Aβ status assessment in a hypothetical scenario prior to treatment with disease‐modifying therapies: Evidence from 10‐year real‐world experience at university memory clinics

Abstract INTRODUCTION With the advent of disease‐modifying therapies, accurate assessment of biomarkers indicating the presence of disease‐associated amyloid beta (Aβ) pathology becomes crucial in patients with clinically suspected Alzheimer's disease (AD). We evaluated Aβ levels in cerebrospin...

Full description

Saved in:
Bibliographic Details
Main Authors: Matthias Brendel, Tandis Parvizi, Johannes Gnörich, Christof Elias Topfstedt, Katharina Buerger, Daniel Janowitz, Boris‐Stephan Rauchmann, Robert Perneczky, Carolin Kurz, Dirk Mehrens, Wolfgang G. Kunz, Julia Kusche‐Palenga, Agnes Bernadette Kling, Antonia Buchal, Elizabet Nestorova, Sara Silvaieh, Raphael Wurm, Tatjana Traub‐Weidinger, Sigrid Klotz, Günther Regelsberger, Axel Rominger, Alexander Drzezga, Johannes Levin, Elisabeth Stögmann, Nicolai Franzmeier, Günter U. Höglinger
Format: Article
Language:English
Published: Wiley 2024-10-01
Series:Alzheimer’s & Dementia: Diagnosis, Assessment & Disease Monitoring
Subjects:
Online Access:https://doi.org/10.1002/dad2.70031
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850041050096205824
author Matthias Brendel
Tandis Parvizi
Johannes Gnörich
Christof Elias Topfstedt
Katharina Buerger
Daniel Janowitz
Boris‐Stephan Rauchmann
Robert Perneczky
Carolin Kurz
Dirk Mehrens
Wolfgang G. Kunz
Julia Kusche‐Palenga
Agnes Bernadette Kling
Antonia Buchal
Elizabet Nestorova
Sara Silvaieh
Raphael Wurm
Tatjana Traub‐Weidinger
Sigrid Klotz
Günther Regelsberger
Axel Rominger
Alexander Drzezga
Johannes Levin
Elisabeth Stögmann
Nicolai Franzmeier
Günter U. Höglinger
author_facet Matthias Brendel
Tandis Parvizi
Johannes Gnörich
Christof Elias Topfstedt
Katharina Buerger
Daniel Janowitz
Boris‐Stephan Rauchmann
Robert Perneczky
Carolin Kurz
Dirk Mehrens
Wolfgang G. Kunz
Julia Kusche‐Palenga
Agnes Bernadette Kling
Antonia Buchal
Elizabet Nestorova
Sara Silvaieh
Raphael Wurm
Tatjana Traub‐Weidinger
Sigrid Klotz
Günther Regelsberger
Axel Rominger
Alexander Drzezga
Johannes Levin
Elisabeth Stögmann
Nicolai Franzmeier
Günter U. Höglinger
author_sort Matthias Brendel
collection DOAJ
description Abstract INTRODUCTION With the advent of disease‐modifying therapies, accurate assessment of biomarkers indicating the presence of disease‐associated amyloid beta (Aβ) pathology becomes crucial in patients with clinically suspected Alzheimer's disease (AD). We evaluated Aβ levels in cerebrospinal fluid (Aβ CSF) and Aβ levels in positron emission tomography (Aβ PET) biomarkers in a real‐world memory‐clinic setting to develop an efficient algorithm for clinical use. METHODS Patients were evaluated for AD‐related Aβ pathology from two independent cohorts (Ludwig Maximilian University [LMU], n = 402, and Medical University of Vienna [MUV], n = 144). Optimal thresholds of CSF biomarkers were deduced from receiver operating characteristic curves and validated against Aβ PET positivity. RESULTS In both cohorts, a CSF Aβ42/40 ratio ≥ 7.1% was associated with a low risk of a positive Aβ PET scan (negative predictive value: 94.3%). Implementing two cutoffs revealed 14% to 16% of patients with intermediate results (CSF Aβ42/40 ratio: 5.5%–7.1%), which had a strong benefit from Aβ PET imaging (44%–52% Aβ PET positivity). DISCUSSION A two‐cutoff approach for CSF Aβ42/40 including Aβ PET imaging at intermediate results provides an effective assessment of Aβ pathology in real‐world settings. Highlights We evaluated cerebrospinal fluid (CSF) and positron emission tomography (PET) amyloid beta (Aβ) biomarkers for Alzheimer's disease in real‐world cohorts. A CSF Aβ 42/40 ratio between 5.5% and 7.1% defines patients at borderline levels. Patients at borderline levels strongly benefit from additional Aβ PET imaging. Two‐cutoff CSF Aβ 42/40 and PET will allow effective treatment stratification.
