A neonatal rat model of progressive left ventricular pressure overload induced by abdominal aortic banding microsurgeryCentral MessagePerspective

Objective: Left ventricular pressure overload models using adult mice or rats were developed 60 years ago; however, a neonatal mouse model of left ventricular pressure overload was reported only 5 years ago. Moreover, how left ventricular pressure overload reshapes the neonatal left ventricle and ho...

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Main Authors: Zheng Wang, MD, Sixie Zheng, MD, Lincai Ye, MD, PhD, Debao Li, MD, He Zhang, MD, Yingying Xiao, MD, PhD, Chenxi Liu, MD, Yuqing Hu, MD, Sijuan Sun, MD, Peisen Ruan, MD, Hao Chen, MD, PhD, Qi Sun, MD, PhD
Format: Article
Language:English
Published: Elsevier 2025-08-01
Series:JTCVS Techniques
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Online Access:http://www.sciencedirect.com/science/article/pii/S2666250725001610
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author Zheng Wang, MD
Sixie Zheng, MD
Lincai Ye, MD, PhD
Debao Li, MD
He Zhang, MD
Yingying Xiao, MD, PhD
Chenxi Liu, MD
Yuqing Hu, MD
Sijuan Sun, MD
Peisen Ruan, MD
Hao Chen, MD, PhD
Qi Sun, MD, PhD
author_facet Zheng Wang, MD
Sixie Zheng, MD
Lincai Ye, MD, PhD
Debao Li, MD
He Zhang, MD
Yingying Xiao, MD, PhD
Chenxi Liu, MD
Yuqing Hu, MD
Sijuan Sun, MD
Peisen Ruan, MD
Hao Chen, MD, PhD
Qi Sun, MD, PhD
author_sort Zheng Wang, MD
collection DOAJ
description Objective: Left ventricular pressure overload models using adult mice or rats were developed 60 years ago; however, a neonatal mouse model of left ventricular pressure overload was reported only 5 years ago. Moreover, how left ventricular pressure overload reshapes the neonatal left ventricle and how it affects cardiomyocyte proliferation remain largely unexplored. The aim of this study is to develop a simple neonatal rat model with clinical features matched to those of left ventricular pressure overload. Methods: A neonatal rat model of progressive left ventricular pressure overload was created via abdominal aortic banding microsurgery at postnatal day 1 and verified by gross examination at postnatal day 7, abdominal ultrasound at postnatal day 21, and left upper limb blood pressure measurement from postoperative day 21 to day 35. A surgical video and detailed surgical procedures were documented for learning purposes. Results: RNA sequencing demonstrated that there were only 171 differentially expressed genes between the abdominal aortic banding surgery and sham left ventricles at postnatal day 3, with 406 differentially expressed genes at postnatal day 7. At postnatal day 3, there was little enrichment of proliferation-associated genes and only a small percentage of proliferating cardiomyocytes; at postnatal day 7, there was an abundant enrichment of proliferation-associated genes and a large percentage of proliferating cardiomyocytes, exactly opposite to the neonatal transverse aortic constriction surgery model, which exhibited decreased cardiomyocyte proliferation over time and even inhibited cardiomyocyte proliferation when severe left ventricular pressure overload was induced by transverse aortic constriction surgery. Moreover, abdominal aortic banding surgery does not require a thoracotomy, resulting in a success rate as high as 100%. Conclusions: A neonatal rat model of progressive left ventricular pressure overload was successfully established and fully documented to provide a platform for pediatric left ventricular pressure overload–associated investigation.
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spelling doaj-art-8467e976a8e943ada804f0c789d7cedf2025-08-20T04:01:00ZengElsevierJTCVS Techniques2666-25072025-08-013211913510.1016/j.xjtc.2025.04.014A neonatal rat model of progressive left ventricular pressure overload induced by abdominal aortic banding microsurgeryCentral MessagePerspectiveZheng Wang, MD0Sixie Zheng, MD1Lincai Ye, MD, PhD2Debao Li, MD3He Zhang, MD4Yingying Xiao, MD, PhD5Chenxi Liu, MD6Yuqing Hu, MD7Sijuan Sun, MD8Peisen Ruan, MD9Hao Chen, MD, PhD10Qi Sun, MD, PhD11Department of Thoracic and Cardiovascular Surgery, Shanghai Children's Medical Center, Shanghai Jiao Tong University School of Medicine, Shanghai, ChinaDepartment of Thoracic and Cardiovascular Surgery, Shanghai Children's Medical Center, Shanghai Jiao Tong University School of Medicine, Shanghai, ChinaDepartment of Thoracic and Cardiovascular Surgery, Shanghai Children's Medical Center, Shanghai Jiao Tong University School of Medicine, Shanghai, China; Shanghai Institute For Pediatric Congenital Heart Disease, Shanghai Children's Medical Center, Shanghai Jiao Tong University School of Medicine, Shanghai, ChinaDepartment of Pediatric Surgery, Children's Hospital of Fudan University, National Children's Medical Center, Shanghai, ChinaDepartment of Critical Care Medicine, Women and Children's Hospital of Ningbo University, Ningbo, Zhejiang, ChinaDepartment of Thoracic and Cardiovascular Surgery, Shanghai Children's Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, ChinaDepartment of Rheumatology and Immunology, Shanghai Children's Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, ChinaDepartment of Cardiology, Shanghai Children's Hospital, School