m6A modification: a novel mechanism that regulates atherosclerosis via macrophage polarization

Atherosclerosis is a chronic vascular inflammatory disease in which macrophages play a pivotal role in modulating its pathology. In response to the intraplaque microenvironment, both pro-inflammatory M1 and anti-inflammatory M2 phenotypes of macrophages have the polarization capability, each influen...

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Main Authors: Xiaying Li, Hengkai Zhang, Yan Zhou, Lei Zhang, Ye Huang
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-06-01
Series:Frontiers in Immunology
Subjects:
Online Access:https://www.frontiersin.org/articles/10.3389/fimmu.2025.1607932/full
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author Xiaying Li
Xiaying Li
Hengkai Zhang
Yan Zhou
Lei Zhang
Ye Huang
Ye Huang
author_facet Xiaying Li
Xiaying Li
Hengkai Zhang
Yan Zhou
Lei Zhang
Ye Huang
Ye Huang
author_sort Xiaying Li
collection DOAJ
description Atherosclerosis is a chronic vascular inflammatory disease in which macrophages play a pivotal role in modulating its pathology. In response to the intraplaque microenvironment, both pro-inflammatory M1 and anti-inflammatory M2 phenotypes of macrophages have the polarization capability, each influencing the inflammatory state through the secretion of distinct cytokines. N6-methyladenosine (m6A) modification, the most prevalent internal chemical modification of RNA, significantly impacts various biological processes, including RNA transcription and protein expression. m6A modification acts as a critical determinant in macrophage polarization, with its molecular mechanisms intricately linked to the progression of atherosclerosis. This review aims to elucidate how different macrophage polarization phenotypes influence the progression of atherosclerosis while also exploring the significance of m6A modifications in this pathological context, thereby providing a theoretical foundation for identifying novel diagnostic and therapeutic targets for atherosclerosis.
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issn 1664-3224
language English
publishDate 2025-06-01
publisher Frontiers Media S.A.
record_format Article
series Frontiers in Immunology
spelling doaj-art-93e08f57f24a42f5bfacd5e20beeaf322025-08-20T02:07:41ZengFrontiers Media S.A.Frontiers in Immunology1664-32242025-06-011610.3389/fimmu.2025.16079321607932m6A modification: a novel mechanism that regulates atherosclerosis via macrophage polarizationXiaying Li0Xiaying Li1Hengkai Zhang2Yan Zhou3Lei Zhang4Ye Huang5Ye Huang6Graduate School, Beijing University of Chinese Medicine, Beijing, ChinaEmergency Department, Xiyuan Hospital, China Academy of Chinese Medical Sciences, Beijing, ChinaEmergency Department, Shanxi Traditional Chinese Medical Hospital, Shanxi, ChinaEmergency Department, Xiyuan Hospital, China Academy of Chinese Medical Sciences, Beijing, ChinaEmergency Department, Xiyuan Hospital, China Academy of Chinese Medical Sciences, Beijing, ChinaGraduate School, Beijing University of Chinese Medicine, Beijing, ChinaEmergency Department, Xiyuan Hospital, China Academy of Chinese Medical Sciences, Beijing, ChinaAtherosclerosis is a chronic vascular inflammatory disease in which macrophages play a pivotal role in modulating its pathology. In response to the intraplaque microenvironment, both pro-inflammatory M1 and anti-inflammatory M2 phenotypes of macrophages have the polarization capability, each influencing the inflammatory state through the secretion of distinct cytokines. N6-methyladenosine (m6A) modification, the most prevalent internal chemical modification of RNA, significantly impacts various biological processes, including RNA transcription and protein expression. m6A modification acts as a critical determinant in macrophage polarization, with its molecular mechanisms intricately linked to the progression of atherosclerosis. This review aims to elucidate how different macrophage polarization phenotypes influence the progression of atherosclerosis while also exploring the significance of m6A modifications in this pathological context, thereby providing a theoretical foundation for identifying novel diagnostic and therapeutic targets for atherosclerosis.https://www.frontiersin.org/articles/10.3389/fimmu.2025.1607932/fullmacrophage polarizationcytokinesatherosclerosisN 6 -methyladenosine modificationinflammatory
spellingShingle Xiaying Li
Xiaying Li
Hengkai Zhang
Yan Zhou
Lei Zhang
Ye Huang
Ye Huang
m6A modification: a novel mechanism that regulates atherosclerosis via macrophage polarization
Frontiers in Immunology
macrophage polarization
cytokines
atherosclerosis
N 6 -methyladenosine modification
inflammatory
title m6A modification: a novel mechanism that regulates atherosclerosis via macrophage polarization
title_full m6A modification: a novel mechanism that regulates atherosclerosis via macrophage polarization
title_fullStr m6A modification: a novel mechanism that regulates atherosclerosis via macrophage polarization
title_full_unstemmed m6A modification: a novel mechanism that regulates atherosclerosis via macrophage polarization
title_short m6A modification: a novel mechanism that regulates atherosclerosis via macrophage polarization
title_sort m6a modification a novel mechanism that regulates atherosclerosis via macrophage polarization
topic macrophage polarization
cytokines
atherosclerosis
N 6 -methyladenosine modification
inflammatory
url https://www.frontiersin.org/articles/10.3389/fimmu.2025.1607932/full
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