format Article
id doaj-art-7fb30f95958a481fb8b2ea89e020010b
institution DOAJ
issn 2352-8729
language English
publishDate 2024-10-01
publisher Wiley
record_format Article
series Alzheimer’s & Dementia: Diagnosis, Assessment & Disease Monitoring
spelling doaj-art-7fb30f95958a481fb8b2ea89e020010b2025-08-20T02:55:53ZengWileyAlzheimer’s & Dementia: Diagnosis, Assessment & Disease Monitoring2352-87292024-10-01164n/an/a10.1002/dad2.70031Aβ status assessment in a hypothetical scenario prior to treatment with disease‐modifying therapies: Evidence from 10‐year real‐world experience at university memory clinicsMatthias Brendel0Tandis Parvizi1Johannes Gnörich2Christof Elias Topfstedt3Katharina Buerger4Daniel Janowitz5Boris‐Stephan Rauchmann6Robert Perneczky7Carolin Kurz8Dirk Mehrens9Wolfgang G. Kunz10Julia Kusche‐Palenga11Agnes Bernadette Kling12Antonia Buchal13Elizabet Nestorova14Sara Silvaieh15Raphael Wurm16Tatjana Traub‐Weidinger17Sigrid Klotz18Günther Regelsberger19Axel Rominger20Alexander Drzezga21Johannes Levin22Elisabeth Stögmann23Nicolai Franzmeier24Günter U. Höglinger25Department of Nuclear MedicineLMU University Hospital, LMU MunichMunich GermanyDepartment of Nuclear MedicineLMU University Hospital, LMU MunichMunich GermanyDepartment of Nuclear MedicineLMU University Hospital, LMU MunichMunich GermanyInstitute for Stroke and Dementia Research (ISD)LMU University Hospital, LMU MunichMunich GermanyGerman Center for Neurodegenerative Diseases (DZNE) MunichMunichGermanyInstitute for Stroke and Dementia Research (ISD)LMU University Hospital, LMU MunichMunich GermanyDepartment of Psychiatry and PsychotherapyLMU University Hospital, LMU MunichMunich GermanyGerman Center for Neurodegenerative Diseases (DZNE) MunichMunichGermanyDepartment of Psychiatry and PsychotherapyLMU University Hospital, LMU MunichMunich GermanyDepartment of RadiologyLMU University Hospital, LMU MunichMunich GermanyDepartment of RadiologyLMU University Hospital, LMU MunichMunich GermanyDepartment of Nuclear MedicineLMU University Hospital, LMU MunichMunich GermanyDepartment of Nuclear MedicineLMU University Hospital, LMU MunichMunich GermanyDepartment of RadiologyLMU University Hospital, LMU MunichMunich GermanyDepartment of Psychiatry and PsychotherapyLMU University Hospital, LMU MunichMunich GermanyDepartment of NeurologyMedical University of ViennaViennaAustriaDepartment of NeurologyMedical University of ViennaViennaAustriaDivision of Nuclear Medicine, Department of Biomedical Imaging and Image‐Guided TherapyMedical University of ViennaViennaAustriaComprehensive Center for Clinical Neurosciences and Mental HealthMedical University of ViennaViennaAustriaComprehensive Center for Clinical Neurosciences and Mental HealthMedical University of ViennaViennaAustriaDepartment of Nuclear Medicine, InselspitalBern University Hospital, University of BernBernSwitzerlandDepartment of Nuclear MedicineFaculty of Medicine and University Hospital CologneCologneGermanyGerman Center for Neurodegenerative Diseases (DZNE) MunichMunichGermanyDepartment of NeurologyMedical University of ViennaViennaAustriaMunich Cluster for Systems Neurology (SyNergy)MunichGermanyGerman Center for Neurodegenerative Diseases (DZNE) MunichMunichGermanyAbstract INTRODUCTION With the advent of disease‐modifying therapies, accurate assessment of biomarkers indicating the presence of disease‐associated amyloid beta (Aβ) pathology becomes crucial in patients with clinically suspected Alzheimer's disease (AD). We evaluated Aβ levels in cerebrospinal fluid (Aβ CSF) and Aβ levels in positron emission tomography (Aβ PET) biomarkers in a real‐world memory‐clinic setting to develop an efficient algorithm for clinical use. METHODS Patients were evaluated for AD‐related Aβ pathology from two independent cohorts (Ludwig Maximilian University [LMU], n = 402, and Medical University of Vienna [MUV], n = 144). Optimal thresholds of CSF biomarkers were deduced from receiver operating characteristic curves and validated against Aβ PET positivity. RESULTS In both cohorts, a CSF Aβ42/40 ratio ≥ 7.1% was associated with a low risk of a positive Aβ PET scan (negative predictive value: 94.3%). Implementing two cutoffs revealed 14% to 16% of patients with intermediate results (CSF Aβ42/40 ratio: 5.5%–7.1%), which had a strong benefit from Aβ PET imaging (44%–52% Aβ PET positivity). DISCUSSION A two‐cutoff approach for CSF Aβ42/40 including Aβ PET imaging at intermediate results provides an effective assessment of Aβ pathology in real‐world settings. Highlights We evaluated cerebrospinal fluid (CSF) and positron emission tomography (PET) amyloid beta (Aβ) biomarkers for Alzheimer's disease in real‐world cohorts. A CSF Aβ 42/40 ratio between 5.5% and 7.1% defines patients at borderline levels. Patients at borderline levels strongly benefit from additional Aβ PET imaging. Two‐cutoff CSF Aβ 42/40 and PET will allow effective treatment stratification.https://doi.org/10.1002/dad2.70031Alzheimer's diseasebiomarkerscerebrospinal fluiddementiapositron emission tomographyreal world
