A novel sequence of the PHKG 2 mutation associated with the first case of glycogen storage diseases type IXc in Syria: a case report and review of literature

Abstract Background Glycogen storage diseases are a group of inherited metabolic disorders that affect the body’s ability to break down and/or store glycogen. Type IX glycogen storage disease is an inherited disorder caused by a deficiency of phosphorylase kinase, which leads to various symptoms. We...

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Main Authors: Seba Harh, Shaza Shahoud, Shady Daher, Diana Alasmar
Format: Article
Language:English
Published: BMC 2025-07-01
Series:Journal of Medical Case Reports
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Online Access:https://doi.org/10.1186/s13256-025-05383-z
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author Seba Harh
Shaza Shahoud
Shady Daher
Diana Alasmar
author_facet Seba Harh
Shaza Shahoud
Shady Daher
Diana Alasmar
author_sort Seba Harh
collection DOAJ
description Abstract Background Glycogen storage diseases are a group of inherited metabolic disorders that affect the body’s ability to break down and/or store glycogen. Type IX glycogen storage disease is an inherited disorder caused by a deficiency of phosphorylase kinase, which leads to various symptoms. We report the first reported case in Syria of glycogen storage disease type IXc caused by a novel phosphorylase B kinase catalytic subunit gamma 2 gene mutation, emphasizing the importance of early diagnosis and genetic counseling. Case presentation A 6-month-old Syrian male infant of Arab ethnicity presented with developmental delay, hepatomegaly, and hypoglycemia. Genetic testing identified a previously unreported phosphorylase B kinase catalytic subunit gamma 2 variant (c.801G > A p.( =)), classified as a variant of uncertain significance. Liver biopsy and clinical features were consistent with glycogen storage disease type IXc. Discussion This report expands the current understanding of phosphorylase B kinase catalytic subunit gamma 2-related glycogen storage disease type IXc by documenting a novel synonymous mutation with potential clinical significance. It underscores the critical role of early genetic testing in consanguineous populations, not only for accurate diagnosis but also for guiding family counseling and long-term management. Conclusion The identification of this novel mutation contributes to expanding the known phosphorylase B kinase catalytic subunit gamma 2 mutation spectrum and stresses the need for genetic counseling in similar populations.
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spelling doaj-art-7e31afc5c0614bf7a9abddf06bfb3db12025-08-20T03:03:25ZengBMCJournal of Medical Case Reports1752-19472025-07-011911510.1186/s13256-025-05383-zA novel sequence of the PHKG 2 mutation associated with the first case of glycogen storage diseases type IXc in Syria: a case report and review of literatureSeba Harh0Shaza Shahoud1Shady Daher2Diana Alasmar3Faculty of Medicine, Damascus UniversityFaculty of Medicine, Damascus UniversityFaculty of Medicine, Damascus UniversityInherited Metabolic Diseases at Damascus UniversityAbstract Background Glycogen storage diseases are a group of inherited metabolic disorders that affect the body’s ability to break down and/or store glycogen. Type IX glycogen storage disease is an inherited disorder caused by a deficiency of phosphorylase kinase, which leads to various symptoms. We report the first reported case in Syria of glycogen storage disease type IXc caused by a novel phosphorylase B kinase catalytic subunit gamma 2 gene mutation, emphasizing the importance of early diagnosis and genetic counseling. Case presentation A 6-month-old Syrian male infant of Arab ethnicity presented with developmental delay, hepatomegaly, and hypoglycemia. Genetic testing identified a previously unreported phosphorylase B kinase catalytic subunit gamma 2 variant (c.801G > A p.( =)), classified as a variant of uncertain significance. Liver biopsy and clinical features were consistent with glycogen storage disease type IXc. Discussion This report expands the current understanding of phosphorylase B kinase catalytic subunit gamma 2-related glycogen storage disease type IXc by documenting a novel synonymous mutation with potential clinical significance. It underscores the critical role of early genetic testing in consanguineous populations, not only for accurate diagnosis but also for guiding family counseling and long-term management. Conclusion The identification of this novel mutation contributes to expanding the known phosphorylase B kinase catalytic subunit gamma 2 mutation spectrum and stresses the need for genetic counseling in similar populations.https://doi.org/10.1186/s13256-025-05383-zGlycogen storage diseases type IXcGSD IXcPHKG2 geneHepatomegalyFasting hypoglycemiaNovel mutation
spellingShingle Seba Harh
Shaza Shahoud
Shady Daher
Diana Alasmar
A novel sequence of the PHKG 2 mutation associated with the first case of glycogen storage diseases type IXc in Syria: a case report and review of literature
Journal of Medical Case Reports
Glycogen storage diseases type IXc
GSD IXc
PHKG2 gene
Hepatomegaly
Fasting hypoglycemia
Novel mutation
title A novel sequence of the PHKG 2 mutation associated with the first case of glycogen storage diseases type IXc in Syria: a case report and review of literature
title_full A novel sequence of the PHKG 2 mutation associated with the first case of glycogen storage diseases type IXc in Syria: a case report and review of literature
title_fullStr A novel sequence of the PHKG 2 mutation associated with the first case of glycogen storage diseases type IXc in Syria: a case report and review of literature
title_full_unstemmed A novel sequence of the PHKG 2 mutation associated with the first case of glycogen storage diseases type IXc in Syria: a case report and review of literature
title_short A novel sequence of the PHKG 2 mutation associated with the first case of glycogen storage diseases type IXc in Syria: a case report and review of literature
title_sort novel sequence of the phkg 2 mutation associated with the first case of glycogen storage diseases type ixc in syria a case report and review of literature
topic Glycogen storage diseases type IXc
GSD IXc
PHKG2 gene
Hepatomegaly
Fasting hypoglycemia
Novel mutation
url https://doi.org/10.1186/s13256-025-05383-z
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