Trypsin promotes porcine deltacoronavirus mediating cell-to-cell fusion in a cell type-dependent manner

Porcine deltacoronavirus (PDCoV) is a newly emerging threat to the global porcine industry. PDCoV has been successfully isolated using various medium additives including trypsin, and although we know it is important for viral replication, the mechanism has not been fully elucidated. Here, we systema...

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Main Authors: Yue-Lin Yang, Fandan Meng, Pan Qin, Georg Herrler, Yao-Wei Huang, Yan-Dong Tang
Format: Article
Language:English
Published: Taylor & Francis Group 2020-01-01
Series:Emerging Microbes and Infections
Subjects:
Online Access:https://www.tandfonline.com/doi/10.1080/22221751.2020.1730245
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author Yue-Lin Yang
Fandan Meng
Pan Qin
Georg Herrler
Yao-Wei Huang
Yan-Dong Tang
author_facet Yue-Lin Yang
Fandan Meng
Pan Qin
Georg Herrler
Yao-Wei Huang
Yan-Dong Tang
author_sort Yue-Lin Yang
collection DOAJ
description Porcine deltacoronavirus (PDCoV) is a newly emerging threat to the global porcine industry. PDCoV has been successfully isolated using various medium additives including trypsin, and although we know it is important for viral replication, the mechanism has not been fully elucidated. Here, we systematically investigated the role of trypsin in PDCoV replication including cell entry, cell-to-cell membrane fusion and virus release. Using pseudovirus entry assays, we demonstrated that PDCoV entry is not trypsin dependent. Furthermore, unlike porcine epidemic diarrhea virus (PEDV), in which trypsin is important for the release of virus from infected cells, PDCoV release was not affected by trypsin. We also demonstrated that trypsin promotes PDCoV replication by enhancing cell-to-cell membrane fusion. Most importantly, our study illustrates two distinct spreading patterns from infected cells to uninfected cells during PDCoV transmission, and the role of trypsin in PDCoV replication in cells with different virus spreading types. Overall, these results clarify that trypsin promotes PDCoV replication by mediating cell-to-cell fusion transmission but is not crucial for viral entry. This knowledge can potentially contribute to improvement of virus production efficiency in culture, not only for vaccine preparation but also to develop antiviral treatments.
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spelling doaj-art-977fad37a4df45419092e1e7f9b534b12025-08-20T03:08:31ZengTaylor & Francis GroupEmerging Microbes and Infections2222-17512020-01-019145746810.1080/22221751.2020.1730245Trypsin promotes porcine deltacoronavirus mediating cell-to-cell fusion in a cell type-dependent mannerYue-Lin Yang0Fandan Meng1Pan Qin2Georg Herrler3Yao-Wei Huang4Yan-Dong Tang5State Key Laboratory of Veterinary Biotechnology, Harbin Veterinary Research Institute of Chinese Academy of Agricultural Sciences, Harbin, People’s Republic of ChinaState Key Laboratory of Veterinary Biotechnology, Harbin Veterinary Research Institute of Chinese Academy of Agricultural Sciences, Harbin, People’s Republic of ChinaInstitute of Preventive Veterinary Medicine and Key Laboratory of Animal Virology of Ministry of Agriculture, College of Animal Sciences, Zhejiang University, Hangzhou, People’s Republic of ChinaInstitute for Virology, University of Veterinary Medicine Hannover, Foundation, Hannover, GermanyInstitute of Preventive Veterinary Medicine and Key Laboratory of Animal Virology of Ministry of Agriculture, College of Animal Sciences, Zhejiang University, Hangzhou, People’s Republic of ChinaState Key Laboratory of Veterinary Biotechnology, Harbin Veterinary Research Institute of Chinese Academy of Agricultural Sciences, Harbin, People’s Republic of ChinaPorcine deltacoronavirus (PDCoV) is a newly emerging threat to the global porcine industry. PDCoV has been successfully isolated using various medium additives including trypsin, and although we know it is important for viral replication, the mechanism has not been fully elucidated. Here, we systematically investigated the role of trypsin in PDCoV replication including cell entry, cell-to-cell membrane fusion and virus release. Using pseudovirus entry assays, we demonstrated that PDCoV entry is not trypsin dependent. Furthermore, unlike porcine epidemic diarrhea virus (PEDV), in which trypsin is important for the release of virus from infected cells, PDCoV release was not affected by trypsin. We also demonstrated that trypsin promotes PDCoV replication by enhancing cell-to-cell membrane fusion. Most importantly, our study illustrates two distinct spreading patterns from infected cells to uninfected cells during PDCoV transmission, and the role of trypsin in PDCoV replication in cells with different virus spreading types. Overall, these results clarify that trypsin promotes PDCoV replication by mediating cell-to-cell fusion transmission but is not crucial for viral entry. This knowledge can potentially contribute to improvement of virus production efficiency in culture, not only for vaccine preparation but also to develop antiviral treatments.https://www.tandfonline.com/doi/10.1080/22221751.2020.1730245PDCoVtrypsinentryvirus releasecell-to-cell fusion
spellingShingle Yue-Lin Yang
Fandan Meng
Pan Qin
Georg Herrler
Yao-Wei Huang
Yan-Dong Tang
Trypsin promotes porcine deltacoronavirus mediating cell-to-cell fusion in a cell type-dependent manner
Emerging Microbes and Infections
PDCoV
trypsin
entry
virus release
cell-to-cell fusion
title Trypsin promotes porcine deltacoronavirus mediating cell-to-cell fusion in a cell type-dependent manner
title_full Trypsin promotes porcine deltacoronavirus mediating cell-to-cell fusion in a cell type-dependent manner
title_fullStr Trypsin promotes porcine deltacoronavirus mediating cell-to-cell fusion in a cell type-dependent manner
title_full_unstemmed Trypsin promotes porcine deltacoronavirus mediating cell-to-cell fusion in a cell type-dependent manner
title_short Trypsin promotes porcine deltacoronavirus mediating cell-to-cell fusion in a cell type-dependent manner
title_sort trypsin promotes porcine deltacoronavirus mediating cell to cell fusion in a cell type dependent manner
topic PDCoV
trypsin
entry
virus release
cell-to-cell fusion
url https://www.tandfonline.com/doi/10.1080/22221751.2020.1730245
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