Role of the endothelial cell glycocalyx in sepsis-induced acute kidney injury

Sepsis-induced acute kidney injury (S-AKI) is a common complication of sepsis. It occurs at high incidence and is associated with a high level of mortality in the intensive care unit (ICU). The pathophysiologic mechanisms underlying S-AKI are complex, and include renal vascular endothelial cell dysf...

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Main Authors: Yixun Wang, Zhaohui Zhang, Xingguang Qu, Gaosheng Zhou
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-04-01
Series:Frontiers in Medicine
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Online Access:https://www.frontiersin.org/articles/10.3389/fmed.2025.1535673/full
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author Yixun Wang
Yixun Wang
Yixun Wang
Zhaohui Zhang
Zhaohui Zhang
Zhaohui Zhang
Xingguang Qu
Xingguang Qu
Xingguang Qu
Gaosheng Zhou
Gaosheng Zhou
Gaosheng Zhou
author_facet Yixun Wang
Yixun Wang
Yixun Wang
Zhaohui Zhang
Zhaohui Zhang
Zhaohui Zhang
Xingguang Qu
Xingguang Qu
Xingguang Qu
Gaosheng Zhou
Gaosheng Zhou
Gaosheng Zhou
author_sort Yixun Wang
collection DOAJ
description Sepsis-induced acute kidney injury (S-AKI) is a common complication of sepsis. It occurs at high incidence and is associated with a high level of mortality in the intensive care unit (ICU). The pathophysiologic mechanisms underlying S-AKI are complex, and include renal vascular endothelial cell dysfunction. The endothelial glycocalyx (EG) is a polysaccharide/protein complex located on the cell membrane at the luminal surface of vascular endothelial cells that has anti-inflammatory, anti-thrombotic, and endothelial protective effects. Recent studies have shown that glycocalyx damage plays a causal role in S-AKI progression. In this review, we first describe the structure, location, and basic function of the EG. Second, we analyze the underlying mechanisms of EG degradation in sepsis and S-AKI. Finally, we provide a summary of the potential therapeutic strategies that target the EG.
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issn 2296-858X
language English
publishDate 2025-04-01
publisher Frontiers Media S.A.
record_format Article
series Frontiers in Medicine
spelling doaj-art-95fbc7ebd7784f148c2d8b8300d35f762025-08-20T01:51:53ZengFrontiers Media S.A.Frontiers in Medicine2296-858X2025-04-011210.3389/fmed.2025.15356731535673Role of the endothelial cell glycocalyx in sepsis-induced acute kidney injuryYixun Wang0Yixun Wang1Yixun Wang2Zhaohui Zhang3Zhaohui Zhang4Zhaohui Zhang5Xingguang Qu6Xingguang Qu7Xingguang Qu8Gaosheng Zhou9Gaosheng Zhou10Gaosheng Zhou11The First College of Clinical Medical Science, China Three Gorges University, Yichang, ChinaDepartment of Critical Care Medicine, Yichang Central People's Hospital, Yichang, ChinaYichang Sepsis Clinical Research Center, Yichang, Hubei, ChinaThe First College of Clinical Medical Science, China Three Gorges University, Yichang, ChinaDepartment of Critical Care Medicine, Yichang Central People's Hospital, Yichang, ChinaYichang Sepsis Clinical Research Center, Yichang, Hubei, ChinaThe First College of Clinical Medical Science, China Three Gorges University, Yichang, ChinaDepartment of Critical Care Medicine, Yichang Central People's Hospital, Yichang, ChinaYichang Sepsis Clinical Research Center, Yichang, Hubei, ChinaThe First College of Clinical Medical Science, China Three Gorges University, Yichang, ChinaDepartment of Critical Care Medicine, Yichang Central People's Hospital, Yichang, ChinaYichang Sepsis Clinical Research Center, Yichang, Hubei, ChinaSepsis-induced acute kidney injury (S-AKI) is a common complication of sepsis. It occurs at high incidence and is associated with a high level of mortality in the intensive care unit (ICU). The pathophysiologic mechanisms underlying S-AKI are complex, and include renal vascular endothelial cell dysfunction. The endothelial glycocalyx (EG) is a polysaccharide/protein complex located on the cell membrane at the luminal surface of vascular endothelial cells that has anti-inflammatory, anti-thrombotic, and endothelial protective effects. Recent studies have shown that glycocalyx damage plays a causal role in S-AKI progression. In this review, we first describe the structure, location, and basic function of the EG. Second, we analyze the underlying mechanisms of EG degradation in sepsis and S-AKI. Finally, we provide a summary of the potential therapeutic strategies that target the EG.https://www.frontiersin.org/articles/10.3389/fmed.2025.1535673/fullglycocalyxsepsisendotheliumkidneyinflammation
spellingShingle Yixun Wang
Yixun Wang
Yixun Wang
Zhaohui Zhang
Zhaohui Zhang
Zhaohui Zhang
Xingguang Qu
Xingguang Qu
Xingguang Qu
Gaosheng Zhou
Gaosheng Zhou
Gaosheng Zhou
Role of the endothelial cell glycocalyx in sepsis-induced acute kidney injury
Frontiers in Medicine
glycocalyx
sepsis
endothelium
kidney
inflammation
title Role of the endothelial cell glycocalyx in sepsis-induced acute kidney injury
title_full Role of the endothelial cell glycocalyx in sepsis-induced acute kidney injury
title_fullStr Role of the endothelial cell glycocalyx in sepsis-induced acute kidney injury
title_full_unstemmed Role of the endothelial cell glycocalyx in sepsis-induced acute kidney injury
title_short Role of the endothelial cell glycocalyx in sepsis-induced acute kidney injury
title_sort role of the endothelial cell glycocalyx in sepsis induced acute kidney injury
topic glycocalyx
sepsis
endothelium
kidney
inflammation
url https://www.frontiersin.org/articles/10.3389/fmed.2025.1535673/full
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