Redox Signaling in Diabetic Nephropathy: Hypertrophy versus Death Choices in Mesangial Cells and Podocytes
This review emphasizes the role of oxidative stress in diabetic nephropathy, acting as trigger, modulator, and linker within the complex network of pathologic events. It highlights key molecular pathways and new hypothesis in diabetic nephropathy, related to the interferences of metabolic, oxidative...
Saved in:
Main Authors: | Gina Manda, Alexandru-Ionel Checherita, Maria Victoria Comanescu, Mihail Eugen Hinescu |
---|---|
Format: | Article |
Language: | English |
Published: |
Wiley
2015-01-01
|
Series: | Mediators of Inflammation |
Online Access: | http://dx.doi.org/10.1155/2015/604208 |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
Research Progress on the Pathological Mechanisms of Podocytes in Diabetic Nephropathy
by: Lili Zhang, et al.
Published: (2020-01-01) -
Podocyte Autophagy: A Potential Therapeutic Target to Prevent the Progression of Diabetic Nephropathy
by: Na Liu, et al.
Published: (2017-01-01) -
Sestrin2 Signaling Pathway Regulates Podocyte Biology and Protects against Diabetic Nephropathy
by: Moein Ala
Published: (2023-01-01) -
Tacrolimus Protects Podocytes from Apoptosis via Downregulation of TRPC6 in Diabetic Nephropathy
by: Ruixia Ma, et al.
Published: (2021-01-01) -
Upregulation of α3β1-Integrin in Podocytes in Early-Stage Diabetic Nephropathy
by: Kaichiro Sawada, et al.
Published: (2016-01-01)