The protective effect of Edaravone against acute renocardiac syndrome in a kidney ischemia-reperfusion model

Introduction: Acute kidney injury (AKI) is a common clinical occurrence causing high mortality and morbidity. In acute renocardiac syndrome, AKI leads to acute cardiac injury or/and dysfunction. This study aimed to investigate the antioxidative effects of Edaravone on cardiac tissues following the i...

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Main Authors: Yasin Bagheri, Mahshid Dehghan, Seyyedeh Mina Hejazian, Mohammadreza Ardalan, Sepideh Zununi Vahed, Bahram Niknafs
Format: Article
Language:English
Published: Tabriz University of Medical Sciences 2024-12-01
Series:Journal of Cardiovascular and Thoracic Research
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Online Access:https://jcvtr.tbzmed.ac.ir/PDF/jcvtr-16-243.pdf
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author Yasin Bagheri
Mahshid Dehghan
Seyyedeh Mina Hejazian
Mohammadreza Ardalan
Sepideh Zununi Vahed
Bahram Niknafs
author_facet Yasin Bagheri
Mahshid Dehghan
Seyyedeh Mina Hejazian
Mohammadreza Ardalan
Sepideh Zununi Vahed
Bahram Niknafs
author_sort Yasin Bagheri
collection DOAJ
description Introduction: Acute kidney injury (AKI) is a common clinical occurrence causing high mortality and morbidity. In acute renocardiac syndrome, AKI leads to acute cardiac injury or/and dysfunction. This study aimed to investigate the antioxidative effects of Edaravone on cardiac tissues following the induction of renal ischemia-reperfusion injury (IRI) in rats. Methods: Twenty-four male Wistar rats were randomly divided into four groups: IR+Edaravone, Edaravone, IR, and Sham groups (six rats per group). Non-traumatic clamps were used to stop the artery and vein blood flow of the left kidney in rats of the IR groups for 45 minutes. Thirty minutes before ischemia induction, Edaravone (3 mg/kg) was injected intraperitoneally in the IR+Edaravone group. Cardiac samples were subjected to biochemical analyses. Results: The Results showed a significant increase in the enzymatic activity of glutathione peroxidase (P=0.01), catalase (P=0.03), and superoxide dismutase (P=0.02), and the levels of glutathione (P=0.012), and total antioxidant capacity (P<0.001) in the IR+Edaravone group in comparison to the IR group. Moreover, the total antioxidant capacity of the heart was increased in the Edaravone group compared to the control and IR groups (P<0.001), indicating the safety of the drug. Conclusion: The results can reveal important insights into the protective effects of Edaravone against acute renocardiac syndrome.
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spelling doaj-art-85029c8a184742f796562f5a30e4a7a02025-08-20T01:57:52ZengTabriz University of Medical SciencesJournal of Cardiovascular and Thoracic Research2008-51172008-68302024-12-0116424324810.34172/jcvtr.33077jcvtr-33077The protective effect of Edaravone against acute renocardiac syndrome in a kidney ischemia-reperfusion modelYasin Bagheri0Mahshid Dehghan1Seyyedeh Mina Hejazian2Mohammadreza Ardalan3Sepideh Zununi Vahed4Bahram Niknafs5Kidney Research Center, Tabriz University of Medical Sciences, Tabriz, IranFaculty of Medicine, Tabriz University of Medical Sciences, Tabriz, IranKidney Research Center, Tabriz University of Medical Sciences, Tabriz, IranKidney Research Center, Tabriz University of Medical Sciences, Tabriz, IranKidney Research Center, Tabriz University of Medical Sciences, Tabriz, IranKidney Research Center, Tabriz University of Medical Sciences, Tabriz, IranIntroduction: Acute kidney injury (AKI) is a common clinical occurrence causing high mortality and morbidity. In acute renocardiac syndrome, AKI leads to acute cardiac injury or/and dysfunction. This study aimed to investigate the antioxidative effects of Edaravone on cardiac tissues following the induction of renal ischemia-reperfusion injury (IRI) in rats. Methods: Twenty-four male Wistar rats were randomly divided into four groups: IR+Edaravone, Edaravone, IR, and Sham groups (six rats per group). Non-traumatic clamps were used to stop the artery and vein blood flow of the left kidney in rats of the IR groups for 45 minutes. Thirty minutes before ischemia induction, Edaravone (3 mg/kg) was injected intraperitoneally in the IR+Edaravone group. Cardiac samples were subjected to biochemical analyses. Results: The Results showed a significant increase in the enzymatic activity of glutathione peroxidase (P=0.01), catalase (P=0.03), and superoxide dismutase (P=0.02), and the levels of glutathione (P=0.012), and total antioxidant capacity (P<0.001) in the IR+Edaravone group in comparison to the IR group. Moreover, the total antioxidant capacity of the heart was increased in the Edaravone group compared to the control and IR groups (P<0.001), indicating the safety of the drug. Conclusion: The results can reveal important insights into the protective effects of Edaravone against acute renocardiac syndrome.https://jcvtr.tbzmed.ac.ir/PDF/jcvtr-16-243.pdfedaravoneischemia-reperfusionantioxidantscardiorenal syndrome
spellingShingle Yasin Bagheri
Mahshid Dehghan
Seyyedeh Mina Hejazian
Mohammadreza Ardalan
Sepideh Zununi Vahed
Bahram Niknafs
The protective effect of Edaravone against acute renocardiac syndrome in a kidney ischemia-reperfusion model
Journal of Cardiovascular and Thoracic Research
edaravone
ischemia-reperfusion
antioxidants
cardiorenal syndrome
title The protective effect of Edaravone against acute renocardiac syndrome in a kidney ischemia-reperfusion model
title_full The protective effect of Edaravone against acute renocardiac syndrome in a kidney ischemia-reperfusion model
title_fullStr The protective effect of Edaravone against acute renocardiac syndrome in a kidney ischemia-reperfusion model
title_full_unstemmed The protective effect of Edaravone against acute renocardiac syndrome in a kidney ischemia-reperfusion model
title_short The protective effect of Edaravone against acute renocardiac syndrome in a kidney ischemia-reperfusion model
title_sort protective effect of edaravone against acute renocardiac syndrome in a kidney ischemia reperfusion model
topic edaravone
ischemia-reperfusion
antioxidants
cardiorenal syndrome
url https://jcvtr.tbzmed.ac.ir/PDF/jcvtr-16-243.pdf
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