An Endocrine-Disrupting Chemical, Bisphenol A Diglycidyl Ether (BADGE), Accelerates Neuritogenesis and Outgrowth of Cortical Neurons via the G-Protein-Coupled Estrogen Receptor

Bisphenol A diglycidyl ether (BADGE) is the main component of epoxy resin and is used for the inner coating of canned foods and plastic food containers. BADGE can easily migrate from containers and result in food contamination; the compound is known as an endocrine-disrupting chemical. We previously...

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Main Authors: Ikuko Miyazaki, Chiharu Nishiyama, Takeru Nagoshi, Akane Miyako, Suzuka Ono, Ichika Misawa, Aika Isse, Kana Tomimoto, Kaori Masai, Kazumasa Zensho, Masato Asanuma
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Language:English
Published: MDPI AG 2025-06-01
Series:NeuroSci
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Online Access:https://www.mdpi.com/2673-4087/6/2/53
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author Ikuko Miyazaki
Chiharu Nishiyama
Takeru Nagoshi
Akane Miyako
Suzuka Ono
Ichika Misawa
Aika Isse
Kana Tomimoto
Kaori Masai
Kazumasa Zensho
Masato Asanuma
author_facet Ikuko Miyazaki
Chiharu Nishiyama
Takeru Nagoshi
Akane Miyako
Suzuka Ono
Ichika Misawa
Aika Isse
Kana Tomimoto
Kaori Masai
Kazumasa Zensho
Masato Asanuma
author_sort Ikuko Miyazaki
collection DOAJ
description Bisphenol A diglycidyl ether (BADGE) is the main component of epoxy resin and is used for the inner coating of canned foods and plastic food containers. BADGE can easily migrate from containers and result in food contamination; the compound is known as an endocrine-disrupting chemical. We previously reported that maternal exposure to bisphenol A bis (2,3-dihydroxypropyl) ether (BADGE·2H<sub>2</sub>O), which is the most detected BADGE derivative not only in canned foods but also in human specimens, during gestation and lactation, could accelerate neuronal differentiation in the cortex of fetuses and induce anxiety-like behavior in juvenile mice. In this study, we investigated the effects of low-dose BADGE·2H<sub>2</sub>O (1–100 pM) treatment on neurites and the mechanism of neurite outgrowth in cortical neurons. BADGE·2H<sub>2</sub>O exposure significantly increased the number of dendrites and neurite length in cortical neurons; these accelerating effects were inhibited by estrogen receptor (ER) antagonist ICI 182,780 and G-protein-coupled estrogen receptor (GPER) antagonist G15. BADGE·2H<sub>2</sub>O down-regulated <i>Hes1</i> expression, which is a transcriptional repressor, and increased levels of neuritogenic factor neurogenin-3 (Ngn3) in the cortical neurons; the changes were significantly blocked by G15. These data suggest that direct BADGE·2H<sub>2</sub>O exposure can accelerate neuritogenesis and outgrowth in cortical neurons through down-regulation of Hes1 and by increasing Ngn3 levels through ERs, particularly GPER.
