Therapeutic Interventions against Inflammatory and Angiogenic Mediators in Proliferative Diabetic Retinopathy

The global prevalence of diabetes is estimated to be 336 million people, with diabetic complications contributing to significant worldwide morbidity and mortality. Diabetic retinopathy results from cumulative microvascular damage to the retina and inflammation is recognized as a critical driver of t...

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Main Authors: Daniel Gologorsky, Aristomenis Thanos, Demetrios Vavvas
Format: Article
Language:English
Published: Wiley 2012-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/2012/629452
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author Daniel Gologorsky
Aristomenis Thanos
Demetrios Vavvas
author_facet Daniel Gologorsky
Aristomenis Thanos
Demetrios Vavvas
author_sort Daniel Gologorsky
collection DOAJ
description The global prevalence of diabetes is estimated to be 336 million people, with diabetic complications contributing to significant worldwide morbidity and mortality. Diabetic retinopathy results from cumulative microvascular damage to the retina and inflammation is recognized as a critical driver of this disease process. This paper outlines the pathophysiology leading to proliferative diabetic retinopathy and highlights many of the inflammatory, angiogenic, and cytokine mediators implicated in the development and progression of this disease. We focus a detailed discussion on the current targeted therapeutic interventions used to treat diabetic retinopathy.
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spelling doaj-art-95c04b940d30467294ac8bd4b7b2cb2d2025-08-20T02:10:06ZengWileyMediators of Inflammation0962-93511466-18612012-01-01201210.1155/2012/629452629452Therapeutic Interventions against Inflammatory and Angiogenic Mediators in Proliferative Diabetic RetinopathyDaniel Gologorsky0Aristomenis Thanos1Demetrios Vavvas2Retina Service, Department of Ophthalmology, Massachusetts Eye and Ear Infirmary, Harvard Medical School, 243 Charles Street, Boston, MA 02131, USARetina Service, Department of Ophthalmology, Massachusetts Eye and Ear Infirmary, Harvard Medical School, 243 Charles Street, Boston, MA 02131, USARetina Service, Department of Ophthalmology, Massachusetts Eye and Ear Infirmary, Harvard Medical School, 243 Charles Street, Boston, MA 02131, USAThe global prevalence of diabetes is estimated to be 336 million people, with diabetic complications contributing to significant worldwide morbidity and mortality. Diabetic retinopathy results from cumulative microvascular damage to the retina and inflammation is recognized as a critical driver of this disease process. This paper outlines the pathophysiology leading to proliferative diabetic retinopathy and highlights many of the inflammatory, angiogenic, and cytokine mediators implicated in the development and progression of this disease. We focus a detailed discussion on the current targeted therapeutic interventions used to treat diabetic retinopathy.http://dx.doi.org/10.1155/2012/629452
spellingShingle Daniel Gologorsky
Aristomenis Thanos
Demetrios Vavvas
Therapeutic Interventions against Inflammatory and Angiogenic Mediators in Proliferative Diabetic Retinopathy
Mediators of Inflammation
title Therapeutic Interventions against Inflammatory and Angiogenic Mediators in Proliferative Diabetic Retinopathy
title_full Therapeutic Interventions against Inflammatory and Angiogenic Mediators in Proliferative Diabetic Retinopathy
title_fullStr Therapeutic Interventions against Inflammatory and Angiogenic Mediators in Proliferative Diabetic Retinopathy
title_full_unstemmed Therapeutic Interventions against Inflammatory and Angiogenic Mediators in Proliferative Diabetic Retinopathy
title_short Therapeutic Interventions against Inflammatory and Angiogenic Mediators in Proliferative Diabetic Retinopathy
title_sort therapeutic interventions against inflammatory and angiogenic mediators in proliferative diabetic retinopathy
url http://dx.doi.org/10.1155/2012/629452
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AT aristomenisthanos therapeuticinterventionsagainstinflammatoryandangiogenicmediatorsinproliferativediabeticretinopathy
AT demetriosvavvas therapeuticinterventionsagainstinflammatoryandangiogenicmediatorsinproliferativediabeticretinopathy