Nanovaccine loaded with seno-antigen target senescent cells to improve metabolic disorders of adipose tissue and cardiac dysfunction

The buildup of senescent cells exacerbates metabolic disorders in adipose tissue and contributes to aging-related cardiac dysfunction. Targeted clearance of senescent cells can markedly ameliorate these aging-related diseases. Here, we developed a novel nanovaccine (GK-NaV) loaded with seno-antigen...

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Main Authors: Kexin Zhang, Qiliang Yin, Yucen Ma, Mengyu Cao, Lingwei Li, Xinliang Jin, Jiyan Leng
Format: Article
Language:English
Published: Taylor & Francis Group 2025-12-01
Series:Human Vaccines & Immunotherapeutics
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Online Access:https://www.tandfonline.com/doi/10.1080/21645515.2025.2479229
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author Kexin Zhang
Qiliang Yin
Yucen Ma
Mengyu Cao
Lingwei Li
Xinliang Jin
Jiyan Leng
author_facet Kexin Zhang
Qiliang Yin
Yucen Ma
Mengyu Cao
Lingwei Li
Xinliang Jin
Jiyan Leng
author_sort Kexin Zhang
collection DOAJ
description The buildup of senescent cells exacerbates metabolic disorders in adipose tissue and contributes to aging-related cardiac dysfunction. Targeted clearance of senescent cells can markedly ameliorate these aging-related diseases. Here, we developed a novel nanovaccine (GK-NaV) loaded with seno-antigen that is self-assembled from the fusion of cationic protein (K36) and seno-antigen peptide (Gpnmb). The GK-NaV could be highly engulfed by bone marrow-derived dendritic cells (BMDCs) and efficiently present antigens on the cellular surface, thereby promoting DCs maturation and activation of CD8+T cells in vitro. Following subcutaneous immunization, GK-NaV not only exhibited a noticeable antigen depot effect but also markedly activated specific cellular immune responses, enhancing the immunoreactivity and cytotoxic effects of CD8+T cells. Consequently, the targeted anti-aging immunity triggered by GK-NaV demonstrated the ability to selectively eliminate senescent adipocytes and cardiomyocytes in high-fat diet (HFD)-induced progeroid mice, leading to a significant improvement in age-related metabolic disorders in adipose tissue and cardiac dysfunction. Hence, our findings indicate that immunization with GK-NaV targeting seno-antigens may represent a promising strategy for novel senolytic therapies.
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institution DOAJ
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publishDate 2025-12-01
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series Human Vaccines & Immunotherapeutics
spelling doaj-art-7d2268ffaa6942f0af9931b2fcc0cbb42025-08-20T03:05:21ZengTaylor & Francis GroupHuman Vaccines & Immunotherapeutics2164-55152164-554X2025-12-0121110.1080/21645515.2025.2479229Nanovaccine loaded with seno-antigen target senescent cells to improve metabolic disorders of adipose tissue and cardiac dysfunctionKexin Zhang0Qiliang Yin1Yucen Ma2Mengyu Cao3Lingwei Li4Xinliang Jin5Jiyan Leng6Department of Cadre Ward, The First Hospital of Jilin University, Changchun, ChinaDepartment of Cadre Ward, The First Hospital of Jilin University, Changchun, ChinaDepartment of Cadre Ward, The First Hospital of Jilin University, Changchun, ChinaDepartment of Cadre Ward, The First Hospital of Jilin University, Changchun, ChinaDepartment of Cadre Ward, The First Hospital of Jilin University, Changchun, ChinaDepartment of General Surgery, First Affiliated Hospital of Changchun University of Traditional Chinese Medicine, Changchun, ChinaDepartment of Cadre Ward, The First Hospital of Jilin University, Changchun, ChinaThe buildup of senescent cells exacerbates metabolic disorders in adipose tissue and contributes to aging-related cardiac dysfunction. Targeted clearance of senescent cells can markedly ameliorate these aging-related diseases. Here, we developed a novel nanovaccine (GK-NaV) loaded with seno-antigen that is self-assembled from the fusion of cationic protein (K36) and seno-antigen peptide (Gpnmb). The GK-NaV could be highly engulfed by bone marrow-derived dendritic cells (BMDCs) and efficiently present antigens on the cellular surface, thereby promoting DCs maturation and activation of CD8+T cells in vitro. Following subcutaneous immunization, GK-NaV not only exhibited a noticeable antigen depot effect but also markedly activated specific cellular immune responses, enhancing the immunoreactivity and cytotoxic effects of CD8+T cells. Consequently, the targeted anti-aging immunity triggered by GK-NaV demonstrated the ability to selectively eliminate senescent adipocytes and cardiomyocytes in high-fat diet (HFD)-induced progeroid mice, leading to a significant improvement in age-related metabolic disorders in adipose tissue and cardiac dysfunction. Hence, our findings indicate that immunization with GK-NaV targeting seno-antigens may represent a promising strategy for novel senolytic therapies.https://www.tandfonline.com/doi/10.1080/21645515.2025.2479229Nanovaccineseno-antigensenescent cellsmetabolic disorderscardiac dysfunction
spellingShingle Kexin Zhang
Qiliang Yin
Yucen Ma
Mengyu Cao
Lingwei Li
Xinliang Jin
Jiyan Leng
Nanovaccine loaded with seno-antigen target senescent cells to improve metabolic disorders of adipose tissue and cardiac dysfunction
Human Vaccines & Immunotherapeutics
Nanovaccine
seno-antigen
senescent cells
metabolic disorders
cardiac dysfunction
title Nanovaccine loaded with seno-antigen target senescent cells to improve metabolic disorders of adipose tissue and cardiac dysfunction
title_full Nanovaccine loaded with seno-antigen target senescent cells to improve metabolic disorders of adipose tissue and cardiac dysfunction
title_fullStr Nanovaccine loaded with seno-antigen target senescent cells to improve metabolic disorders of adipose tissue and cardiac dysfunction
title_full_unstemmed Nanovaccine loaded with seno-antigen target senescent cells to improve metabolic disorders of adipose tissue and cardiac dysfunction
title_short Nanovaccine loaded with seno-antigen target senescent cells to improve metabolic disorders of adipose tissue and cardiac dysfunction
title_sort nanovaccine loaded with seno antigen target senescent cells to improve metabolic disorders of adipose tissue and cardiac dysfunction
topic Nanovaccine
seno-antigen
senescent cells
metabolic disorders
cardiac dysfunction
url https://www.tandfonline.com/doi/10.1080/21645515.2025.2479229
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