Antioxidant-supplemented media modulates ROS by regulating complex I during mouse oocyte maturation

Abstract In Vitro Oocyte Maturation (IVM) is a technique used to mature oocytes in laboratory setting. However, IVM can lead to an imbalance of reactive oxygen species (ROS), which can damage the oocytes. To prevent this, antioxidants are added to the culture medium. How these antioxidants affect Co...

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Main Authors: Nazlican Bozdemir, Ceren Cakir, Ozgur Cinar, Fatma Uysal Cinar
Format: Article
Language:English
Published: Nature Portfolio 2025-07-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-025-08056-5
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author Nazlican Bozdemir
Ceren Cakir
Ozgur Cinar
Fatma Uysal Cinar
author_facet Nazlican Bozdemir
Ceren Cakir
Ozgur Cinar
Fatma Uysal Cinar
author_sort Nazlican Bozdemir
collection DOAJ
description Abstract In Vitro Oocyte Maturation (IVM) is a technique used to mature oocytes in laboratory setting. However, IVM can lead to an imbalance of reactive oxygen species (ROS), which can damage the oocytes. To prevent this, antioxidants are added to the culture medium. How these antioxidants affect Complex I, a crucial ROS-producing protein within the mitochondrial membrane, remains uncertain. To address this gap, our study aimed to achieve two key objectives. First, we investigated, for the first time, the Complex I expression during mouse oogenesis. Second, we examined the influence of an antioxidant-containing medium on Complex I and ROS levels. Germinal vesicle (GV)-stage oocytes were incubated in culture media containing acetyl-l-carnitine (ALC), α-lipoic acid (ALA), MitoQ, N-acetyl-l-cysteine (NAC) until the metaphase I (MI) and metaphase II (MII) stages. Complex I and ROS levels increased in MI and MII oocytes. Additionally, ALA and NAC increased Complex I and ROS levels, while MitoQ decreased these levels in MI and MII oocytes. Interestingly, ALC did not affect MI oocytes, but decreased the Complex I and ROS levels in MII oocytes. By elucidating the interplay between antioxidants, Complex I, and ROS during oogenesis, we pave the way for future research to improve female fertility.
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institution Kabale University
issn 2045-2322
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publishDate 2025-07-01
publisher Nature Portfolio
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series Scientific Reports
spelling doaj-art-90e2f37276b84888bbd774c97d9900c72025-08-20T03:45:24ZengNature PortfolioScientific Reports2045-23222025-07-0115111110.1038/s41598-025-08056-5Antioxidant-supplemented media modulates ROS by regulating complex I during mouse oocyte maturationNazlican Bozdemir0Ceren Cakir1Ozgur Cinar2Fatma Uysal Cinar3Department of Histology and Embryology, Ankara Medipol University School of MedicineDepartment of Histology and Embryology, Ankara Medipol University School of MedicineDepartment of Histology and Embryology, Ankara University School of MedicineDepartment of Histology and Embryology, Ankara Medipol University School of MedicineAbstract In Vitro Oocyte Maturation (IVM) is a technique used to mature oocytes in laboratory setting. However, IVM can lead to an imbalance of reactive oxygen species (ROS), which can damage the oocytes. To prevent this, antioxidants are added to the culture medium. How these antioxidants affect Complex I, a crucial ROS-producing protein within the mitochondrial membrane, remains uncertain. To address this gap, our study aimed to achieve two key objectives. First, we investigated, for the first time, the Complex I expression during mouse oogenesis. Second, we examined the influence of an antioxidant-containing medium on Complex I and ROS levels. Germinal vesicle (GV)-stage oocytes were incubated in culture media containing acetyl-l-carnitine (ALC), α-lipoic acid (ALA), MitoQ, N-acetyl-l-cysteine (NAC) until the metaphase I (MI) and metaphase II (MII) stages. Complex I and ROS levels increased in MI and MII oocytes. Additionally, ALA and NAC increased Complex I and ROS levels, while MitoQ decreased these levels in MI and MII oocytes. Interestingly, ALC did not affect MI oocytes, but decreased the Complex I and ROS levels in MII oocytes. By elucidating the interplay between antioxidants, Complex I, and ROS during oogenesis, we pave the way for future research to improve female fertility.https://doi.org/10.1038/s41598-025-08056-5Complex IROSAntioxidantsMitochondriaOogenesis
spellingShingle Nazlican Bozdemir
Ceren Cakir
Ozgur Cinar
Fatma Uysal Cinar
Antioxidant-supplemented media modulates ROS by regulating complex I during mouse oocyte maturation
Scientific Reports
Complex I
ROS
Antioxidants
Mitochondria
Oogenesis
title Antioxidant-supplemented media modulates ROS by regulating complex I during mouse oocyte maturation
title_full Antioxidant-supplemented media modulates ROS by regulating complex I during mouse oocyte maturation
title_fullStr Antioxidant-supplemented media modulates ROS by regulating complex I during mouse oocyte maturation
title_full_unstemmed Antioxidant-supplemented media modulates ROS by regulating complex I during mouse oocyte maturation
title_short Antioxidant-supplemented media modulates ROS by regulating complex I during mouse oocyte maturation
title_sort antioxidant supplemented media modulates ros by regulating complex i during mouse oocyte maturation
topic Complex I
ROS
Antioxidants
Mitochondria
Oogenesis
url https://doi.org/10.1038/s41598-025-08056-5
work_keys_str_mv AT nazlicanbozdemir antioxidantsupplementedmediamodulatesrosbyregulatingcomplexiduringmouseoocytematuration
AT cerencakir antioxidantsupplementedmediamodulatesrosbyregulatingcomplexiduringmouseoocytematuration
AT ozgurcinar antioxidantsupplementedmediamodulatesrosbyregulatingcomplexiduringmouseoocytematuration
AT fatmauysalcinar antioxidantsupplementedmediamodulatesrosbyregulatingcomplexiduringmouseoocytematuration