Persistently Elevated Level of IL-8 in Chlamydia trachomatis Infected HeLa 229 Cells is Dependent on Intracellular Available Iron

Chlamydia trachomatis is a leading cause of sexually transmitted infection worldwide and responsible for myriad of immunopathological changes associated with reproductive health. Delayed secretion of proinflammatory chemokine interleukin (IL)-8 is a hallmark of chlamydial infection and is dependent...

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Main Authors: Harsh Vardhan, Raini Dutta, Vikas Vats, Rishein Gupta, Rajneesh Jha, Hem Chandra Jha, Pragya Srivastava, Apurb Rashmi Bhengraj, Aruna Singh Mittal
Format: Article
Language:English
Published: Wiley 2009-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/2009/417658
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author Harsh Vardhan
Raini Dutta
Vikas Vats
Rishein Gupta
Rajneesh Jha
Hem Chandra Jha
Pragya Srivastava
Apurb Rashmi Bhengraj
Aruna Singh Mittal
author_facet Harsh Vardhan
Raini Dutta
Vikas Vats
Rishein Gupta
Rajneesh Jha
Hem Chandra Jha
Pragya Srivastava
Apurb Rashmi Bhengraj
Aruna Singh Mittal
author_sort Harsh Vardhan
collection DOAJ
description Chlamydia trachomatis is a leading cause of sexually transmitted infection worldwide and responsible for myriad of immunopathological changes associated with reproductive health. Delayed secretion of proinflammatory chemokine interleukin (IL)-8 is a hallmark of chlamydial infection and is dependent on chlamydial growth. We examined the effect of iron chelators on IL-8 production in HeLa 229 (cervix epitheloid cell, CCL2) cells infected with C. trachomatis. IL-8 production was induced by Iron chelator DFO and Mimosine, however, synergy with chlamydial infection was obtained with DFO only. Temporal expression of proinflammatory secreted cytokines IL-1beta, TNF-alpha, and IL-8 did not show synchrony in Chlamydia trachomatis infected cells. Secretion of IL-8 from Hela cells infected with C. trachomatis was not dependent on IL-1 beta and TNF- alpha induction. These results indicate towards involvement of iron in chlamydia induced IL-8 production.
format Article
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institution Kabale University
issn 0962-9351
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language English
publishDate 2009-01-01
publisher Wiley
record_format Article
series Mediators of Inflammation
spelling doaj-art-827ec608abba404ea33403a74c66eb4e2025-08-20T03:37:31ZengWileyMediators of Inflammation0962-93511466-18612009-01-01200910.1155/2009/417658417658Persistently Elevated Level of IL-8 in Chlamydia trachomatis Infected HeLa 229 Cells is Dependent on Intracellular Available IronHarsh Vardhan0Raini Dutta1Vikas Vats2Rishein Gupta3Rajneesh Jha4Hem Chandra Jha5Pragya Srivastava6Apurb Rashmi Bhengraj7Aruna Singh Mittal8Institute of Pathology (ICMR), Safdarjung Hospital Campus, New Delhi 110029, IndiaInstitute of Pathology (ICMR), Safdarjung Hospital Campus, New Delhi 110029, IndiaInstitute of Pathology (ICMR), Safdarjung Hospital Campus, New Delhi 110029, IndiaInstitute of Pathology (ICMR), Safdarjung Hospital Campus, New Delhi 110029, IndiaInstitute of Pathology (ICMR), Safdarjung Hospital Campus, New Delhi 110029, IndiaInstitute of Pathology (ICMR), Safdarjung Hospital Campus, New Delhi 110029, IndiaInstitute of Pathology (ICMR), Safdarjung Hospital Campus, New Delhi 110029, IndiaInstitute of Pathology (ICMR), Safdarjung Hospital Campus, New Delhi 110029, IndiaInstitute of Pathology (ICMR), Safdarjung Hospital Campus, New Delhi 110029, IndiaChlamydia trachomatis is a leading cause of sexually transmitted infection worldwide and responsible for myriad of immunopathological changes associated with reproductive health. Delayed secretion of proinflammatory chemokine interleukin (IL)-8 is a hallmark of chlamydial infection and is dependent on chlamydial growth. We examined the effect of iron chelators on IL-8 production in HeLa 229 (cervix epitheloid cell, CCL2) cells infected with C. trachomatis. IL-8 production was induced by Iron chelator DFO and Mimosine, however, synergy with chlamydial infection was obtained with DFO only. Temporal expression of proinflammatory secreted cytokines IL-1beta, TNF-alpha, and IL-8 did not show synchrony in Chlamydia trachomatis infected cells. Secretion of IL-8 from Hela cells infected with C. trachomatis was not dependent on IL-1 beta and TNF- alpha induction. These results indicate towards involvement of iron in chlamydia induced IL-8 production.http://dx.doi.org/10.1155/2009/417658
spellingShingle Harsh Vardhan
Raini Dutta
Vikas Vats
Rishein Gupta
Rajneesh Jha
Hem Chandra Jha
Pragya Srivastava
Apurb Rashmi Bhengraj
Aruna Singh Mittal
Persistently Elevated Level of IL-8 in Chlamydia trachomatis Infected HeLa 229 Cells is Dependent on Intracellular Available Iron
Mediators of Inflammation
title Persistently Elevated Level of IL-8 in Chlamydia trachomatis Infected HeLa 229 Cells is Dependent on Intracellular Available Iron
title_full Persistently Elevated Level of IL-8 in Chlamydia trachomatis Infected HeLa 229 Cells is Dependent on Intracellular Available Iron
title_fullStr Persistently Elevated Level of IL-8 in Chlamydia trachomatis Infected HeLa 229 Cells is Dependent on Intracellular Available Iron
title_full_unstemmed Persistently Elevated Level of IL-8 in Chlamydia trachomatis Infected HeLa 229 Cells is Dependent on Intracellular Available Iron
title_short Persistently Elevated Level of IL-8 in Chlamydia trachomatis Infected HeLa 229 Cells is Dependent on Intracellular Available Iron
title_sort persistently elevated level of il 8 in chlamydia trachomatis infected hela 229 cells is dependent on intracellular available iron
url http://dx.doi.org/10.1155/2009/417658
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