The possible involvement of interleukin-6 in congestive heart failure in a postmenopausal woman with uterine fibromatosis

Interleukin-6 (IL-6) has been shown to contribute to the progression of myocardial damage and dysfunction in congestive heart failure. We report a case of a 53-year old woman with congestive heart failure that may have been caused by IL-6 secreting uterine leiomyoma. The woman developed progressive...

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Main Authors: M. Ohta, N. Keira, S. Yoshida, R. Yasuda, Y. Tarumi, H. Tsuchiya, M. Kikai, T. Nomura, T. Tatsumi
Format: Article
Language:English
Published: IMR Press 2020-08-01
Series:Clinical and Experimental Obstetrics & Gynecology
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Online Access:https://www.imrpress.com/journal/CEOG/47/4/10.31083/j.ceog.2020.04.5062
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author M. Ohta
N. Keira
S. Yoshida
R. Yasuda
Y. Tarumi
H. Tsuchiya
M. Kikai
T. Nomura
T. Tatsumi
author_facet M. Ohta
N. Keira
S. Yoshida
R. Yasuda
Y. Tarumi
H. Tsuchiya
M. Kikai
T. Nomura
T. Tatsumi
author_sort M. Ohta
collection DOAJ
description Interleukin-6 (IL-6) has been shown to contribute to the progression of myocardial damage and dysfunction in congestive heart failure. We report a case of a 53-year old woman with congestive heart failure that may have been caused by IL-6 secreting uterine leiomyoma. The woman developed progressive dyspnea, pleural effusion, and leg edema with a decreased left ventricular ejection fraction (LVEF). Imaging studies indicated uterine leiomyoma compressing the vena cava. Due to unresponsiveness to conventional medical treatment, she underwent a total abdominal hysterectomy with bilateral salpingo-oophorectomy to alleviate compression of the vena cava. After surgery, pleural effusion and leg edema completely resolved. Concomitant with the subsequent improvement of LVEF, a pre-operative serum IL-6 measurement of 36.2 pg/mL fell to 1.6 pg/mL postoperatively. Immunologically positive staining for IL-6 was demonstrated in the cytoplasm of most of the uterine leiomyoma cells. These findings suggest that IL-6 produced by leiomyoma cells may have been involved in the pathophysiological progression of heart failure and associated hemodynamic changes.
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institution Kabale University
issn 0390-6663
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publishDate 2020-08-01
publisher IMR Press
record_format Article
series Clinical and Experimental Obstetrics & Gynecology
spelling doaj-art-7fa29f5af22d4053ad652c94cce200072025-08-20T03:48:35ZengIMR PressClinical and Experimental Obstetrics & Gynecology0390-66632020-08-0147459059510.31083/j.ceog.2020.04.5062S0390-6663(20)00320-6The possible involvement of interleukin-6 in congestive heart failure in a postmenopausal woman with uterine fibromatosisM. Ohta0N. Keira1S. Yoshida2R. Yasuda3Y. Tarumi4H. Tsuchiya5M. Kikai6T. Nomura7T. Tatsumi8Department of Obstetrics and Gynecology, Chibune General Hospital, Osaka, JapanDepartment of Cardiovascular Medicine, Kyoto Chubu Medical Center, Kyoto, JapanDepartment of Obstetrics and Gynecology, Chibune General Hospital, Osaka, JapanDepartment of Obstetrics and Gynecology, Chibune General Hospital, Osaka, JapanDepartment of Obstetrics and Gynecology, Kyoto Chubu Medical Center, Kyoto, JapanDepartment of Obstetrics and Gynecology, Kyoto Chubu Medical Center, Kyoto, JapanDepartment of Cardiovascular Medicine, Kyoto Chubu Medical Center, Kyoto, JapanDepartment of Cardiovascular Medicine, Kyoto Chubu Medical Center, Kyoto, JapanDepartment of Cardiovascular Medicine, Kyoto Chubu Medical Center, Kyoto, JapanInterleukin-6 (IL-6) has been shown to contribute to the progression of myocardial damage and dysfunction in congestive heart failure. We report a case of a 53-year old woman with congestive heart failure that may have been caused by IL-6 secreting uterine leiomyoma. The woman developed progressive dyspnea, pleural effusion, and leg edema with a decreased left ventricular ejection fraction (LVEF). Imaging studies indicated uterine leiomyoma compressing the vena cava. Due to unresponsiveness to conventional medical treatment, she underwent a total abdominal hysterectomy with bilateral salpingo-oophorectomy to alleviate compression of the vena cava. After surgery, pleural effusion and leg edema completely resolved. Concomitant with the subsequent improvement of LVEF, a pre-operative serum IL-6 measurement of 36.2 pg/mL fell to 1.6 pg/mL postoperatively. Immunologically positive staining for IL-6 was demonstrated in the cytoplasm of most of the uterine leiomyoma cells. These findings suggest that IL-6 produced by leiomyoma cells may have been involved in the pathophysiological progression of heart failure and associated hemodynamic changes.https://www.imrpress.com/journal/CEOG/47/4/10.31083/j.ceog.2020.04.5062leiomyomainterleukin-6heart failurepleural effusion
spellingShingle M. Ohta
N. Keira
S. Yoshida
R. Yasuda
Y. Tarumi
H. Tsuchiya
M. Kikai
T. Nomura
T. Tatsumi
The possible involvement of interleukin-6 in congestive heart failure in a postmenopausal woman with uterine fibromatosis
Clinical and Experimental Obstetrics & Gynecology
leiomyoma
interleukin-6
heart failure
pleural effusion
title The possible involvement of interleukin-6 in congestive heart failure in a postmenopausal woman with uterine fibromatosis
title_full The possible involvement of interleukin-6 in congestive heart failure in a postmenopausal woman with uterine fibromatosis
title_fullStr The possible involvement of interleukin-6 in congestive heart failure in a postmenopausal woman with uterine fibromatosis
title_full_unstemmed The possible involvement of interleukin-6 in congestive heart failure in a postmenopausal woman with uterine fibromatosis
title_short The possible involvement of interleukin-6 in congestive heart failure in a postmenopausal woman with uterine fibromatosis
title_sort possible involvement of interleukin 6 in congestive heart failure in a postmenopausal woman with uterine fibromatosis
topic leiomyoma
interleukin-6
heart failure
pleural effusion
url https://www.imrpress.com/journal/CEOG/47/4/10.31083/j.ceog.2020.04.5062
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