Cerebral circulation in hypertensive encephalopathy and chronic heart failure

Aim. To compare cerebral circulation in chronic hypertensive encephalopathy (HE) with and without chronic heart failure (CHF).Material and methods. In total, 122 patients with Stage I-III HE, but free from occlusive carotid disease, were examined. Duplex scanning was used to measure volume blood flo...

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Main Authors: L. A. Geraskina, T. N. Sharypova, V. V. Mashin, V. Vl. Mashin, A. V. Fonyakin, Z. A. Suslina
Format: Article
Language:Russian
Published: «SILICEA-POLIGRAF» LLC 2009-10-01
Series:Кардиоваскулярная терапия и профилактика
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Online Access:https://cardiovascular.elpub.ru/jour/article/view/1852
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author L. A. Geraskina
T. N. Sharypova
V. V. Mashin
V. Vl. Mashin
A. V. Fonyakin
Z. A. Suslina
author_facet L. A. Geraskina
T. N. Sharypova
V. V. Mashin
V. Vl. Mashin
A. V. Fonyakin
Z. A. Suslina
author_sort L. A. Geraskina
collection DOAJ
description Aim. To compare cerebral circulation in chronic hypertensive encephalopathy (HE) with and without chronic heart failure (CHF).Material and methods. In total, 122 patients with Stage I-III HE, but free from occlusive carotid disease, were examined. Duplex scanning was used to measure volume blood flow in common carotid arteries (CCA), vertebral arteries (VA), and middle cerebral arteries (MCA). Single photon emission computer tomography was used for cortical cerebral perfusion (CCP) assessment.Results. Stage I diastolic CHF was diagnosed in 37 patients (30%), and Stage II diastolic CHF — in 68 (56%). Regardless from CHF presence, HE was characterised by unchanged CCA and VA hemodynamics, reduced flow velocity in MCA, and increased MCA resistance parameters. Compared to CHF-free patients, those with Stage ICHF demonstrated increased frontal CCP (p<0,05) and higher prevalence of diffuse leukoaraiosis. This reflected selective deterioration of subcortical perfusion, due to progressing atherosclerosis of penetrating cerebral arteries, which supply deep brain tissue. Compared to Stage I CHF, Stage II CHF was characterised by additional blood flow reduction and resistance index increase in MCA, CCP reduction (p<0,1), and leukoaraiosis prevalence of 40% (p<0,02).Conclusion. In HE patients, Stage II CHF was associated with reduced cortical and subcortical brain tissue per­fusion and therefore could be regarded as a marker of diffuse hypertensive remodelling of cerebral vessels.
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language Russian
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publisher «SILICEA-POLIGRAF» LLC
record_format Article
series Кардиоваскулярная терапия и профилактика
spelling doaj-art-8b77b2b387f444d4b590dde2fb403a5c2025-08-20T03:43:27Zrus«SILICEA-POLIGRAF» LLCКардиоваскулярная терапия и профилактика1728-88002619-01252009-10-018528321567Cerebral circulation in hypertensive encephalopathy and chronic heart failureL. A. Geraskina0T. N. Sharypova1V. V. Mashin2V. Vl. Mashin3A. V. Fonyakin4Z. A. Suslina5Research Neurology Centre, Russian Academy of Medical SciencesResearch Neurology Centre, Russian Academy of Medical SciencesUlyanovsk State UniversityUlyanovsk State UniversityResearch Neurology Centre, Russian Academy of Medical SciencesResearch Neurology Centre, Russian Academy of Medical SciencesAim. To compare cerebral circulation in chronic hypertensive encephalopathy (HE) with and without chronic heart failure (CHF).Material and methods. In total, 122 patients with Stage I-III HE, but free from occlusive carotid disease, were examined. Duplex scanning was used to measure volume blood flow in common carotid arteries (CCA), vertebral arteries (VA), and middle cerebral arteries (MCA). Single photon emission computer tomography was used for cortical cerebral perfusion (CCP) assessment.Results. Stage I diastolic CHF was diagnosed in 37 patients (30%), and Stage II diastolic CHF — in 68 (56%). Regardless from CHF presence, HE was characterised by unchanged CCA and VA hemodynamics, reduced flow velocity in MCA, and increased MCA resistance parameters. Compared to CHF-free patients, those with Stage ICHF demonstrated increased frontal CCP (p<0,05) and higher prevalence of diffuse leukoaraiosis. This reflected selective deterioration of subcortical perfusion, due to progressing atherosclerosis of penetrating cerebral arteries, which supply deep brain tissue. Compared to Stage I CHF, Stage II CHF was characterised by additional blood flow reduction and resistance index increase in MCA, CCP reduction (p<0,1), and leukoaraiosis prevalence of 40% (p<0,02).Conclusion. In HE patients, Stage II CHF was associated with reduced cortical and subcortical brain tissue per­fusion and therefore could be regarded as a marker of diffuse hypertensive remodelling of cerebral vessels.https://cardiovascular.elpub.ru/jour/article/view/1852cerebral circulationhypertensive encephalopathychronic heart failure
spellingShingle L. A. Geraskina
T. N. Sharypova
V. V. Mashin
V. Vl. Mashin
A. V. Fonyakin
Z. A. Suslina
Cerebral circulation in hypertensive encephalopathy and chronic heart failure
Кардиоваскулярная терапия и профилактика
cerebral circulation
hypertensive encephalopathy
chronic heart failure
title Cerebral circulation in hypertensive encephalopathy and chronic heart failure
title_full Cerebral circulation in hypertensive encephalopathy and chronic heart failure
title_fullStr Cerebral circulation in hypertensive encephalopathy and chronic heart failure
title_full_unstemmed Cerebral circulation in hypertensive encephalopathy and chronic heart failure
title_short Cerebral circulation in hypertensive encephalopathy and chronic heart failure
title_sort cerebral circulation in hypertensive encephalopathy and chronic heart failure
topic cerebral circulation
hypertensive encephalopathy
chronic heart failure
url https://cardiovascular.elpub.ru/jour/article/view/1852
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AT tnsharypova cerebralcirculationinhypertensiveencephalopathyandchronicheartfailure
AT vvmashin cerebralcirculationinhypertensiveencephalopathyandchronicheartfailure
AT vvlmashin cerebralcirculationinhypertensiveencephalopathyandchronicheartfailure
AT avfonyakin cerebralcirculationinhypertensiveencephalopathyandchronicheartfailure
AT zasuslina cerebralcirculationinhypertensiveencephalopathyandchronicheartfailure