Reverse epitope mapping of the E2 glycoprotein in antibody associated hepatitis C virus.

The humoral immune system responds to chronic hepatitis C virus (HCV) infection by producing neutralising antibodies (nAb). In this study we generated three HCV pseudoparticles in which E1E2 glycoprotein sequence was targeted by the host humoral immune system. We used patient derived virus free Fabs...

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Main Authors: Amruta S Naik, Ania Owsianka, Brendan A Palmer, Ciaran J O'Halloran, Nicole Walsh, Orla Crosbie, Elizabeth Kenny-Walsh, Arvind H Patel, Liam J Fanning
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2017-01-01
Series:PLoS ONE
Online Access:https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0175349&type=printable
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author Amruta S Naik
Ania Owsianka
Brendan A Palmer
Ciaran J O'Halloran
Nicole Walsh
Orla Crosbie
Elizabeth Kenny-Walsh
Arvind H Patel
Liam J Fanning
author_facet Amruta S Naik
Ania Owsianka
Brendan A Palmer
Ciaran J O'Halloran
Nicole Walsh
Orla Crosbie
Elizabeth Kenny-Walsh
Arvind H Patel
Liam J Fanning
author_sort Amruta S Naik
collection DOAJ
description The humoral immune system responds to chronic hepatitis C virus (HCV) infection by producing neutralising antibodies (nAb). In this study we generated three HCV pseudoparticles in which E1E2 glycoprotein sequence was targeted by the host humoral immune system. We used patient derived virus free Fabs (VF-Fabs) obtained from HCV genotype 1a (n = 3), genotype 1b (n = 7) and genotype 3a (n = 1) for neutralisation of HCVpp produced in this study both individually and in combination. Based on the available anti-HCV monoclonal nAb mapping information we selected amino acid region 384-619 for conformational epitope mapping. Amongst our notable findings, we observed significant reduction in HCVpp infectivity (p<0.05) when challenged with a combination of inter genotype and subtype VF-Fabs. We also identified five binding motifs targeted by patient derived VF-Fab upon peptide mapping, of which two shared the residues with previously reported epitopes. One epitope lies within an immunodominant HVR1 and two were novel. In summary, we used a reverse epitope mapping strategy to identify preferred epitopes by the host humoral immune system. Additionally, we have combined different VF-Fabs to further reduce the HCVpp infectivity. Our data indicates that combining the antigen specificity of antibodies may be a useful strategy to reduce (in-vitro) infectivity.
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spelling doaj-art-7c0920706b6d4cb28d45f68a59c79b592025-08-20T02:31:59ZengPublic Library of Science (PLoS)PLoS ONE1932-62032017-01-01125e017534910.1371/journal.pone.0175349Reverse epitope mapping of the E2 glycoprotein in antibody associated hepatitis C virus.Amruta S NaikAnia OwsiankaBrendan A PalmerCiaran J O'HalloranNicole WalshOrla CrosbieElizabeth Kenny-WalshArvind H PatelLiam J FanningThe humoral immune system responds to chronic hepatitis C virus (HCV) infection by producing neutralising antibodies (nAb). In this study we generated three HCV pseudoparticles in which E1E2 glycoprotein sequence was targeted by the host humoral immune system. We used patient derived virus free Fabs (VF-Fabs) obtained from HCV genotype 1a (n = 3), genotype 1b (n = 7) and genotype 3a (n = 1) for neutralisation of HCVpp produced in this study both individually and in combination. Based on the available anti-HCV monoclonal nAb mapping information we selected amino acid region 384-619 for conformational epitope mapping. Amongst our notable findings, we observed significant reduction in HCVpp infectivity (p<0.05) when challenged with a combination of inter genotype and subtype VF-Fabs. We also identified five binding motifs targeted by patient derived VF-Fab upon peptide mapping, of which two shared the residues with previously reported epitopes. One epitope lies within an immunodominant HVR1 and two were novel. In summary, we used a reverse epitope mapping strategy to identify preferred epitopes by the host humoral immune system. Additionally, we have combined different VF-Fabs to further reduce the HCVpp infectivity. Our data indicates that combining the antigen specificity of antibodies may be a useful strategy to reduce (in-vitro) infectivity.https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0175349&type=printable
spellingShingle Amruta S Naik
Ania Owsianka
Brendan A Palmer
Ciaran J O'Halloran
Nicole Walsh
Orla Crosbie
Elizabeth Kenny-Walsh
Arvind H Patel
Liam J Fanning
Reverse epitope mapping of the E2 glycoprotein in antibody associated hepatitis C virus.
PLoS ONE
title Reverse epitope mapping of the E2 glycoprotein in antibody associated hepatitis C virus.
title_full Reverse epitope mapping of the E2 glycoprotein in antibody associated hepatitis C virus.
title_fullStr Reverse epitope mapping of the E2 glycoprotein in antibody associated hepatitis C virus.
title_full_unstemmed Reverse epitope mapping of the E2 glycoprotein in antibody associated hepatitis C virus.
title_short Reverse epitope mapping of the E2 glycoprotein in antibody associated hepatitis C virus.
title_sort reverse epitope mapping of the e2 glycoprotein in antibody associated hepatitis c virus
url https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0175349&type=printable
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