Cytomegalovirus-Specific IL-10-Producing CD4+ T Cells Are Governed by Type-I IFN-Induced IL-27 and Promote Virus Persistence.

CD4+ T cells support host defence against herpesviruses and other viral pathogens. We identified that CD4+ T cells from systemic and mucosal tissues of hosts infected with the β-herpesviridae human cytomegalovirus (HCMV) or murine cytomegalovirus (MCMV) express the regulatory cytokine interleukin (I...

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Main Authors: Mathew Clement, Morgan Marsden, Maria A Stacey, Juneid Abdul-Karim, Silvia Gimeno Brias, Diana Costa Bento, Martin J Scurr, Peter Ghazal, Casey T Weaver, Gianluca Carlesso, Simon Clare, Simon A Jones, Andrew Godkin, Gareth W Jones, Ian R Humphreys
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2016-12-01
Series:PLoS Pathogens
Online Access:https://journals.plos.org/plospathogens/article/file?id=10.1371/journal.ppat.1006050&type=printable
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author Mathew Clement
Morgan Marsden
Maria A Stacey
Juneid Abdul-Karim
Silvia Gimeno Brias
Diana Costa Bento
Martin J Scurr
Peter Ghazal
Casey T Weaver
Gianluca Carlesso
Simon Clare
Simon A Jones
Andrew Godkin
Gareth W Jones
Ian R Humphreys
author_facet Mathew Clement
Morgan Marsden
Maria A Stacey
Juneid Abdul-Karim
Silvia Gimeno Brias
Diana Costa Bento
Martin J Scurr
Peter Ghazal
Casey T Weaver
Gianluca Carlesso
Simon Clare
Simon A Jones
Andrew Godkin
Gareth W Jones
Ian R Humphreys
author_sort Mathew Clement
collection DOAJ
description CD4+ T cells support host defence against herpesviruses and other viral pathogens. We identified that CD4+ T cells from systemic and mucosal tissues of hosts infected with the β-herpesviridae human cytomegalovirus (HCMV) or murine cytomegalovirus (MCMV) express the regulatory cytokine interleukin (IL)-10. IL-10+CD4+ T cells co-expressed TH1-associated transcription factors and chemokine receptors. Mice lacking T cell-derived IL-10 elicited enhanced antiviral T cell responses and restricted MCMV persistence in salivary glands and secretion in saliva. Thus, IL-10+CD4+ T cells suppress antiviral immune responses against CMV. Expansion of this T-cell population in the periphery was promoted by IL-27 whereas mucosal IL-10+ T cell responses were ICOS-dependent. Infected Il27rα-deficient mice with reduced peripheral IL-10+CD4+ T cell accumulation displayed robust T cell responses and restricted MCMV persistence and shedding. Temporal inhibition experiments revealed that IL-27R signaling during initial infection was required for the suppression of T cell immunity and control of virus shedding during MCMV persistence. IL-27 production was promoted by type-I IFN, suggesting that β-herpesviridae exploit the immune-regulatory properties of this antiviral pathway to establish chronicity. Further, our data reveal that cytokine signaling events during initial infection profoundly influence virus chronicity.
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institution Kabale University
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publishDate 2016-12-01
publisher Public Library of Science (PLoS)
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spelling doaj-art-7fe395ac2dd442b6bb7558097586ab3e2025-08-20T03:24:27ZengPublic Library of Science (PLoS)PLoS Pathogens1553-73661553-73742016-12-011212e100605010.1371/journal.ppat.1006050Cytomegalovirus-Specific IL-10-Producing CD4+ T Cells Are Governed by Type-I IFN-Induced IL-27 and Promote Virus Persistence.Mathew ClementMorgan MarsdenMaria A StaceyJuneid Abdul-KarimSilvia Gimeno BriasDiana Costa BentoMartin J ScurrPeter GhazalCasey T WeaverGianluca CarlessoSimon ClareSimon A JonesAndrew GodkinGareth W JonesIan R HumphreysCD4+ T cells support host defence against herpesviruses and other viral pathogens. We identified that CD4+ T cells from systemic and mucosal tissues of hosts infected with the β-herpesviridae human cytomegalovirus (HCMV) or murine cytomegalovirus (MCMV) express the regulatory cytokine interleukin (IL)-10. IL-10+CD4+ T cells co-expressed TH1-associated transcription factors and chemokine receptors. Mice lacking T cell-derived IL-10 elicited enhanced antiviral T cell responses and restricted MCMV persistence in salivary glands and secretion in saliva. Thus, IL-10+CD4+ T cells suppress antiviral immune responses against CMV. Expansion of this T-cell population in the periphery was promoted by IL-27 whereas mucosal IL-10+ T cell responses were ICOS-dependent. Infected Il27rα-deficient mice with reduced peripheral IL-10+CD4+ T cell accumulation displayed robust T cell responses and restricted MCMV persistence and shedding. Temporal inhibition experiments revealed that IL-27R signaling during initial infection was required for the suppression of T cell immunity and control of virus shedding during MCMV persistence. IL-27 production was promoted by type-I IFN, suggesting that β-herpesviridae exploit the immune-regulatory properties of this antiviral pathway to establish chronicity. Further, our data reveal that cytokine signaling events during initial infection profoundly influence virus chronicity.https://journals.plos.org/plospathogens/article/file?id=10.1371/journal.ppat.1006050&type=printable
spellingShingle Mathew Clement
Morgan Marsden
Maria A Stacey
Juneid Abdul-Karim
Silvia Gimeno Brias
Diana Costa Bento
Martin J Scurr
Peter Ghazal
Casey T Weaver
Gianluca Carlesso
Simon Clare
Simon A Jones
Andrew Godkin
Gareth W Jones
Ian R Humphreys
Cytomegalovirus-Specific IL-10-Producing CD4+ T Cells Are Governed by Type-I IFN-Induced IL-27 and Promote Virus Persistence.
PLoS Pathogens
title Cytomegalovirus-Specific IL-10-Producing CD4+ T Cells Are Governed by Type-I IFN-Induced IL-27 and Promote Virus Persistence.
title_full Cytomegalovirus-Specific IL-10-Producing CD4+ T Cells Are Governed by Type-I IFN-Induced IL-27 and Promote Virus Persistence.
title_fullStr Cytomegalovirus-Specific IL-10-Producing CD4+ T Cells Are Governed by Type-I IFN-Induced IL-27 and Promote Virus Persistence.
title_full_unstemmed Cytomegalovirus-Specific IL-10-Producing CD4+ T Cells Are Governed by Type-I IFN-Induced IL-27 and Promote Virus Persistence.
title_short Cytomegalovirus-Specific IL-10-Producing CD4+ T Cells Are Governed by Type-I IFN-Induced IL-27 and Promote Virus Persistence.
title_sort cytomegalovirus specific il 10 producing cd4 t cells are governed by type i ifn induced il 27 and promote virus persistence
url https://journals.plos.org/plospathogens/article/file?id=10.1371/journal.ppat.1006050&type=printable
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