THE HEAT SHOCK PROTEIN 70-2 GENE (RS1061581) ASSOCIATES WITH THE ACUTE OTITIS MEDIUM IN CHILDREN

Acute otitis media (AOM) represents one of the most prevalent pediatric conditions worldwide. Affecting millions of children / adolescents each year, AOM constitutes a significant public health concern. Although the fundamental mechanisms underlying AOM development are understood, the molecular and...

Full description

Saved in:
Bibliographic Details
Main Authors: Л. Сидорчук, О. Саковець
Format: Article
Language:English
Published: Bukovynian State Medical University 2025-04-01
Series:Неонатологія, хірургія та перинатальна медицина
Subjects:
Online Access:https://neonatology.bsmu.edu.ua/article/view/326008
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850047027468042240
author Л. Сидорчук
О. Саковець
author_facet Л. Сидорчук
О. Саковець
author_sort Л. Сидорчук
collection DOAJ
description Acute otitis media (AOM) represents one of the most prevalent pediatric conditions worldwide. Affecting millions of children / adolescents each year, AOM constitutes a significant public health concern. Although the fundamental mechanisms underlying AOM development are understood, the molecular and genetic aspects of its pathogenesis remain insufficiently explored. The Aim of this study is to investigate the association between the HSP70-2 gene (rs1061581) A1267G polymorphism and AOM in children, as well as to evaluate the inheritance patterns of the disease and the risks associated with the severity of AOM in the studied cohort. Materials and Methods. A prospective cohort study was conducted involving 95 children with AOM, aged 7 to 18 years. The cohort comprised 34.74% (n=33) girls and 65.26% (n=62) boys. The study adhered to the principles of Good Clinical Practice (GCP), Good Laboratory Practice (GLP), and biomedical ethics standards for scientific research involving human participants. Informed consent was obtained from the parents or guardians of each child. Participants were stratified by age (7–11 years (n=81) and 12–18 years (n=14)), AOM severity (severe – 45.26% (n=43), non-severe – 54.74% (n=52)), and the nature of mucosal inflammation (catarrhal – 52.63% (n=50), purulent – 47.37% (n=45)). A control group of 50 healthy children was included, consisting of 20 girls (40.0%) and 30 boys (60.0%). The groups were age-matched (p>0.05). The HSP70-2 (rs1061581) gene polymorphism was analyzed using qualitative polymerase chain reaction. Risk assessment was performed using Risk Ratio, Odds Ratio, and 95% Confidence Intervals. Results. The mutation frequency of the heat shock protein gene HSP70-2 (rs1061581) in children with AOM from Northern Bukovina was 35.79% (8.42% in homozygous state), which was 11.79% higher than in healthy children (χ²=4.20; p=0.04). The distribution of HSP70-2 (rs1061581) genotypes in the studied cohort generally conformed to the Hardy-Weinberg equilibrium (χ²=1.36; p=0.243) and did not deviate in allele frequency from the average values observed in Caucasian populations (PA=0.24–0.36; PG=0.64–0.76). Binary logistic regression analysis proved a significant probability of AOM development in A-allele carriers' children under both dominant and additive models (OR=2.01; 95%CI: 1.01–4.06; p=0.05 and OR=2.01; 95%CI: 1.10–3.77; p=0.03), with the lowest out-of-sample Akaike prediction coefficients (AC=16.67 and 15.33). Epidemiological analysis confirmed an almost twofold increased risk of AOM in children with A-allele of the HSP70-2 gene (rs1061581) (OR=1.76; OR 95%CI: 1.02-3.05; p=0.026), with the lowest chances in G-allele carriers, especially in the homozygous state (OR=0.50; OR95%CI: 0.25-0.99; p=0.035). Furthermore, the risk of severe AOM increases more than 7 times for purulent discharge from the ear at the age of 7-11 years (OR=7.29; OR 95%CI: 1.44-37.01; p=0.045) with low chances of its development at the pubertal age (12-18 yo) (OR=0.14; OR 95%CI: 0.03-0.70; p=0.019). Conclusions. The A-allele of the HSP70-2 gene (rs1061581) is associated with an almost twofold increase in AOM risk in the studied pediatric cohort. The risk of severe AOM increases more than sevenfold in cases of purulent ear discharge among children aged 7-11 years
format Article
id doaj-art-926bab43d9ec4587bc33990b95d795c8
institution DOAJ
issn 2226-1230
2413-4260
language English
publishDate 2025-04-01
publisher Bukovynian State Medical University
record_format Article
series Неонатологія, хірургія та перинатальна медицина
