S‐ketamine Alleviates Neuroinflammation and Attenuates Lipopolysaccharide‐Induced Depression Via Targeting SIRT2

Abstract Depression, a pervasive mental health condition, has increasingly been linked to neuroinflammation, as evidenced by elevated levels of pro‐inflammatory markers such as TNF‐α and IL‐1β observed in patients, which underscores the role of inflammation in its pathophysiology. This study investi...

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Main Authors: Cong Lin, Xiaoxuan Zhou, Mingqi Li, Cong Zhang, Haojiang Zhai, Haohong Li, Hongshuang Wang, Xiaohui Wang
Format: Article
Language:English
Published: Wiley 2025-06-01
Series:Advanced Science
Subjects:
Online Access:https://doi.org/10.1002/advs.202416481
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author Cong Lin
Xiaoxuan Zhou
Mingqi Li
Cong Zhang
Haojiang Zhai
Haohong Li
Hongshuang Wang
Xiaohui Wang
author_facet Cong Lin
Xiaoxuan Zhou
Mingqi Li
Cong Zhang
Haojiang Zhai
Haohong Li
Hongshuang Wang
Xiaohui Wang
author_sort Cong Lin
collection DOAJ
description Abstract Depression, a pervasive mental health condition, has increasingly been linked to neuroinflammation, as evidenced by elevated levels of pro‐inflammatory markers such as TNF‐α and IL‐1β observed in patients, which underscores the role of inflammation in its pathophysiology. This study investigates the differential effects of S‐ketamine (S‐KET) and R‐ketamine (R‐KET) on inflammation‐induced depression using a lipopolysaccharide (LPS)‐induced mouse model. Results showed that S‐KET, but not R‐KET, significantly alleviated depressive‐like behaviors and reduced levels of pro‐inflammatory factors in the medial prefrontal cortex (mPFC). Activity‐based protein profiling identified SIRT2 as a key intracellular target of S‐KET, with direct binding observed at the Q167 residue, whereas R‐KET showed no such binding. S‐KET enhanced SIRT2 interaction with NF‐κB subunit p65, reducing its acetylation and suppressing pro‐inflammatory gene expression, effects not seen with R‐KET. In vitro studies with RNA interference and the SIRT2 inhibitor AK‐7, along with in vivo pharmacological blockade, confirmed that SIRT2 is crucial for the anti‐inflammatory and antidepressant actions of S‐KET. These findings suggest that SIRT2 mediates the therapeutic effects of S‐KET, highlighting its potential as a target for treating inflammation‐associated depression. This study provides novel insights into the stereospecific actions of ketamine enantiomers and the promise of targeting SIRT2 for neuroinflammatory depression.
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spelling doaj-art-87e946a64cf142d5bf5e27ccca54766c2025-08-20T03:22:15ZengWileyAdvanced Science2198-38442025-06-011223n/an/a10.1002/advs.202416481S‐ketamine Alleviates Neuroinflammation and Attenuates Lipopolysaccharide‐Induced Depression Via Targeting SIRT2Cong Lin0Xiaoxuan Zhou1Mingqi Li2Cong Zhang3Haojiang Zhai4Haohong Li5Hongshuang Wang6Xiaohui Wang7Laboratory of Chemical Biology Changchun Institute of Applied Chemistry Chinese Academy of Sciences Changchun Jilin 130022 ChinaLaboratory of Chemical Biology Changchun Institute of Applied Chemistry Chinese Academy of Sciences Changchun Jilin 130022 ChinaLaboratory of Chemical Biology Changchun Institute of Applied Chemistry Chinese Academy of Sciences Changchun Jilin 130022 ChinaLaboratory of Chemical Biology Changchun Institute of Applied Chemistry Chinese Academy of Sciences Changchun Jilin 130022 ChinaLaboratory of Chemical Biology Changchun Institute of Applied Chemistry Chinese Academy of Sciences Changchun Jilin 130022 ChinaThe MOE Frontier Research Center of Brain and Brain‐Machine Integration Zhejiang University School of Brain Science and Brain Medicine Hangzhou Zhejiang 310058 ChinaLaboratory of Chemical Biology Changchun Institute of Applied Chemistry Chinese Academy of Sciences Changchun Jilin 130022 ChinaLaboratory of Chemical Biology Changchun Institute of Applied Chemistry Chinese Academy of Sciences Changchun Jilin 130022 ChinaAbstract Depression, a pervasive mental health condition, has increasingly been linked to neuroinflammation, as evidenced by elevated levels of pro‐inflammatory markers such as TNF‐α and IL‐1β observed in patients, which underscores the role of inflammation in its pathophysiology. This study investigates the differential effects of S‐ketamine (S‐KET) and R‐ketamine (R‐KET) on inflammation‐induced depression using a lipopolysaccharide (LPS)‐induced mouse model. Results showed that S‐KET, but not R‐KET, significantly alleviated depressive‐like behaviors and reduced levels of pro‐inflammatory factors in the medial prefrontal cortex (mPFC). Activity‐based protein profiling identified SIRT2 as a key intracellular target of S‐KET, with direct binding observed at the Q167 residue, whereas R‐KET showed no such binding. S‐KET enhanced SIRT2 interaction with NF‐κB subunit p65, reducing its acetylation and suppressing pro‐inflammatory gene expression, effects not seen with R‐KET. In vitro studies with RNA interference and the SIRT2 inhibitor AK‐7, along with in vivo pharmacological blockade, confirmed that SIRT2 is crucial for the anti‐inflammatory and antidepressant actions of S‐KET. These findings suggest that SIRT2 mediates the therapeutic effects of S‐KET, highlighting its potential as a target for treating inflammation‐associated depression. This study provides novel insights into the stereospecific actions of ketamine enantiomers and the promise of targeting SIRT2 for neuroinflammatory depression.https://doi.org/10.1002/advs.202416481DepressionNeuroinflammationR‐ketamineSIRT2S‐ketamine
spellingShingle Cong Lin
Xiaoxuan Zhou
Mingqi Li
Cong Zhang
Haojiang Zhai
Haohong Li
Hongshuang Wang
Xiaohui Wang
S‐ketamine Alleviates Neuroinflammation and Attenuates Lipopolysaccharide‐Induced Depression Via Targeting SIRT2
Advanced Science
Depression
Neuroinflammation
R‐ketamine
SIRT2
S‐ketamine
title S‐ketamine Alleviates Neuroinflammation and Attenuates Lipopolysaccharide‐Induced Depression Via Targeting SIRT2
title_full S‐ketamine Alleviates Neuroinflammation and Attenuates Lipopolysaccharide‐Induced Depression Via Targeting SIRT2
title_fullStr S‐ketamine Alleviates Neuroinflammation and Attenuates Lipopolysaccharide‐Induced Depression Via Targeting SIRT2
title_full_unstemmed S‐ketamine Alleviates Neuroinflammation and Attenuates Lipopolysaccharide‐Induced Depression Via Targeting SIRT2
title_short S‐ketamine Alleviates Neuroinflammation and Attenuates Lipopolysaccharide‐Induced Depression Via Targeting SIRT2
title_sort s ketamine alleviates neuroinflammation and attenuates lipopolysaccharide induced depression via targeting sirt2
topic Depression
Neuroinflammation
R‐ketamine
SIRT2
S‐ketamine
url https://doi.org/10.1002/advs.202416481
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