Mitochondrial fission produces a Warburg effect via the oxidative inhibition of prolyl hydroxylase domain-2

Excessive mitochondrial fission and a shift to a Warburg phenotype are hallmarks of pulmonary hypertension (PH), although the mechanistic link between these processes remains unclear. We show that in pulmonary arterial endothelial cells (PAEC), Drp1 overexpression induces mitochondrial fission and i...

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Main Authors: Xutong Sun, Manivannan Yegambaram, Qing Lu, Alejandro E. Garcia Flores, Marissa D. Pokharel, Jamie Soto, Saurabh Aggarwal, Ting Wang, Jeffrey R. Fineman, Stephen M. Black
Format: Article
Language:English
Published: Elsevier 2025-04-01
Series:Redox Biology
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Online Access:http://www.sciencedirect.com/science/article/pii/S2213231725000424
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author Xutong Sun
Manivannan Yegambaram
Qing Lu
Alejandro E. Garcia Flores
Marissa D. Pokharel
Jamie Soto
Saurabh Aggarwal
Ting Wang
Jeffrey R. Fineman
Stephen M. Black
author_facet Xutong Sun
Manivannan Yegambaram
Qing Lu
Alejandro E. Garcia Flores
Marissa D. Pokharel
Jamie Soto
Saurabh Aggarwal
Ting Wang
Jeffrey R. Fineman
Stephen M. Black
author_sort Xutong Sun
collection DOAJ
description Excessive mitochondrial fission and a shift to a Warburg phenotype are hallmarks of pulmonary hypertension (PH), although the mechanistic link between these processes remains unclear. We show that in pulmonary arterial endothelial cells (PAEC), Drp1 overexpression induces mitochondrial fission and increases glycolytic ATP production and glycolysis. This is due to mitochondrial reactive oxygen species (mito-ROS)-mediated activation of hypoxia-inducible factor-1α (HIF-1α) signaling, and this is linked to hydrogen peroxide (H2O2)-mediated inhibition of prolyl hydroxylase domain-2 (PHD2) due to its cysteine 326 oxidation and dimerization. Furthermore, these findings are validated in PAEC isolated from a lamb model of PH, which are glycolytic (Shunt PAEC), exhibit increases in both H2O2 and PHD2 dimer levels. The overexpression of catalase reversed the PHD2 dimerization, decreased HIF-1α levels, and attenuated glycolysis in Shunt PAEC. Our data suggest that reducing PHD2 dimerization could be a potential therapeutic target for PH.
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publishDate 2025-04-01
publisher Elsevier
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series Redox Biology
spelling doaj-art-8f42a596f7874ab38d2b2b79836e37ae2025-08-20T03:02:56ZengElsevierRedox Biology2213-23172025-04-018110352910.1016/j.redox.2025.103529Mitochondrial fission produces a Warburg effect via the oxidative inhibition of prolyl hydroxylase domain-2Xutong Sun0Manivannan Yegambaram1Qing Lu2Alejandro E. Garcia Flores3Marissa D. Pokharel4Jamie Soto5Saurabh Aggarwal6Ting Wang7Jeffrey R. Fineman8Stephen M. Black9Florida International University, Center for Translational Science, Port Saint Lucie, FL, 34987, USAFlorida International University, Center for Translational Science, Port Saint Lucie, FL, 34987, USAFlorida International University, Center for Translational Science, Port Saint Lucie, FL, 34987, USAFlorida International University, Center for Translational Science, Port Saint Lucie, FL, 34987, USAFlorida International University, Center for Translational Science, Port Saint Lucie, FL, 34987, USA; The Departments of Cellular & Molecular Medicine, Herbert Wertheim College of Medicine, Florida International University, Miami, FL, 33199, USAFlorida International University, Center for Translational Science, Port Saint Lucie, FL, 34987, USAThe Departments of Cellular & Molecular Medicine, Herbert Wertheim College of Medicine, Florida International University, Miami, FL, 33199, USAFlorida International University, Center for Translational Science, Port Saint Lucie, FL, 34987, USA; The Departments of Environmental Health Sciences, Robert Stempel College of Public Health and Social Work, University of California San Francisco, San Francisco, CA, 94143, USAThe Department of Pediatrics, University of California San Francisco, San Francisco, CA, 94143, USA; The Cardiovascular Research Institute, University of California San Francisco, San Francisco, CA, 94143, USAFlorida International University, Center for Translational Science, Port Saint Lucie, FL, 34987, USA; The Departments of Cellular & Molecular Medicine, Herbert Wertheim College of Medicine, Florida International University, Miami, FL, 33199, USA; The Departments of Environmental Health Sciences, Robert Stempel College of Public Health and Social Work, University of California San Francisco, San Francisco, CA, 94143, USA; Corresponding author. FIU-Center for Translational Science, Florida International University, Port Saint Lucie, FL, 34987, USA.Excessive mitochondrial fission and a shift to a Warburg phenotype are hallmarks of pulmonary hypertension (PH), although the mechanistic link between these processes remains unclear. We show that in pulmonary arterial endothelial cells (PAEC), Drp1 overexpression induces mitochondrial fission and increases glycolytic ATP production and glycolysis. This is due to mitochondrial reactive oxygen species (mito-ROS)-mediated activation of hypoxia-inducible factor-1α (HIF-1α) signaling, and this is linked to hydrogen peroxide (H2O2)-mediated inhibition of prolyl hydroxylase domain-2 (PHD2) due to its cysteine 326 oxidation and dimerization. Furthermore, these findings are validated in PAEC isolated from a lamb model of PH, which are glycolytic (Shunt PAEC), exhibit increases in both H2O2 and PHD2 dimer levels. The overexpression of catalase reversed the PHD2 dimerization, decreased HIF-1α levels, and attenuated glycolysis in Shunt PAEC. Our data suggest that reducing PHD2 dimerization could be a potential therapeutic target for PH.http://www.sciencedirect.com/science/article/pii/S2213231725000424Mitochondrial fissionWarburg effectDrp1HIF-1 alphaPHD2Hydrogen peroxide
spellingShingle Xutong Sun
Manivannan Yegambaram
Qing Lu
Alejandro E. Garcia Flores
Marissa D. Pokharel
Jamie Soto
Saurabh Aggarwal
Ting Wang
Jeffrey R. Fineman
Stephen M. Black
Mitochondrial fission produces a Warburg effect via the oxidative inhibition of prolyl hydroxylase domain-2
Redox Biology
Mitochondrial fission
Warburg effect
Drp1
HIF-1 alpha
PHD2
Hydrogen peroxide
title Mitochondrial fission produces a Warburg effect via the oxidative inhibition of prolyl hydroxylase domain-2
title_full Mitochondrial fission produces a Warburg effect via the oxidative inhibition of prolyl hydroxylase domain-2
title_fullStr Mitochondrial fission produces a Warburg effect via the oxidative inhibition of prolyl hydroxylase domain-2
title_full_unstemmed Mitochondrial fission produces a Warburg effect via the oxidative inhibition of prolyl hydroxylase domain-2
title_short Mitochondrial fission produces a Warburg effect via the oxidative inhibition of prolyl hydroxylase domain-2
title_sort mitochondrial fission produces a warburg effect via the oxidative inhibition of prolyl hydroxylase domain 2
topic Mitochondrial fission
Warburg effect
Drp1
HIF-1 alpha
PHD2
Hydrogen peroxide
url http://www.sciencedirect.com/science/article/pii/S2213231725000424
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