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spelling doaj-art-91ba6f944bef4e929e1269831f8897db2025-08-20T03:27:40ZengMDPI AGNeuroSci2673-40872025-06-01625310.3390/neurosci6020053An Endocrine-Disrupting Chemical, Bisphenol A Diglycidyl Ether (BADGE), Accelerates Neuritogenesis and Outgrowth of Cortical Neurons via the G-Protein-Coupled Estrogen ReceptorIkuko Miyazaki0Chiharu Nishiyama1Takeru Nagoshi2Akane Miyako3Suzuka Ono4Ichika Misawa5Aika Isse6Kana Tomimoto7Kaori Masai8Kazumasa Zensho9Masato Asanuma10Department of Medical Neurobiology, Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama 700-8558, JapanDepartment of Medical Neurobiology, Okayama University Medical School, Okayama 700-8558, JapanDepartment of Medical Neurobiology, Okayama University Medical School, Okayama 700-8558, JapanDepartment of Medical Neurobiology, Okayama University Medical School, Okayama 700-8558, JapanDepartment of Medical Neurobiology, Okayama University Medical School, Okayama 700-8558, JapanDepartment of Medical Neurobiology, Okayama University Medical School, Okayama 700-8558, JapanDepartment of Medical Neurobiology, Okayama University Medical School, Okayama 700-8558, JapanDepartment of Medical Neurobiology, Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama 700-8558, JapanDepartment of Medical Neurobiology, Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama 700-8558, JapanDepartment of Medical Neurobiology, Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama 700-8558, JapanDepartment of Medical Neurobiology, Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama 700-8558, JapanBisphenol A diglycidyl ether (BADGE) is the main component of epoxy resin and is used for the inner coating of canned foods and plastic food containers. BADGE can easily migrate from containers and result in food contamination; the compound is known as an endocrine-disrupting chemical. We previously reported that maternal exposure to bisphenol A bis (2,3-dihydroxypropyl) ether (BADGE·2H<sub>2</sub>O), which is the most detected BADGE derivative not only in canned foods but also in human specimens, during gestation and lactation, could accelerate neuronal differentiation in the cortex of fetuses and induce anxiety-like behavior in juvenile mice. In this study, we investigated the effects of low-dose BADGE·2H<sub>2</sub>O (1–100 pM) treatment on neurites and the mechanism of neurite outgrowth in cortical neurons. BADGE·2H<sub>2</sub>O exposure significantly increased the number of dendrites and neurite length in cortical neurons; these accelerating effects were inhibited by estrogen receptor (ER) antagonist ICI 182,780 and G-protein-coupled estrogen receptor (GPER) antagonist G15. BADGE·2H<sub>2</sub>O down-regulated <i>Hes1</i> expression, which is a transcriptional repressor, and increased levels of neuritogenic factor neurogenin-3 (Ngn3) in the cortical neurons; the changes were significantly blocked by G15. These data suggest that direct BADGE·2H<sub>2</sub>O exposure can accelerate neuritogenesis and outgrowth in cortical neurons through down-regulation of Hes1 and by increasing Ngn3 levels through ERs, particularly GPER.https://www.mdpi.com/2673-4087/6/2/53BADGEneurite outgrowthestrogen receptorGPERHes1neurogenin-3
spellingShingle Ikuko Miyazaki
Chiharu Nishiyama
Takeru Nagoshi
Akane Miyako
Suzuka Ono
Ichika Misawa
Aika Isse
Kana Tomimoto
Kaori Masai
Kazumasa Zensho
Masato Asanuma
An Endocrine-Disrupting Chemical, Bisphenol A Diglycidyl Ether (BADGE), Accelerates Neuritogenesis and Outgrowth of Cortical Neurons via the G-Protein-Coupled Estrogen Receptor
NeuroSci
BADGE
neurite outgrowth
estrogen receptor
GPER
Hes1
neurogenin-3
title An Endocrine-Disrupting Chemical, Bisphenol A Diglycidyl Ether (BADGE), Accelerates Neuritogenesis and Outgrowth of Cortical Neurons via the G-Protein-Coupled Estrogen Receptor
title_full An Endocrine-Disrupting Chemical, Bisphenol A Diglycidyl Ether (BADGE), Accelerates Neuritogenesis and Outgrowth of Cortical Neurons via the G-Protein-Coupled Estrogen Receptor
title_fullStr An Endocrine-Disrupting Chemical, Bisphenol A Diglycidyl Ether (BADGE), Accelerates Neuritogenesis and Outgrowth of Cortical Neurons via the G-Protein-Coupled Estrogen Receptor
title_full_unstemmed An Endocrine-Disrupting Chemical, Bisphenol A Diglycidyl Ether (BADGE), Accelerates Neuritogenesis and Outgrowth of Cortical Neurons via the G-Protein-Coupled Estrogen Receptor
title_short An Endocrine-Disrupting Chemical, Bisphenol A Diglycidyl Ether (BADGE), Accelerates Neuritogenesis and Outgrowth of Cortical Neurons via the G-Protein-Coupled Estrogen Receptor
title_sort endocrine disrupting chemical bisphenol a diglycidyl ether badge accelerates neuritogenesis and outgrowth of cortical neurons via the g protein coupled estrogen receptor
topic BADGE
neurite outgrowth
estrogen receptor
GPER
Hes1
neurogenin-3
url https://www.mdpi.com/2673-4087/6/2/53
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