of Medicine, Shanghai Jiao Tong University, Shanghai, ChinaDepartment of Pediatric Intensive Care Unit, Shanghai Children's Medical Center, Shanghai Jiao Tong University School of Medicine, Shanghai, ChinaDepartment of Critical Care Medicine, Women and Children's Hospital of Ningbo University, Ningbo, Zhejiang, China; Address for reprints: Peisen Ruan, MD, Department of Critical Care Medicine, The Affiliated Women and Children's Hospital of Ningbo University, No. 339 Liuting St, Haishu District, Ningbo, Zhejiang Province, China.Department of Thoracic and Cardiovascular Surgery, Shanghai Children's Medical Center, Shanghai Jiao Tong University School of Medicine, Shanghai, China; Hao Chen, MD, and Qi Sun, MD, PhD, Department of Thoracic and Cardiovascular Surgery, Shanghai Children’s Medical Center, Shanghai Jiao Tong University School of Medicine, 1678 Dongfang Rd, Shanghai 200127, China.Department of Thoracic and Cardiovascular Surgery, Shanghai Children's Medical Center, Shanghai Jiao Tong University School of Medicine, Shanghai, China; Shanghai Institute For Pediatric Congenital Heart Disease, Shanghai Children's Medical Center, Shanghai Jiao Tong University School of Medicine, Shanghai, China; Hao Chen, MD, and Qi Sun, MD, PhD, Department of Thoracic and Cardiovascular Surgery, Shanghai Children’s Medical Center, Shanghai Jiao Tong University School of Medicine, 1678 Dongfang Rd, Shanghai 200127, China.Objective: Left ventricular pressure overload models using adult mice or rats were developed 60 years ago; however, a neonatal mouse model of left ventricular pressure overload was reported only 5 years ago. Moreover, how left ventricular pressure overload reshapes the neonatal left ventricle and how it affects cardiomyocyte proliferation remain largely unexplored. The aim of this study is to develop a simple neonatal rat model with clinical features matched to those of left ventricular pressure overload. Methods: A neonatal rat model of progressive left ventricular pressure overload was created via abdominal aortic banding microsurgery at postnatal day 1 and verified by gross examination at postnatal day 7, abdominal ultrasound at postnatal day 21, and left upper limb blood pressure measurement from postoperative day 21 to day 35. A surgical video and detailed surgical procedures were documented for learning purposes. Results: RNA sequencing demonstrated that there were only 171 differentially expressed genes between the abdominal aortic banding surgery and sham left ventricles at postnatal day 3, with 406 differentially expressed genes at postnatal day 7. At postnatal day 3, there was little enrichment of proliferation-associated genes and only a small percentage of proliferating cardiomyocytes; at postnatal day 7, there was an abundant enrichment of proliferation-associated genes and a large percentage of proliferating cardiomyocytes, exactly opposite to the neonatal transverse aortic constriction surgery model, which exhibited decreased cardiomyocyte proliferation over time and even inhibited cardiomyocyte proliferation when severe left ventricular pressure overload was induced by transverse aortic constriction surgery. Moreover, abdominal aortic banding surgery does not require a thoracotomy, resulting in a success rate as high as 100%. Conclusions: A neonatal rat model of progressive left ventricular pressure overload was successfully established and fully documented to provide a platform for pediatric left ventricular pressure overload–associated investigation.http://www.sciencedirect.com/science/article/pii/S2666250725001610cardiomyocyteleft ventricular pressure overloadpediatricsproliferation
spellingShingle Zheng Wang, MD
Sixie Zheng, MD
Lincai Ye, MD, PhD
Debao Li, MD
He Zhang, MD
Yingying Xiao, MD, PhD
Chenxi Liu, MD
Yuqing Hu, MD
Sijuan Sun, MD
Peisen Ruan, MD
Hao Chen, MD, PhD
Qi Sun, MD, PhD
A neonatal rat model of progressive left ventricular pressure overload induced by abdominal aortic banding microsurgeryCentral MessagePerspective
JTCVS Techniques
cardiomyocyte
left ventricular pressure overload
pediatrics
proliferation
title A neonatal rat model of progressive left ventricular pressure overload induced by abdominal aortic banding microsurgeryCentral MessagePerspective
title_full A neonatal rat model of progressive left ventricular pressure overload induced by abdominal aortic banding microsurgeryCentral MessagePerspective
title_fullStr A neonatal rat model of progressive left ventricular pressure overload induced by abdominal aortic banding microsurgeryCentral MessagePerspective
title_full_unstemmed A neonatal rat model of progressive left ventricular pressure overload induced by abdominal aortic banding microsurgeryCentral MessagePerspective
title_short A neonatal rat model of progressive left ventricular pressure overload induced by abdominal aortic banding microsurgeryCentral MessagePerspective
title_sort neonatal rat model of progressive left ventricular pressure overload induced by abdominal aortic banding microsurgerycentral messageperspective
topic cardiomyocyte
left ventricular pressure overload
pediatrics
proliferation
url http://www.sciencedirect.com/science/article/pii/S2666250725001610
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