PPAR-Alpha Agonist Used at the Acute Phase of Experimental Ischemic Stroke Reduces Occurrence of Thrombolysis-Induced Hemorrhage in Rats

The impact of fenofibrate, a peroxisome proliferator-activated receptor-alpha (PPAR-α) agonist, on the risk of thrombolysis-induced hemorrhage during the acute phase of stroke in a rat model of stroke was studied. One-hour middle cerebral artery occlusion followed by thrombolysis with tissue plasmin...

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Main Authors: Sophie Gautier, Thavarak Ouk, Maud Pétrault, Olivier Pétrault, Vincent Bérézowski, Régis Bordet
Format: Article
Language:English
Published: Wiley 2015-01-01
Series:PPAR Research
Online Access:http://dx.doi.org/10.1155/2015/246329
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author Sophie Gautier
Thavarak Ouk
Maud Pétrault
Olivier Pétrault
Vincent Bérézowski
Régis Bordet
author_facet Sophie Gautier
Thavarak Ouk
Maud Pétrault
Olivier Pétrault
Vincent Bérézowski
Régis Bordet
author_sort Sophie Gautier
collection DOAJ
description The impact of fenofibrate, a peroxisome proliferator-activated receptor-alpha (PPAR-α) agonist, on the risk of thrombolysis-induced hemorrhage during the acute phase of stroke in a rat model of stroke was studied. One-hour middle cerebral artery occlusion followed by thrombolysis with tissue plasminogen activator was made in rats receiving either fenofibrate or vehicle for 72 h after stroke. Evaluation of infarct, hemorrhage, middle cerebral artery vasoreactivity, and immunochemistry (CD11b for microglial activation, myeloperoxidase, and ICAM-1 for neutrophil infiltration) was performed. The PPAR-alpha agonist significantly reduced the risk of hemorrhage after thrombolysis in parallel with a decrease in the infarct volume and in the stroke-induced vascular endothelial dysfunction. These effects are concomitant with a reduction in microglial activation and neutrophil infiltration in infarct area. Our results strengthen the idea that using drugs such as fenofibrate, with pleiotropic properties due to PPAR-alpha agonism, may be of value to reduce thrombolysis-induced hemorrhage during acute stroke.
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institution Kabale University
issn 1687-4757
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publishDate 2015-01-01
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spelling doaj-art-8a59ee8437524526b834b56274b2aa672025-08-20T03:37:50ZengWileyPPAR Research1687-47571687-47652015-01-01201510.1155/2015/246329246329PPAR-Alpha Agonist Used at the Acute Phase of Experimental Ischemic Stroke Reduces Occurrence of Thrombolysis-Induced Hemorrhage in RatsSophie Gautier0Thavarak Ouk1Maud Pétrault2Olivier Pétrault3Vincent Bérézowski4Régis Bordet5U1171, Departement de Pharmacologie Medicale, University Lille Nord de France, Faculté de Médecine, CHU Lille, 1 Place de Verdun, 59037 Lille Cedex, FranceU1171, Departement de Pharmacologie Medicale, University Lille Nord de France, Faculté de Médecine, CHU Lille, 1 Place de Verdun, 59037 Lille Cedex, FranceU1171, Departement de Pharmacologie Medicale, University Lille Nord de France, Faculté de Médecine, CHU Lille, 1 Place de Verdun, 59037 Lille Cedex, FranceU1171, Departement de Pharmacologie Medicale, University Lille Nord de France, Faculté de Médecine, CHU Lille, 1 Place de Verdun, 59037 Lille Cedex, FranceU1171, Departement de Pharmacologie Medicale, University Lille Nord de France, Faculté de Médecine, CHU Lille, 1 Place de Verdun, 59037 Lille Cedex, FranceU1171, Departement de Pharmacologie Medicale, University Lille Nord de France, Faculté de Médecine, CHU Lille, 1 Place de Verdun, 59037 Lille Cedex, FranceThe impact of fenofibrate, a peroxisome proliferator-activated receptor-alpha (PPAR-α) agonist, on the risk of thrombolysis-induced hemorrhage during the acute phase of stroke in a rat model of stroke was studied. One-hour middle cerebral artery occlusion followed by thrombolysis with tissue plasminogen activator was made in rats receiving either fenofibrate or vehicle for 72 h after stroke. Evaluation of infarct, hemorrhage, middle cerebral artery vasoreactivity, and immunochemistry (CD11b for microglial activation, myeloperoxidase, and ICAM-1 for neutrophil infiltration) was performed. The PPAR-alpha agonist significantly reduced the risk of hemorrhage after thrombolysis in parallel with a decrease in the infarct volume and in the stroke-induced vascular endothelial dysfunction. These effects are concomitant with a reduction in microglial activation and neutrophil infiltration in infarct area. Our results strengthen the idea that using drugs such as fenofibrate, with pleiotropic properties due to PPAR-alpha agonism, may be of value to reduce thrombolysis-induced hemorrhage during acute stroke.http://dx.doi.org/10.1155/2015/246329
spellingShingle Sophie Gautier
Thavarak Ouk
Maud Pétrault
Olivier Pétrault
Vincent Bérézowski
Régis Bordet
PPAR-Alpha Agonist Used at the Acute Phase of Experimental Ischemic Stroke Reduces Occurrence of Thrombolysis-Induced Hemorrhage in Rats
PPAR Research
title PPAR-Alpha Agonist Used at the Acute Phase of Experimental Ischemic Stroke Reduces Occurrence of Thrombolysis-Induced Hemorrhage in Rats
title_full PPAR-Alpha Agonist Used at the Acute Phase of Experimental Ischemic Stroke Reduces Occurrence of Thrombolysis-Induced Hemorrhage in Rats
title_fullStr PPAR-Alpha Agonist Used at the Acute Phase of Experimental Ischemic Stroke Reduces Occurrence of Thrombolysis-Induced Hemorrhage in Rats
title_full_unstemmed PPAR-Alpha Agonist Used at the Acute Phase of Experimental Ischemic Stroke Reduces Occurrence of Thrombolysis-Induced Hemorrhage in Rats
title_short PPAR-Alpha Agonist Used at the Acute Phase of Experimental Ischemic Stroke Reduces Occurrence of Thrombolysis-Induced Hemorrhage in Rats
title_sort ppar alpha agonist used at the acute phase of experimental ischemic stroke reduces occurrence of thrombolysis induced hemorrhage in rats
url http://dx.doi.org/10.1155/2015/246329
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