CETP Lowers TLR4 Expression Which Attenuates the Inflammatory Response Induced by LPS and Polymicrobial Sepsis

Sepsis is a systemic inflammatory response to infection eliciting high mortality rate which is a serious health problem. Despite numerous studies seeking for therapeutic alternatives, the mechanisms involved in this disease remain elusive. In this study we evaluated the influence of cholesteryl este...

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Main Authors: Tatiana Martins Venancio, Roberta Marcondes Machado, Angela Castoldi, Mariane Tami Amano, Valeria Sutti Nunes, Eder Carlos Rocha Quintao, Niels Olsen Saraiva Camara, Francisco Garcia Soriano, Patrícia Miralda Cazita
Format: Article
Language:English
Published: Wiley 2016-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/2016/1784014
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author Tatiana Martins Venancio
Roberta Marcondes Machado
Angela Castoldi
Mariane Tami Amano
Valeria Sutti Nunes
Eder Carlos Rocha Quintao
Niels Olsen Saraiva Camara
Francisco Garcia Soriano
Patrícia Miralda Cazita
author_facet Tatiana Martins Venancio
Roberta Marcondes Machado
Angela Castoldi
Mariane Tami Amano
Valeria Sutti Nunes
Eder Carlos Rocha Quintao
Niels Olsen Saraiva Camara
Francisco Garcia Soriano
Patrícia Miralda Cazita
author_sort Tatiana Martins Venancio
collection DOAJ
description Sepsis is a systemic inflammatory response to infection eliciting high mortality rate which is a serious health problem. Despite numerous studies seeking for therapeutic alternatives, the mechanisms involved in this disease remain elusive. In this study we evaluated the influence of cholesteryl ester transfer protein (CETP), a glycoprotein that promotes the transfer of lipids between lipoproteins, on the inflammatory response in mice. Human CETP transgenic mice were compared to control mice (wild type, WT) after polymicrobial sepsis induced by cecal ligation and puncture (CLP), aiming at investigating their survival rate and inflammatory profiles. Macrophages from the peritoneal cavity were stimulated with LPS in the presence or absence of recombinant CETP for phenotypic and functional studies. In comparison to WT mice, CETP mice showed higher survival rate, lower IL-6 plasma concentration, and decreased liver toll-like receptor 4 (TLR4) and acyloxyacyl hydrolase (AOAH) protein. Moreover, macrophages from WT mice to which recombinant human CETP was added decreased LPS uptake, TLR4 expression, NF-κB activation and IL-6 secretion. This raises the possibility for new therapeutic tools in sepsis while suggesting that lowering CETP by pharmacological inhibitors should be inconvenient in the context of sepsis and infectious diseases.
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spelling doaj-art-90049180dcf5424dbe5cd5b5a9c8ab872025-08-20T03:37:19ZengWileyMediators of Inflammation0962-93511466-18612016-01-01201610.1155/2016/17840141784014CETP Lowers TLR4 Expression Which Attenuates the Inflammatory Response Induced by LPS and Polymicrobial SepsisTatiana Martins Venancio0Roberta Marcondes Machado1Angela Castoldi2Mariane Tami Amano3Valeria Sutti Nunes4Eder Carlos Rocha Quintao5Niels Olsen Saraiva Camara6Francisco Garcia Soriano7Patrícia Miralda Cazita8Lipids Laboratory (LIM 10), Faculty of Medical Sciences, The University of São Paulo, São Paulo, SP, BrazilLipids Laboratory (LIM 10), Faculty of Medical Sciences, The University of São Paulo, São Paulo, SP, BrazilLaboratory of Transplantation Immunobiology, Department of Immunology, Institute of Biomedical Science IV, University of São Paulo, São Paulo, SP, BrazilLaboratory of Transplantation Immunobiology, Department of Immunology, Institute of Biomedical Science IV, University of São Paulo, São Paulo, SP, BrazilLipids Laboratory (LIM 10), Faculty of Medical Sciences, The University of São Paulo, São Paulo, SP, BrazilLipids Laboratory (LIM 10), Faculty of Medical Sciences, The University of São Paulo, São Paulo, SP, BrazilLaboratory of Transplantation Immunobiology, Department of Immunology, Institute of Biomedical Science IV, University of São Paulo, São Paulo, SP, BrazilEmergency Care Research Unit Laboratory (LIM 51), Faculty of Medical Sciences, The University of São Paulo, São Paulo, SP, BrazilLipids Laboratory (LIM 10), Faculty of Medical Sciences, The University of São Paulo, São Paulo, SP, BrazilSepsis is a systemic inflammatory response to infection eliciting high mortality rate which is a serious health problem. Despite numerous studies seeking for therapeutic alternatives, the mechanisms involved in this disease remain elusive. In this study we evaluated the influence of cholesteryl ester transfer protein (CETP), a glycoprotein that promotes the transfer of lipids between lipoproteins, on the inflammatory response in mice. Human CETP transgenic mice were compared to control mice (wild type, WT) after polymicrobial sepsis induced by cecal ligation and puncture (CLP), aiming at investigating their survival rate and inflammatory profiles. Macrophages from the peritoneal cavity were stimulated with LPS in the presence or absence of recombinant CETP for phenotypic and functional studies. In comparison to WT mice, CETP mice showed higher survival rate, lower IL-6 plasma concentration, and decreased liver toll-like receptor 4 (TLR4) and acyloxyacyl hydrolase (AOAH) protein. Moreover, macrophages from WT mice to which recombinant human CETP was added decreased LPS uptake, TLR4 expression, NF-κB activation and IL-6 secretion. This raises the possibility for new therapeutic tools in sepsis while suggesting that lowering CETP by pharmacological inhibitors should be inconvenient in the context of sepsis and infectious diseases.http://dx.doi.org/10.1155/2016/1784014
spellingShingle Tatiana Martins Venancio
Roberta Marcondes Machado
Angela Castoldi
Mariane Tami Amano
Valeria Sutti Nunes
Eder Carlos Rocha Quintao
Niels Olsen Saraiva Camara
Francisco Garcia Soriano
Patrícia Miralda Cazita
CETP Lowers TLR4 Expression Which Attenuates the Inflammatory Response Induced by LPS and Polymicrobial Sepsis
Mediators of Inflammation
title CETP Lowers TLR4 Expression Which Attenuates the Inflammatory Response Induced by LPS and Polymicrobial Sepsis
title_full CETP Lowers TLR4 Expression Which Attenuates the Inflammatory Response Induced by LPS and Polymicrobial Sepsis
title_fullStr CETP Lowers TLR4 Expression Which Attenuates the Inflammatory Response Induced by LPS and Polymicrobial Sepsis
title_full_unstemmed CETP Lowers TLR4 Expression Which Attenuates the Inflammatory Response Induced by LPS and Polymicrobial Sepsis
title_short CETP Lowers TLR4 Expression Which Attenuates the Inflammatory Response Induced by LPS and Polymicrobial Sepsis
title_sort cetp lowers tlr4 expression which attenuates the inflammatory response induced by lps and polymicrobial sepsis
url http://dx.doi.org/10.1155/2016/1784014
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