Serum Cytokeratin 18 M30 Levels in Chronic Hepatitis B Reflect Both Phase and Histological Activities of Disease

Chronic hepatitis B has highly a dynamic course with significant fluctuations of HBV-DNA and ALT impeding assessment of disease activity. New biomarkers of inflammatory versus noninflammatory stages of HBV infection are urgently needed. Cytokeratin 18 epitope M30 (M30 CK-18) is a sensitive marker of...

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Main Authors: Magdalena Świderska, Jerzy Jaroszewicz, Anna Parfieniuk-Kowerda, Magdalena Rogalska-Płońska, Agnieszka Stawicka, Anatol Panasiuk, Robert Flisiak
Format: Article
Language:English
Published: Wiley 2017-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/2017/3480234
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author Magdalena Świderska
Jerzy Jaroszewicz
Anna Parfieniuk-Kowerda
Magdalena Rogalska-Płońska
Agnieszka Stawicka
Anatol Panasiuk
Robert Flisiak
author_facet Magdalena Świderska
Jerzy Jaroszewicz
Anna Parfieniuk-Kowerda
Magdalena Rogalska-Płońska
Agnieszka Stawicka
Anatol Panasiuk
Robert Flisiak
author_sort Magdalena Świderska
collection DOAJ
description Chronic hepatitis B has highly a dynamic course with significant fluctuations of HBV-DNA and ALT impeding assessment of disease activity. New biomarkers of inflammatory versus noninflammatory stages of HBV infection are urgently needed. Cytokeratin 18 epitope M30 (M30 CK-18) is a sensitive marker of cell death. We aimed to investigate an association between serum M30 CK-18 and histological activity and phase of HBV infection. 150 Caucasian patients with HBV-infection were included in the study. Serum M30 CK-18 levels reflected phase of disease, being significantly higher in both HBeAg(+) and HBeAg(−) hepatitis B in comparison to HBsAg(+) carrier groups. The highest serum M30 CK-18 levels were observed in subjects with the most advanced stages of HBV. Moreover, its serum concentrations correlated with both inflammatory activity and fibrosis advancement (ANOVA P<0.001). Importantly, serum M30 CK-18 levels were able to discriminate patients with mild versus moderate-advanced fibrosis (AUC: 0.86) and mild versus active liver inflammation (AUC: 0.79). M30 CK-18 serum concentration has good sensitivity and specificity in discriminating mild versus moderate/severe fibrosis and inflammation even in patients with normal ALT activity. This study suggests M30 CK-18 as a potential noninvasive marker of disease activity and also a marker of phase of persistent HBV infection.
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institution Kabale University
issn 0962-9351
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publishDate 2017-01-01
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spelling doaj-art-885293c900ef41bcb1d18331e694c3782025-08-20T03:38:06ZengWileyMediators of Inflammation0962-93511466-18612017-01-01201710.1155/2017/34802343480234Serum Cytokeratin 18 M30 Levels in Chronic Hepatitis B Reflect Both Phase and Histological Activities of DiseaseMagdalena Świderska0Jerzy Jaroszewicz1Anna Parfieniuk-Kowerda2Magdalena Rogalska-Płońska3Agnieszka Stawicka4Anatol Panasiuk5Robert Flisiak6Department of Infectious Diseases and Hepatology, Medical University of Bialystok, Bialystok, PolandDepartment of Infectious Diseases and Hepatology, Medical University of Bialystok, Bialystok, PolandDepartment of Infectious Diseases and Hepatology, Medical University of Bialystok, Bialystok, PolandDepartment of Infectious Diseases and Hepatology, Medical University of Bialystok, Bialystok, PolandDepartment of Infectious Diseases and Hepatology, Medical University of Bialystok, Bialystok, PolandDepartment of Infectious Diseases and Hepatology, Medical University of Bialystok, Bialystok, PolandDepartment of Infectious Diseases and Hepatology, Medical University of Bialystok, Bialystok, PolandChronic hepatitis B has highly a dynamic course with significant fluctuations of HBV-DNA and ALT impeding assessment of disease activity. New biomarkers of inflammatory versus noninflammatory stages of HBV infection are urgently needed. Cytokeratin 18 epitope M30 (M30 CK-18) is a sensitive marker of cell death. We aimed to investigate an association between serum M30 CK-18 and histological activity and phase of HBV infection. 150 Caucasian patients with HBV-infection were included in the study. Serum M30 CK-18 levels reflected phase of disease, being significantly higher in both HBeAg(+) and HBeAg(−) hepatitis B in comparison to HBsAg(+) carrier groups. The highest serum M30 CK-18 levels were observed in subjects with the most advanced stages of HBV. Moreover, its serum concentrations correlated with both inflammatory activity and fibrosis advancement (ANOVA P<0.001). Importantly, serum M30 CK-18 levels were able to discriminate patients with mild versus moderate-advanced fibrosis (AUC: 0.86) and mild versus active liver inflammation (AUC: 0.79). M30 CK-18 serum concentration has good sensitivity and specificity in discriminating mild versus moderate/severe fibrosis and inflammation even in patients with normal ALT activity. This study suggests M30 CK-18 as a potential noninvasive marker of disease activity and also a marker of phase of persistent HBV infection.http://dx.doi.org/10.1155/2017/3480234
spellingShingle Magdalena Świderska
Jerzy Jaroszewicz
Anna Parfieniuk-Kowerda
Magdalena Rogalska-Płońska
Agnieszka Stawicka
Anatol Panasiuk
Robert Flisiak
Serum Cytokeratin 18 M30 Levels in Chronic Hepatitis B Reflect Both Phase and Histological Activities of Disease
Mediators of Inflammation
title Serum Cytokeratin 18 M30 Levels in Chronic Hepatitis B Reflect Both Phase and Histological Activities of Disease
title_full Serum Cytokeratin 18 M30 Levels in Chronic Hepatitis B Reflect Both Phase and Histological Activities of Disease
title_fullStr Serum Cytokeratin 18 M30 Levels in Chronic Hepatitis B Reflect Both Phase and Histological Activities of Disease
title_full_unstemmed Serum Cytokeratin 18 M30 Levels in Chronic Hepatitis B Reflect Both Phase and Histological Activities of Disease
title_short Serum Cytokeratin 18 M30 Levels in Chronic Hepatitis B Reflect Both Phase and Histological Activities of Disease
title_sort serum cytokeratin 18 m30 levels in chronic hepatitis b reflect both phase and histological activities of disease
url http://dx.doi.org/10.1155/2017/3480234
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