p75NTR/proBDNF Modulates Basal Cell Carcinoma (BCC) Immune Microenvironment via Necroptosis Signaling Pathway

Basal cell carcinoma (BCC) is the most common skin cancer. While most of the basal cell carcinomas were localized lesion and can be easily managed, the treatment options to the advanced basal cell carcinomas are still remarkably limited. In recent years, proBDNF and its receptor p75NTR have been rep...

Full description

Saved in:
Bibliographic Details
Main Authors: Qingli Lu, Yuanyuan Qu, Yuan Ding, Xiaojing Kang
Format: Article
Language:English
Published: Wiley 2021-01-01
Series:Journal of Immunology Research
Online Access:http://dx.doi.org/10.1155/2021/6652846
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850213109534294016
author Qingli Lu
Yuanyuan Qu
Yuan Ding
Xiaojing Kang
author_facet Qingli Lu
Yuanyuan Qu
Yuan Ding
Xiaojing Kang
author_sort Qingli Lu
collection DOAJ
description Basal cell carcinoma (BCC) is the most common skin cancer. While most of the basal cell carcinomas were localized lesion and can be easily managed, the treatment options to the advanced basal cell carcinomas are still remarkably limited. In recent years, proBDNF and its receptor p75NTR have been reported to play important roles in various diseases, including cancers and psychotic disorders. However, the role of p75NTR/proBDNF signaling in basal cell carcinoma remains unclear. Here, we found that the expression level of p75NTR/proBDNF was decreased in basal cell carcinoma patient samples and cell lines. In vitro study showed overexpression of p75NTR/proBDNF could significantly facilitate tumor cell death, including inflammatory-silent apoptosis and lytic inflammatory activated necroptosis. In vivo study showed overexpression of p75NTR/proBDNF dramatically promotes tumor-associated macrophage (M1) and T cell recruitment in a syngeneic mouse model of BCC. These results show a crucial role for p75NTR/proBDNF signaling in basal cell carcinoma immune microenvironment.
format Article
id doaj-art-8bb113b4ff7c4bb7aedb18f4b6f085d3
institution OA Journals
issn 2314-8861
2314-7156
language English
publishDate 2021-01-01
publisher Wiley
record_format Article
series Journal of Immunology Research
spelling doaj-art-8bb113b4ff7c4bb7aedb18f4b6f085d32025-08-20T02:09:11ZengWileyJournal of Immunology Research2314-88612314-71562021-01-01202110.1155/2021/66528466652846p75NTR/proBDNF Modulates Basal Cell Carcinoma (BCC) Immune Microenvironment via Necroptosis Signaling PathwayQingli Lu0Yuanyuan Qu1Yuan Ding2Xiaojing Kang3Department of Dermatology and Venereology, People’s Hospital of Xinjiang Uygur Autonomous Region, Urumqi 830000, ChinaDepartment of Dermatology and Venereology, People’s Hospital of Xinjiang Uygur Autonomous Region, Urumqi 830000, ChinaDepartment of Dermatology and Venereology, People’s Hospital of Xinjiang Uygur Autonomous Region, Urumqi 830000, ChinaDepartment of Dermatology and Venereology, People’s Hospital of Xinjiang Uygur Autonomous Region, Urumqi 830000, ChinaBasal cell carcinoma (BCC) is the most common skin cancer. While most of the basal cell carcinomas were localized lesion and can be easily managed, the treatment options to the advanced basal cell carcinomas are still remarkably limited. In recent years, proBDNF and its receptor p75NTR have been reported to play important roles in various diseases, including cancers and psychotic disorders. However, the role of p75NTR/proBDNF signaling in basal cell carcinoma remains unclear. Here, we found that the expression level of p75NTR/proBDNF was decreased in basal cell carcinoma patient samples and cell lines. In vitro study showed overexpression of p75NTR/proBDNF could significantly facilitate tumor cell death, including inflammatory-silent apoptosis and lytic inflammatory activated necroptosis. In vivo study showed overexpression of p75NTR/proBDNF dramatically promotes tumor-associated macrophage (M1) and T cell recruitment in a syngeneic mouse model of BCC. These results show a crucial role for p75NTR/proBDNF signaling in basal cell carcinoma immune microenvironment.http://dx.doi.org/10.1155/2021/6652846
spellingShingle Qingli Lu
Yuanyuan Qu
Yuan Ding
Xiaojing Kang
p75NTR/proBDNF Modulates Basal Cell Carcinoma (BCC) Immune Microenvironment via Necroptosis Signaling Pathway
Journal of Immunology Research
title p75NTR/proBDNF Modulates Basal Cell Carcinoma (BCC) Immune Microenvironment via Necroptosis Signaling Pathway
title_full p75NTR/proBDNF Modulates Basal Cell Carcinoma (BCC) Immune Microenvironment via Necroptosis Signaling Pathway
title_fullStr p75NTR/proBDNF Modulates Basal Cell Carcinoma (BCC) Immune Microenvironment via Necroptosis Signaling Pathway
title_full_unstemmed p75NTR/proBDNF Modulates Basal Cell Carcinoma (BCC) Immune Microenvironment via Necroptosis Signaling Pathway
title_short p75NTR/proBDNF Modulates Basal Cell Carcinoma (BCC) Immune Microenvironment via Necroptosis Signaling Pathway
title_sort p75ntr probdnf modulates basal cell carcinoma bcc immune microenvironment via necroptosis signaling pathway
url http://dx.doi.org/10.1155/2021/6652846
work_keys_str_mv AT qinglilu p75ntrprobdnfmodulatesbasalcellcarcinomabccimmunemicroenvironmentvianecroptosissignalingpathway
AT yuanyuanqu p75ntrprobdnfmodulatesbasalcellcarcinomabccimmunemicroenvironmentvianecroptosissignalingpathway
AT yuanding p75ntrprobdnfmodulatesbasalcellcarcinomabccimmunemicroenvironmentvianecroptosissignalingpathway
AT xiaojingkang p75ntrprobdnfmodulatesbasalcellcarcinomabccimmunemicroenvironmentvianecroptosissignalingpathway