Bridging inflammation and venous thrombosis: the NLRP3 inflammasome connection

NLRP3 (NACHT, LRR, and PYD domains-containing protein 3) inflammasome is a critical regulator of inflammatory responses in the body and is closely associated with the inflammatory processes of various diseases. In recent years, research has increasingly focused on the role of the NLRP3 inflammasome...

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Main Authors: Jian Wu, Zilong Wang, Wei Shao, Jianwen Mo
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-05-01
Series:Frontiers in Cardiovascular Medicine
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Online Access:https://www.frontiersin.org/articles/10.3389/fcvm.2025.1584745/full
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author Jian Wu
Jian Wu
Zilong Wang
Zilong Wang
Wei Shao
Wei Shao
Jianwen Mo
author_facet Jian Wu
Jian Wu
Zilong Wang
Zilong Wang
Wei Shao
Wei Shao
Jianwen Mo
author_sort Jian Wu
collection DOAJ
description NLRP3 (NACHT, LRR, and PYD domains-containing protein 3) inflammasome is a critical regulator of inflammatory responses in the body and is closely associated with the inflammatory processes of various diseases. In recent years, research has increasingly focused on the role of the NLRP3 inflammasome in venous thromboembolism (VTE). Venous thromboembolism is a common and potentially fatal vascular disease with a complex pathophysiology involving multiple cellular and molecular pathways. The NLRP3 inflammasome activates caspase-1 downstream, facilitating the maturation and secretion of pro-inflammatory cytokines such as IL-1β and IL-18, triggering local and systemic inflammatory responses. These inflammatory reactions can promote the recruitment and activation of immune cells (such as monocytes and neutrophils), platelet activation, endothelial cell damage, and aggregation, ultimately leading to thrombus formation. Additionally, the interaction of the NLRP3 inflammasome with the coagulation system further exacerbates the risk of thrombosis. In summary, the NLRP3 inflammasome plays a critical role in the development of venous thrombosis, and interventions targeting it may offer new insights and strategies for the prevention and treatment of venous thrombosis. This review provides an overview of the current understanding of how the NLRP3 inflammasome promotes venous thrombosis, highlighting recent preclinical research advancements and potential therapeutic agents in this field.
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spelling doaj-art-85cfddd070874bc4a7c54418feb98ef52025-08-20T03:05:53ZengFrontiers Media S.A.Frontiers in Cardiovascular Medicine2297-055X2025-05-011210.3389/fcvm.2025.15847451584745Bridging inflammation and venous thrombosis: the NLRP3 inflammasome connectionJian Wu0Jian Wu1Zilong Wang2Zilong Wang3Wei Shao4Wei Shao5Jianwen Mo6The First Clinical College, Gannan Medical University, Ganzhou, Jiangxi, ChinaGanzhou Key Laboratory of Osteoporosis Research, The First Affiliated Hospital of Gannan Medical University, Ganzhou, Jiangxi, ChinaThe First Clinical College, Gannan Medical University, Ganzhou, Jiangxi, ChinaGanzhou Key Laboratory of Osteoporosis Research, The First Affiliated Hospital of Gannan Medical University, Ganzhou, Jiangxi, ChinaThe First Clinical College, Gannan Medical University, Ganzhou, Jiangxi, ChinaGanzhou Key Laboratory of Osteoporosis Research, The First Affiliated Hospital of Gannan Medical University, Ganzhou, Jiangxi, ChinaDepartment of Orthopedics, The First Affiliated Hospital of Gannan Medical University, Ganzhou, Jiangxi, ChinaNLRP3 (NACHT, LRR, and PYD domains-containing protein 3) inflammasome is a critical regulator of inflammatory responses in the body and is closely associated with the inflammatory processes of various diseases. In recent years, research has increasingly focused on the role of the NLRP3 inflammasome in venous thromboembolism (VTE). Venous thromboembolism is a common and potentially fatal vascular disease with a complex pathophysiology involving multiple cellular and molecular pathways. The NLRP3 inflammasome activates caspase-1 downstream, facilitating the maturation and secretion of pro-inflammatory cytokines such as IL-1β and IL-18, triggering local and systemic inflammatory responses. These inflammatory reactions can promote the recruitment and activation of immune cells (such as monocytes and neutrophils), platelet activation, endothelial cell damage, and aggregation, ultimately leading to thrombus formation. Additionally, the interaction of the NLRP3 inflammasome with the coagulation system further exacerbates the risk of thrombosis. In summary, the NLRP3 inflammasome plays a critical role in the development of venous thrombosis, and interventions targeting it may offer new insights and strategies for the prevention and treatment of venous thrombosis. This review provides an overview of the current understanding of how the NLRP3 inflammasome promotes venous thrombosis, highlighting recent preclinical research advancements and potential therapeutic agents in this field.https://www.frontiersin.org/articles/10.3389/fcvm.2025.1584745/fullNLRP3 inflammasomethrombosisinflammationIL-1βNETs
spellingShingle Jian Wu
Jian Wu
Zilong Wang
Zilong Wang
Wei Shao
Wei Shao
Jianwen Mo
Bridging inflammation and venous thrombosis: the NLRP3 inflammasome connection
Frontiers in Cardiovascular Medicine
NLRP3 inflammasome
thrombosis
inflammation
IL-1β
NETs
title Bridging inflammation and venous thrombosis: the NLRP3 inflammasome connection
title_full Bridging inflammation and venous thrombosis: the NLRP3 inflammasome connection
title_fullStr Bridging inflammation and venous thrombosis: the NLRP3 inflammasome connection
title_full_unstemmed Bridging inflammation and venous thrombosis: the NLRP3 inflammasome connection
title_short Bridging inflammation and venous thrombosis: the NLRP3 inflammasome connection
title_sort bridging inflammation and venous thrombosis the nlrp3 inflammasome connection
topic NLRP3 inflammasome
thrombosis
inflammation
IL-1β
NETs
url https://www.frontiersin.org/articles/10.3389/fcvm.2025.1584745/full
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