Toxin-Induced and Genetic Animal Models of Parkinson's Disease

Parkinson's disease (PD) is a common progressive neurodegenerative disorder. The major pathological hallmarks of PD are the selective loss of nigrostriatal dopaminergic neurons and the presence of intraneuronal aggregates termed Lewy bodies (LBs), but the pathophysiological mechanisms are not f...

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Main Authors: Shin Hisahara, Shun Shimohama
Format: Article
Language:English
Published: Wiley 2011-01-01
Series:Parkinson's Disease
Online Access:http://dx.doi.org/10.4061/2011/951709
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author Shin Hisahara
Shun Shimohama
author_facet Shin Hisahara
Shun Shimohama
author_sort Shin Hisahara
collection DOAJ
description Parkinson's disease (PD) is a common progressive neurodegenerative disorder. The major pathological hallmarks of PD are the selective loss of nigrostriatal dopaminergic neurons and the presence of intraneuronal aggregates termed Lewy bodies (LBs), but the pathophysiological mechanisms are not fully understood. Epidemiologically, environmental neurotoxins such as pesticides are promising candidates for causative factors of PD. Oxidative stress and mitochondrial dysfunction induced by these toxins could contribute to the progression of PD. While most cases of PD are sporadic, specific mutations in genes that cause familial forms of PD have led to provide new insights into its pathogenesis. This paper focuses on animal models of both toxin-induced and genetically determined PD that have provided significant insight for understanding this disease. We also discuss the validity, benefits, and limitations of representative models.
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spelling doaj-art-9147f209706146d4be0e474b971364cf2025-08-20T02:09:44ZengWileyParkinson's Disease2042-00802011-01-01201110.4061/2011/951709951709Toxin-Induced and Genetic Animal Models of Parkinson's DiseaseShin Hisahara0Shun Shimohama1Department of Neurology, Sapporo Medical University, South1, West17, chuo-ku, Sapporo 060-8556, JapanDepartment of Neurology, Sapporo Medical University, South1, West17, chuo-ku, Sapporo 060-8556, JapanParkinson's disease (PD) is a common progressive neurodegenerative disorder. The major pathological hallmarks of PD are the selective loss of nigrostriatal dopaminergic neurons and the presence of intraneuronal aggregates termed Lewy bodies (LBs), but the pathophysiological mechanisms are not fully understood. Epidemiologically, environmental neurotoxins such as pesticides are promising candidates for causative factors of PD. Oxidative stress and mitochondrial dysfunction induced by these toxins could contribute to the progression of PD. While most cases of PD are sporadic, specific mutations in genes that cause familial forms of PD have led to provide new insights into its pathogenesis. This paper focuses on animal models of both toxin-induced and genetically determined PD that have provided significant insight for understanding this disease. We also discuss the validity, benefits, and limitations of representative models.http://dx.doi.org/10.4061/2011/951709
spellingShingle Shin Hisahara
Shun Shimohama
Toxin-Induced and Genetic Animal Models of Parkinson's Disease
Parkinson's Disease
title Toxin-Induced and Genetic Animal Models of Parkinson's Disease
title_full Toxin-Induced and Genetic Animal Models of Parkinson's Disease
title_fullStr Toxin-Induced and Genetic Animal Models of Parkinson's Disease
title_full_unstemmed Toxin-Induced and Genetic Animal Models of Parkinson's Disease
title_short Toxin-Induced and Genetic Animal Models of Parkinson's Disease
title_sort toxin induced and genetic animal models of parkinson s disease
url http://dx.doi.org/10.4061/2011/951709
work_keys_str_mv AT shinhisahara toxininducedandgeneticanimalmodelsofparkinsonsdisease
AT shunshimohama toxininducedandgeneticanimalmodelsofparkinsonsdisease