spellingShingle Matthias Brendel
Tandis Parvizi
Johannes Gnörich
Christof Elias Topfstedt
Katharina Buerger
Daniel Janowitz
Boris‐Stephan Rauchmann
Robert Perneczky
Carolin Kurz
Dirk Mehrens
Wolfgang G. Kunz
Julia Kusche‐Palenga
Agnes Bernadette Kling
Antonia Buchal
Elizabet Nestorova
Sara Silvaieh
Raphael Wurm
Tatjana Traub‐Weidinger
Sigrid Klotz
Günther Regelsberger
Axel Rominger
Alexander Drzezga
Johannes Levin
Elisabeth Stögmann
Nicolai Franzmeier
Günter U. Höglinger
Aβ status assessment in a hypothetical scenario prior to treatment with disease‐modifying therapies: Evidence from 10‐year real‐world experience at university memory clinics
Alzheimer’s & Dementia: Diagnosis, Assessment & Disease Monitoring
Alzheimer's disease
biomarkers
cerebrospinal fluid
dementia
positron emission tomography
real world
title Aβ status assessment in a hypothetical scenario prior to treatment with disease‐modifying therapies: Evidence from 10‐year real‐world experience at university memory clinics
title_full Aβ status assessment in a hypothetical scenario prior to treatment with disease‐modifying therapies: Evidence from 10‐year real‐world experience at university memory clinics
title_fullStr Aβ status assessment in a hypothetical scenario prior to treatment with disease‐modifying therapies: Evidence from 10‐year real‐world experience at university memory clinics
title_full_unstemmed Aβ status assessment in a hypothetical scenario prior to treatment with disease‐modifying therapies: Evidence from 10‐year real‐world experience at university memory clinics
title_short Aβ status assessment in a hypothetical scenario prior to treatment with disease‐modifying therapies: Evidence from 10‐year real‐world experience at university memory clinics
title_sort aβ status assessment in a hypothetical scenario prior to treatment with disease modifying therapies evidence from 10 year real world experience at university memory clinics
topic Alzheimer's disease
biomarkers
cerebrospinal fluid
dementia
positron emission tomography
real world
url https://doi.org/10.1002/dad2.70031
work_keys_str_mv AT matthiasbrendel abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT tandisparvizi abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT johannesgnorich abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT christofeliastopfstedt abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT katharinabuerger abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT danieljanowitz abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT borisstephanrauchmann abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT robertperneczky abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT carolinkurz abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT dirkmehrens abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT wolfganggkunz abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT juliakuschepalenga abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT agnesbernadettekling abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT antoniabuchal abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT elizabetnestorova abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT sarasilvaieh abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT raphaelwurm abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT tatjanatraubweidinger abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT sigridklotz abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT guntherregelsberger abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT axelrominger abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT alexanderdrzezga abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT johanneslevin abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT elisabethstogmann abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT nicolaifranzmeier abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics
AT gunteruhoglinger abstatusassessmentinahypotheticalscenariopriortotreatmentwithdiseasemodifyingtherapiesevidencefrom10yearrealworldexperienceatuniversitymemoryclinics