spelling doaj-art-926bab43d9ec4587bc33990b95d795c82025-08-20T02:54:19ZengBukovynian State Medical UniversityНеонатологія, хірургія та перинатальна медицина2226-12302413-42602025-04-01151(55)10.24061/2413-4260.XV.1.55.2025.6THE HEAT SHOCK PROTEIN 70-2 GENE (RS1061581) ASSOCIATES WITH THE ACUTE OTITIS MEDIUM IN CHILDRENЛ. Сидорчук0О. Саковець1Bukovinian State Medical University Bukovinian State Medical University Acute otitis media (AOM) represents one of the most prevalent pediatric conditions worldwide. Affecting millions of children / adolescents each year, AOM constitutes a significant public health concern. Although the fundamental mechanisms underlying AOM development are understood, the molecular and genetic aspects of its pathogenesis remain insufficiently explored. The Aim of this study is to investigate the association between the HSP70-2 gene (rs1061581) A1267G polymorphism and AOM in children, as well as to evaluate the inheritance patterns of the disease and the risks associated with the severity of AOM in the studied cohort. Materials and Methods. A prospective cohort study was conducted involving 95 children with AOM, aged 7 to 18 years. The cohort comprised 34.74% (n=33) girls and 65.26% (n=62) boys. The study adhered to the principles of Good Clinical Practice (GCP), Good Laboratory Practice (GLP), and biomedical ethics standards for scientific research involving human participants. Informed consent was obtained from the parents or guardians of each child. Participants were stratified by age (7–11 years (n=81) and 12–18 years (n=14)), AOM severity (severe – 45.26% (n=43), non-severe – 54.74% (n=52)), and the nature of mucosal inflammation (catarrhal – 52.63% (n=50), purulent – 47.37% (n=45)). A control group of 50 healthy children was included, consisting of 20 girls (40.0%) and 30 boys (60.0%). The groups were age-matched (p>0.05). The HSP70-2 (rs1061581) gene polymorphism was analyzed using qualitative polymerase chain reaction. Risk assessment was performed using Risk Ratio, Odds Ratio, and 95% Confidence Intervals. Results. The mutation frequency of the heat shock protein gene HSP70-2 (rs1061581) in children with AOM from Northern Bukovina was 35.79% (8.42% in homozygous state), which was 11.79% higher than in healthy children (χ²=4.20; p=0.04). The distribution of HSP70-2 (rs1061581) genotypes in the studied cohort generally conformed to the Hardy-Weinberg equilibrium (χ²=1.36; p=0.243) and did not deviate in allele frequency from the average values observed in Caucasian populations (PA=0.24–0.36; PG=0.64–0.76). Binary logistic regression analysis proved a significant probability of AOM development in A-allele carriers' children under both dominant and additive models (OR=2.01; 95%CI: 1.01–4.06; p=0.05 and OR=2.01; 95%CI: 1.10–3.77; p=0.03), with the lowest out-of-sample Akaike prediction coefficients (AC=16.67 and 15.33). Epidemiological analysis confirmed an almost twofold increased risk of AOM in children with A-allele of the HSP70-2 gene (rs1061581) (OR=1.76; OR 95%CI: 1.02-3.05; p=0.026), with the lowest chances in G-allele carriers, especially in the homozygous state (OR=0.50; OR95%CI: 0.25-0.99; p=0.035). Furthermore, the risk of severe AOM increases more than 7 times for purulent discharge from the ear at the age of 7-11 years (OR=7.29; OR 95%CI: 1.44-37.01; p=0.045) with low chances of its development at the pubertal age (12-18 yo) (OR=0.14; OR 95%CI: 0.03-0.70; p=0.019). Conclusions. The A-allele of the HSP70-2 gene (rs1061581) is associated with an almost twofold increase in AOM risk in the studied pediatric cohort. The risk of severe AOM increases more than sevenfold in cases of purulent ear discharge among children aged 7-11 years https://neonatology.bsmu.edu.ua/article/view/326008Acute Otitis Media; HSP70-2 gene (rs1061581); Children; Heat Shock Protein; Risks; Inflammation; Disease severity
spellingShingle Л. Сидорчук
О. Саковець
THE HEAT SHOCK PROTEIN 70-2 GENE (RS1061581) ASSOCIATES WITH THE ACUTE OTITIS MEDIUM IN CHILDREN
Неонатологія, хірургія та перинатальна медицина
Acute Otitis Media; HSP70-2 gene (rs1061581); Children; Heat Shock Protein; Risks; Inflammation; Disease severity
title THE HEAT SHOCK PROTEIN 70-2 GENE (RS1061581) ASSOCIATES WITH THE ACUTE OTITIS MEDIUM IN CHILDREN
title_full THE HEAT SHOCK PROTEIN 70-2 GENE (RS1061581) ASSOCIATES WITH THE ACUTE OTITIS MEDIUM IN CHILDREN
title_fullStr THE HEAT SHOCK PROTEIN 70-2 GENE (RS1061581) ASSOCIATES WITH THE ACUTE OTITIS MEDIUM IN CHILDREN
title_full_unstemmed THE HEAT SHOCK PROTEIN 70-2 GENE (RS1061581) ASSOCIATES WITH THE ACUTE OTITIS MEDIUM IN CHILDREN
title_short THE HEAT SHOCK PROTEIN 70-2 GENE (RS1061581) ASSOCIATES WITH THE ACUTE OTITIS MEDIUM IN CHILDREN
title_sort heat shock protein 70 2 gene rs1061581 associates with the acute otitis medium in children
topic Acute Otitis Media; HSP70-2 gene (rs1061581); Children; Heat Shock Protein; Risks; Inflammation; Disease severity
url https://neonatology.bsmu.edu.ua/article/view/326008
work_keys_str_mv AT lsidorčuk theheatshockprotein702geners1061581associateswiththeacuteotitismediuminchildren
AT osakovecʹ theheatshockprotein702geners1061581associateswiththeacuteotitismediuminchildren
AT lsidorčuk heatshockprotein702geners1061581associateswiththeacuteotitismediuminchildren
AT osakovecʹ heatshockprotein702geners1061581associateswiththeacuteotitismediuminchildren