), --MED (5.2%), and -α4.2 (2.6%). No - SEA deletions were identified. Conclusion: This study represents the first molecular characterization of α-thalassemia in Denizli province, identifying four different α-thalassemia deletions and eight distinct genotypes. The findings provide valuable insights into the regional distribution and clinical implications of these mutations.https://www.actaoncologicaturcica.com/articles/molecular-and-hematological-characterization-of-a-thalassemia-in-denizli-province/doi/ahot.galenos.2025.2025-3-3alpha thalassemiaalpha-globin genegene deletiongenotype-phenotype correlation
spellingShingle Derya Karaer
Tuba Uğurlu Er
Nevin Alayvaz Aslan
Taner Durak
Molecular and Hematological Characterization of α-Thalassemia in Denizli Province
Acta Haematologica Oncologica Turcica
alpha thalassemia
alpha-globin gene
gene deletion
genotype-phenotype correlation
title Molecular and Hematological Characterization of α-Thalassemia in Denizli Province
title_full Molecular and Hematological Characterization of α-Thalassemia in Denizli Province
title_fullStr Molecular and Hematological Characterization of α-Thalassemia in Denizli Province
title_full_unstemmed Molecular and Hematological Characterization of α-Thalassemia in Denizli Province
title_short Molecular and Hematological Characterization of α-Thalassemia in Denizli Province
title_sort molecular and hematological characterization of α thalassemia in denizli province
topic alpha thalassemia
alpha-globin gene
gene deletion
genotype-phenotype correlation
url https://www.actaoncologicaturcica.com/articles/molecular-and-hematological-characterization-of-a-thalassemia-in-denizli-province/doi/ahot.galenos.2025.2025-3-3
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AT tubaugurluer molecularandhematologicalcharacterizationofathalassemiaindenizliprovince
AT nevinalayvazaslan molecularandhematologicalcharacterizationofathalassemiaindenizliprovince
AT tanerdurak molecularandhematologicalcharacterizationofathalassemiaindenizliprovince