Mucin Overproduction in Chronic Inflammatory Lung Disease

Mucus overproduction and hypersecretion are commonly observed in chronic inflammatory lung disease. Mucins are gel-forming glycoproteins that can be stimulated by a variety of mediators. The present review addresses the mechanisms involved in the upregulation of secreted mucins. Mucin induction by n...

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Main Authors: Hans-Peter Hauber, Susan C Foley, Qutayba Hamid
Format: Article
Language:English
Published: Wiley 2006-01-01
Series:Canadian Respiratory Journal
Online Access:http://dx.doi.org/10.1155/2006/901417
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author Hans-Peter Hauber
Susan C Foley
Qutayba Hamid
author_facet Hans-Peter Hauber
Susan C Foley
Qutayba Hamid
author_sort Hans-Peter Hauber
collection DOAJ
description Mucus overproduction and hypersecretion are commonly observed in chronic inflammatory lung disease. Mucins are gel-forming glycoproteins that can be stimulated by a variety of mediators. The present review addresses the mechanisms involved in the upregulation of secreted mucins. Mucin induction by neutrophil elastase, bacteria, cytokines, growth factors, smoke and cystic fibrosis transmembrane conductance regulator malfunction are also discussed.
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institution Kabale University
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series Canadian Respiratory Journal
spelling doaj-art-93fcb393478e43f2b09faadc16665bbd2025-08-20T03:37:19ZengWileyCanadian Respiratory Journal1198-22412006-01-0113632733510.1155/2006/901417Mucin Overproduction in Chronic Inflammatory Lung DiseaseHans-Peter Hauber0Susan C Foley1Qutayba Hamid2Meakins-Christie Laboratories, McGill University, Montreal, Quebec, CanadaMeakins-Christie Laboratories, McGill University, Montreal, Quebec, CanadaMeakins-Christie Laboratories, McGill University, Montreal, Quebec, CanadaMucus overproduction and hypersecretion are commonly observed in chronic inflammatory lung disease. Mucins are gel-forming glycoproteins that can be stimulated by a variety of mediators. The present review addresses the mechanisms involved in the upregulation of secreted mucins. Mucin induction by neutrophil elastase, bacteria, cytokines, growth factors, smoke and cystic fibrosis transmembrane conductance regulator malfunction are also discussed.http://dx.doi.org/10.1155/2006/901417
spellingShingle Hans-Peter Hauber
Susan C Foley
Qutayba Hamid
Mucin Overproduction in Chronic Inflammatory Lung Disease
Canadian Respiratory Journal
title Mucin Overproduction in Chronic Inflammatory Lung Disease
title_full Mucin Overproduction in Chronic Inflammatory Lung Disease
title_fullStr Mucin Overproduction in Chronic Inflammatory Lung Disease
title_full_unstemmed Mucin Overproduction in Chronic Inflammatory Lung Disease
title_short Mucin Overproduction in Chronic Inflammatory Lung Disease
title_sort mucin overproduction in chronic inflammatory lung disease
url http://dx.doi.org/10.1155/2006/901417
work_keys_str_mv AT hanspeterhauber mucinoverproductioninchronicinflammatorylungdisease
AT susancfoley mucinoverproductioninchronicinflammatorylungdisease
AT qutaybahamid mucinoverproductioninchronicinflammatorylungdisease