MicroRNA-520a Suppresses Pathogenesis and Progression of Non-Small-Cell Lung Cancer through Targeting the RRM2/Wnt Axis

MicroRNAs (miRNAs) regulate multiple cellular behaviors, and their aberrant expression is frequently associated with disease progression. This research focused on the effects of miR-520a on the development of non-small-cell lung cancer (NSCLC) and the molecules involved. Tumor and normal tissues fro...

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Main Authors: Yi Xie, Congyu Xue, Shuai Guo, Lei Yang
Format: Article
Language:English
Published: Wiley 2021-01-01
Series:Analytical Cellular Pathology
Online Access:http://dx.doi.org/10.1155/2021/9652420
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author Yi Xie
Congyu Xue
Shuai Guo
Lei Yang
author_facet Yi Xie
Congyu Xue
Shuai Guo
Lei Yang
author_sort Yi Xie
collection DOAJ
description MicroRNAs (miRNAs) regulate multiple cellular behaviors, and their aberrant expression is frequently associated with disease progression. This research focused on the effects of miR-520a on the development of non-small-cell lung cancer (NSCLC) and the molecules involved. Tumor and normal tissues from 24 patients with NSCLC were collected. Differentially expressed miRNAs between tumor tissues and normal tissues were screened using microarrays, and miR-520a was screened to be significantly poorly expressed in tumor samples. Artificial upregulation of miR-520a reduced proliferation, migration and invasion, and resistance to death of NSCLC A549 and H460 cells according to the MTT, EdU labeling, transwell, and flow cytometry assays, respectively. miR-520a upregulation suppressed growth and metastasis of xenograft tumors in vivo. The integrated bioinformatic analysis and dual luciferase assays suggested that miR-520a targeted ribonucleotide reductase subunit 2 (RRM2) mRNA and inactivated the Wnt/β-catenin signaling pathway in NSCLC cells. Upregulation of RRM2 enhanced the malignant behaviors of NSCLCs, but the oncogenic effects of RRM2 were blocked upon miR-520a overexpression. To conclude, this study evidenced that miR-520a inhibits NSCLC progression through suppressing RRM2 and the Wnt signaling pathway. This paper may offer novel insights into NSCLC treatment.
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spelling doaj-art-7f7382f56d3749109294fc971f3b40db2025-08-20T03:35:19ZengWileyAnalytical Cellular Pathology2210-71772210-71852021-01-01202110.1155/2021/96524209652420MicroRNA-520a Suppresses Pathogenesis and Progression of Non-Small-Cell Lung Cancer through Targeting the RRM2/Wnt AxisYi Xie0Congyu Xue1Shuai Guo2Lei Yang3Department of Respiratory Oncology, Shandong Provincial Chest Hospital, Jinan, 250013 Shandong, ChinaDepartment of Tuberculosis, Shandong Provincial Chest Hospital, Jinan, 250013 Shandong, ChinaDepartment of Respiratory Oncology, Shandong Provincial Chest Hospital, Jinan, 250013 Shandong, ChinaDepartment of Tuberculosis, Shandong Provincial Chest Hospital, Jinan, 250013 Shandong, ChinaMicroRNAs (miRNAs) regulate multiple cellular behaviors, and their aberrant expression is frequently associated with disease progression. This research focused on the effects of miR-520a on the development of non-small-cell lung cancer (NSCLC) and the molecules involved. Tumor and normal tissues from 24 patients with NSCLC were collected. Differentially expressed miRNAs between tumor tissues and normal tissues were screened using microarrays, and miR-520a was screened to be significantly poorly expressed in tumor samples. Artificial upregulation of miR-520a reduced proliferation, migration and invasion, and resistance to death of NSCLC A549 and H460 cells according to the MTT, EdU labeling, transwell, and flow cytometry assays, respectively. miR-520a upregulation suppressed growth and metastasis of xenograft tumors in vivo. The integrated bioinformatic analysis and dual luciferase assays suggested that miR-520a targeted ribonucleotide reductase subunit 2 (RRM2) mRNA and inactivated the Wnt/β-catenin signaling pathway in NSCLC cells. Upregulation of RRM2 enhanced the malignant behaviors of NSCLCs, but the oncogenic effects of RRM2 were blocked upon miR-520a overexpression. To conclude, this study evidenced that miR-520a inhibits NSCLC progression through suppressing RRM2 and the Wnt signaling pathway. This paper may offer novel insights into NSCLC treatment.http://dx.doi.org/10.1155/2021/9652420
spellingShingle Yi Xie
Congyu Xue
Shuai Guo
Lei Yang
MicroRNA-520a Suppresses Pathogenesis and Progression of Non-Small-Cell Lung Cancer through Targeting the RRM2/Wnt Axis
Analytical Cellular Pathology
title MicroRNA-520a Suppresses Pathogenesis and Progression of Non-Small-Cell Lung Cancer through Targeting the RRM2/Wnt Axis
title_full MicroRNA-520a Suppresses Pathogenesis and Progression of Non-Small-Cell Lung Cancer through Targeting the RRM2/Wnt Axis
title_fullStr MicroRNA-520a Suppresses Pathogenesis and Progression of Non-Small-Cell Lung Cancer through Targeting the RRM2/Wnt Axis
title_full_unstemmed MicroRNA-520a Suppresses Pathogenesis and Progression of Non-Small-Cell Lung Cancer through Targeting the RRM2/Wnt Axis
title_short MicroRNA-520a Suppresses Pathogenesis and Progression of Non-Small-Cell Lung Cancer through Targeting the RRM2/Wnt Axis
title_sort microrna 520a suppresses pathogenesis and progression of non small cell lung cancer through targeting the rrm2 wnt axis
url http://dx.doi.org/10.1155/2021/9652420
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AT shuaiguo microrna520asuppressespathogenesisandprogressionofnonsmallcelllungcancerthroughtargetingtherrm2wntaxis
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